Rabbit diet

RC-4 Standard rabbit diet (Oriental yeast, Tokyo, Japan). 

Fecal material from captive rabbits and foxes was collected within one hour of excretion and frozen until required. Rabbits were maintained on a standard laboratory rabbit diet (Economy Rabbit Diet Special Diet Services Witham, Essex, UK). Foxes were maintained on a diet of rodents, chicks and rabbits. Approximately Ig of fecal material was defrosted on ice as required and smeared onto a 2cm piece of plastic-backed card (BenchGuard Bibby Sterilin Ltd, Staffs, UK) which was placed in nest boxes during trials. Distilled water was used as a non-odor control and was presented as lOOpl spots of distilled water on a 2cm piece of BenchGuard. 

Another approach to take is to assign a pair of identical rabbits (same sex) to each station and then give one of the rabbits Diet A and the other Diet B. The analysis would then be performed by considering the difference between the two rabbits at each station. Since it can be assumed that each station has two, nearly identical rabbits with very similar environmental factors, the effect of these envirOTimental variables on the two rabbits should be the same (or similar). Therefore, any difference between the pair can be attributed to the difference between the diets. 

The polyethers have been reported to increase growth performance in monogastric animals such as horses, swine, and rabbits. No commercial use has been approved for swine diets. The sensitivity of horses to the toxic effects of these antibiotics precludes consideration of their use. 

Vitamin Bjg is not synthesized by animals or by plants. Only a few species of bacteria synthesize this complex substance. Carnivorous animals easily acquire sufficient amounts of Bjg from meat in their diet, but herbivorous creatures typically depend on intestinal bacteria to synthesize Bjg for them. This is sometimes not sufficient, and certain animals, including rabbits, occasionally eat their feces in order to accumulate the necessary quantities of Big. 

There was a suggestion of dose-related immunosuppressive effects in rabbits fed methyl parathion in the diet at 0.04, 0.16, 0.57, and 1.48 mg/kg/day for 4 weeks. These effects consisted of decreased numbers of plasma cells in popliteal lymph nodes (at all doses compared to controls), decreased numbers of germinal... 

Minor, RL., Myers, P.R, Guerra, R, Bates, J.N. and Harrison, D.G. (1990). Diet-induced atherosclerosis increases the release of nitrogen oxides from rabbit aorta. J. Clin. Invest. 86, 2109-2116. 

Rabbit Biodynamic versus conventionally fertilised feed Larger number of eggs and higher fertilisation rate in rabbits fed biodynamic diet (Aehnelt and Hahn, 1978)... 

The expression of 15-LOX in atherosclerotic lesions is one of the major causes of LDL oxidative modification during atherosclerosis. To obtain the experimental evidence of a principal role of 15-LOX in atherosclerosis under in vivo conditions, Kuhn et al. [67] studied the structure of oxidized LDL isolated from the aorta of rabbits fed with a cholesterol-rich diet. It was found that specific LOX products were present in early atherosclerotic lesions. On the later stages of atherosclerosis the content of these products diminished while the amount of products originating from nonenzymatic lipid peroxidation increased. It was concluded that arachidonate 15-LOX is of pathophysiological importance at the early stages of atherosclerosis. Folcik et al. [68] demonstrated that 15-LOX contributed to the oxidation of LDL in human atherosclerotic plaques because they observed an increase in the stereospecificity of oxidation in oxidized products. Arachidonate 15-LOX is apparently more active in young human lesions and therefore, may be of pathophysiological importance for earlier atherosclerosis. In advanced human plaques nonenzymatic lipid peroxidation products prevailed [69],... 

Lead adversely affected the survival of sensitive mammals tested at different concentrations (Table 4.8) 5 to 108 mg Pb/kg BW in rats (acute oral), 0.32 mg Pb/kg BW daily in dogs (chronic oral), and 1.7 mg Pb/kg diet in horses (chronic dietary). Adverse sublethal effects (Table 4.8) were noted in monkeys given 0.1 mg Pb/kg BW daily (impaired learning 2 years postadministration) or fed diets containing 0.5 mg Pb/kg (abnormal social behavior) in rabbits given 0.005 mg Pb/kg BW (reduced blood ALAD activity) or 0.03 mg Pb/kg BW (elevated blood lead levels) in mice at 0.05 mg Pb/kg BW (reduced ALAD activity) or in sheep at 0.05 mg Pb/kg BW (tissue accumulations). 

Rabbit 40 mg/kg diet Muscular weakness after chronic exposure 21... 

Chlordane is readily absorbed by warm-blooded animals through skin, diet, and inhalation. It is quickly distributed in the body and tends to concentrate in liver and fat (WHO 1984). Up to 75% of a single oral dose of chlordane administered to rats and mice was absorbed in the gut, and up to 76% of an aerosol dose was absorbed in the respiratory tract (Nomeir and Hajjar 1987). Rabbits absorbed 33% in the gut following oral administration (USEPA 1988). Chlordane residues in mammals were usually not measurable 4 to 8 weeks after cessation of exposure (Ingle 1965). Chlordane persistence in human serum and whole body was estimated at 88 days and 21 days, respectively this compares to a Tb 1/2 of about 23 days in rats fed chlordane for 56 days (USEPA 1980). 

No data are available on effects of diflubenzuron on mammalian wildlife. However, results of studies on small laboratory animals and domestic livestock are available (Table 17.7), and these indicate several trends. Adverse effects levels occurred in dogs fed diets containing 160 mg/kg (6.2mg/kg BW daily) for 13 weeks (abnormal blood chemistry), in mice given 125 mg/kg BW daily for 30 days (hepatocellular changes), in rabbits fed diets containing 640 mg/kg for 3 weeks... 

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