Reflexive vision

Be aware of, anticipate and avoid the Normal Chemistiy Education reflex, i.e. the introduction into design and teaching of more chemical concepts than are needed for the chosen curriculum emphasis. Alternatively, deal in time with any difficulties related to the dominant school chemistiy curriculum at all stages of development, starting with the initial vision and the first design of the curriculum. 

The symptoms of acute exposure include headache, giddiness, nervousness, blurred vision, weakness, nausea, cramps, loss of muscle control or reflexes, convulsion, or coma. It has been suggested that impurities or additives may be associated with some cases of delayed polyneuropathy (damage to nerve cells) attributed to ingestion of large amounts of trichlorfon.87-90... 

About 30% of adult Tangier patients present with peripheral neuropathy, the onset of which usually takes place after 10 years of age. These neuropathic symptoms may be subtle or overt, transient or permanent, and include weakness, increased sweating, diplopia (double vision), ptosis (abnormally drooping eyelids), ocular muscle palsies, and diminished or absent deep-tendon reflexes. There are two prototypes of neuropathy found in Tangier dis-... 

Disturbed vision, ringing in the ears, tinnitus, loss of equilibrium, dizziness, pain, tactile disorders, tinghng, numbness, increased coldness Weakness, convulsions, tremors, paresis, twitching, lack of coordination, gait change, reflex abnormahties... 

During an acute imilateral attack, pupil testing reveals RAPD, because demyelinating disease can disrupt the impulses traveling within the pupillary fibers of the light reflex pathway. Color vision is reduced in most cases. Contrast sensitivity is reduced in cases of MS and may remain reduced after visual recovery occms. The ONTT reported that diffuse visual field loss occurred in 48.2% of eyes and that altitudinal field defects or other nerve fiber bimdle-type defects were present in 20.1% of eyes. Significantly, there was asymptomatic visual field involvement in the fellow eye in 68.8% of patients. 

Transient central blindness has followed an intravenous injection of atropine 0.8 mg in the course of spinal anesthesia blink reflex and pupillary response to light and accommodation were lost vision returned slowly after some hours after the instillation of pilocarpine (6). 

Mild ethanol intoxication is observed at blood alcohol levels in the range of 0.05-0.15%. Symptoms of exposure include impairment of visual acuity, muscular incoordination, decreased reaction time, and changes in mood, personality, or behavior. At blood alcohol levels of 0.15-0.3%, visual impairment, sensory loss, muscle incoordination, slowed reaction time, and slurred speech is observed. At levels of 0.3-0.5% blood alcohol, there is severe intoxication characterized by muscular incoordination, blurred or double vision, and sometimes stupor, hypothermia, vomiting, nausea, and occasionally, hypoglycemia and convulsions. At 0.4% and above, symptoms include coma, depressed reflexes, respiratory depression, hypertension, hypothermia, and possibly death from respiratory or... 

Typical effects of inhalation abuse are dizziness, palpitations, blurred vision, headache, nausea, and stinging of the nasal passages, eyes, and throat. There is a fall in blood pressure with a reflexive increase in pulse rate. Pulmonary irritation, tachypnea, and shortness of breath are described. Cardiovascular... 

IMS in OP-poisoned patients appears 24-96 h after an apparently well-treated acute cholinergic crisis phase. By definition, OP-poisoned patients should completely recover from the cholinergic crisis and then develop a syndrome. Clinically, IMS is characterized by acute paralysis and weakness in the territories of several cranial motor nerves, neck flexors, facial, extraocular, palatal, nuchal, proximal limb, and respiratory muscles 24-96 h after poisoning. Generalized weakness, depressed deep tendon reflexes, ptosis (drooping of the upper eyelids due to paralysis of the third cranial nerve), and diplopia (double vision of an object) are also evident. These symptoms may last for several days or weeks depending on the OP involved. Despite severe AChE inhibition, muscle fasciculations and muscarinic receptor-associated hypersecretory activities are absent. 

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