Inflammatory genes expression

Bai, S.K., Lee, S.J., Na, H.J. et al. 2005. Beta-carotene inhibits inflammatory gene expression in lipopolysaccharide-stimulated macrophages by suppressing redox-based NF-kappaB activation. Exp Mol Med. 37 323-334. 

Marcheselh, V. L., Hong, S., Lukiw, W. J. et al. Novel docosanoids inhibit brain ischema-reperfusion-mediated leukocyte infiltration and pro-inflammatory gene expression. J. Biol. Chem. 278,43807-43817,2003. 

Modulation of Inflammatory Gene Expression by Trichothecene Mycotoxins... 

Trichothecene-Induced Inflammatory Gene Expression and Apoptosis... 

TRICHOTHECENE-INDUCED INFLAMMATORY GENE EXPRESSION AND APOPTOSIS... 

Pathway selective ER ligands have been reported that selectively inhibit nuclear factor kB (NF-kB) mediated gene expression [71], Since NF-kB is a pivotal regulator of pro-inflammatory gene expression, ligands that selectively inhibit the NF-kB pathway could be developed for the treatment of chronic inflammatory diseases such as arthritis, atherosclerosis, sepsis, and inflammatory bowel disease... 

Ito et al A molecular mechanism of action of theophylline Induction of histone deacetylase activity to decrease inflammatory gene expression. Proc Natl Acad Sci USA 2002 99 8921. [PMID 12070353]... 

Legendre, F., Bauge, M., Roche, R., Saurel, A. S., and Pujol, J. P. (2008). Chondroitin sulfate modulation of matrix and inflammatory gene expression in IL-l-stimulated chondrocytes-study in hypoxic alginate bead cultures. Osteoarthr. Cartil. 16,105-114. 

Feinstein DL, Heneka MT, Gavrilyuk V, Dello Russo C, Weinberg G, Galea E (2002) Noradrenergic regulation of inflammatory gene expression in brain. Neurochem. Int. 41 357-365. 

Novel docosanoids inhibit brain ischemia-reperfusion-mediated 137. leukocyte infiltration and pro-inflammatory gene expression. J. 

Lew W, Bowcock AM, Krueger JG. Psoriasis vulgaris cutaneous lymphoid tissue supports T-cell activation and Type T inflammatory gene expression. Trends Immunol 2004 25 295-305. 

Significantly, inflammation and lipid metabolism exhibit close functional interrelationships and are subject to coordinate, reciprocal regulation. PPARy and LXRs have been reported to reciprocally regulate genes involved in both immunity and lipid metabolism [6,90]. While the primary focus of the action of PPARy in inflammation has focused on receptor-mediated inhibition of inflammatory gene expression, there is a reciprocal effect of inflammation on nuclear hormone expression. Feingold and colleagues have extensively examined the inflammation-mediated suppression of PPARy and RXR expression [91]. 

Rahman, I., Marwick, J., and Kirkham, P. (2004). Redox modulation of chromatin remodehng impact on histone acetylation and deacetylation, NF-kB and pro-inflammatory gene expression. Biochem Pharmacol 68, 1255-67. 

Pei, L., Castrillo, A. and Tontonoz, P. (2006) Regulation of macrophage inflammatory gene expression by the orphan nuclear receptor Nur77. Molecular Endocrinology, 20, 786-794. 

Jobin, C., Bradham, C.A., Russo, M.P., Juma, B., Narula, A.S., Brenner, D.A., and Sartor, R.B., Curcumin blocks cytokine-mediated NF-kappa B activation and pro inflammatory gene expression by inhibiting inhibitory factor I-kappa B kinase activity, J. Immunol., 163 (6), 3474-... 

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