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Insulin types

Insulin—types how dosage is expressed calculating die insulin dosage importance of using only die type, source, and brand name recommended by die health care provider importance of not changing brands unless the health care provider approves keeping a spare vial on hand prescription for insulin purchase not required. [Pg.498]

Skyler JS, Cefalu WT, Kourides IA, Landschulz WH, Balagtas CC, Cheng S-L, Gelfand RA (2001) Efficacy of inhaled human insulin type a diabetes mellitus a randomized proof-of-concept study. Lancet 357 331-335. [Pg.161]

Diabetic ketoacidosis, with or without coma (treat with insulin) type 1 diabetes ... [Pg.280]

Brand name Onset of action (h) Duration of action (h) Insulin type... [Pg.395]

Receptor-mediated endocytosis may be possible because receptors have a high affinity for BDNF (Deckner et al. 1993). A linear relation between intranasal administration of -labeled NGF and brain concentrations of the compound suggest that this transportation is not mediated by receptors and that this releasing method of agents to the brain via olfactory nerves may be effective for many therapies (Frey et al. 1995). BDNF and the insulin-type growth factor (IGF-1) are currently used in clinical studies (Appel 1997). [Pg.507]

When cells are continually occupied, they reduce the number of receptors to avoid having the metabolic effects overstimulated. For example, two kinds of diabetes exist, Type I and Type II. Type I diabetes, sometimes called juvenile diabetes, results from the inability of the pancreas to supply insulin. Type II diabetes, sometimes called adult-onset diabetes, is more common and correlates with obesity. In this situation, the body senses itself to be in a well-fed state and releases insulin from the pancreas. The large concentration of insulin causes the recipient cells to be fully stimulated. Consequently, they down-regulate their insulin receptor population to bring the response... [Pg.125]

Insulin [IN suh lin] is a small protein consisting of two polypeptide chains that are connected by disulfide bonds. It is synthesized as a precursor protein (pro-insulin) that undergoes proteolytic cleavage to form insulin and peptide C, both of which are secreted by the p-cells of the pancreas.4 [Note Normal individuals secrete less pro-insulin than insulin, whereas NIDDM patients secrete high levels of the prohormone. Since radioimmunoassays do not distinguish between the two insulin types, NIDDM patients may have lower levels of the active hormone than the assay indicates. Thus measurement of circulating C peptide provides a better index of insulin levels.]... [Pg.268]

Diabetes mellitus ( sweet urine ) involves relative over-production of glucose by the liver and under-utilization by other organs. Diabetes is the most serious metabolic disease in terms of its social impact. Obesity and the indulgent Western diet correlates with mature age diabetes. Type 1 diabetes (juvenile diabetes) typically manifests at less than 20 years from autoimmune destruction of the insulin-producing pancreatic (3 cells. Type 1 diabetes is insulin-dependent diabetes mellitus (IDDM) and is fatal without exogenous insulin. Type 2 diabetes mellitus (mature age diabetes) occurs later in life and typically involves both deficient insulin production and insulin resistance , that is, the target cells are less responsive to insulin. Type 2 diabetes is initially non-insulin-dependent diabetes (NIDDM) but insulin therapy (in addition to oral antidiabetics) may eventually be required. Hyperglycaemia due... [Pg.599]

Diabetes mellitus (DM) is a metabolic carbohydrate disorder that results from either insufficient insulin (type 1 DM) or the body s inability to recognize available insulin (type 2 DM). DM is a multifactorially inherited disorder this means that although people can inherit a propensity toward this condition, environment and diet can trigger onset of the actual disease. People who suffer from DM experience abnormally high blood... [Pg.294]

USP Insulin Type Strengths and Sources Usual Adult Dose ... [Pg.852]

Type 1 diabetes is due to autoimmune destruction of pancreatic islet beta cells causing loss of insulin. Type 2 diabetes is due to the combination of cellular resistance to insulin and beta cell failure. Tissue lesions are common to both types of diabetes, and chronic hyperglycemia (or a closely related metabolic abnormality) is responsible for diabetic complications including diabetic nephropathy. [Pg.1699]

Diabetes mellitus is due to either absolute deficiency of insulin (Type 1) or insulin resistance and reduced insulin secretion (Type 2). In both types metabolic disturbances of carbohydrates, proteins and lipids occur with serious long-term consequences including atheroma, micro-angiopathy (which can lead to blindness and kidney damage) and neuropathy. [Pg.112]

Keywords Insulin, insulin analogues, basal bolus insulin treatment, continuous subcutaneous insulin infusion, insulin pump, inhaled insulin, type 1 diabetes mellitus. [Pg.41]

Diabetes is a chronic disease that occurs either when the pancreas does not produce enough insulin (type 1) or in case of liver and peripheral insulin resistance (type 2). It affects 347 million people worldwide ... [Pg.456]

Diabetes is the third leading cause of death (heart disease and cancer are the first and second) in the United States. It is caused either by insufficient secretion of insulin (type 1 diabetes) or its inability to stimulate its target cells (type 2 diabetes). Injections of insulin can control some of the synptoms associated with diabetes. [Pg.1074]

A substance with insulin-type actions which may be produced in some tumours. [Pg.206]


See other pages where Insulin types is mentioned: [Pg.798]    [Pg.498]    [Pg.231]    [Pg.159]    [Pg.481]    [Pg.122]    [Pg.184]    [Pg.243]    [Pg.46]    [Pg.51]    [Pg.278]    [Pg.239]    [Pg.619]    [Pg.620]    [Pg.498]    [Pg.361]    [Pg.1272]    [Pg.79]    [Pg.418]    [Pg.176]    [Pg.206]    [Pg.64]    [Pg.100]    [Pg.283]    [Pg.420]    [Pg.197]    [Pg.420]    [Pg.561]    [Pg.230]   
See also in sourсe #XX -- [ Pg.360 , Pg.361 ]




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