Diabetes Arteriosclerosis

High-blood pressure, diabetes, arteriosclerosis Improves oxygen consumption for strokes, etc. 

Bjomtorp, P. (1990) Portal Adipose Tissue as a Generator of Risk Factors for Cardiovascular Disease and Diabetes, Arteriosclerosis 70,493-496. 

Hollis, T. M., Gallik, S. G., Orlidge, A, and Yost, J. C., 1983, Aortic endothelial and smooth muscle histamine metabolism. Relationship to aortic st. lbumin accumulation in experimental diabetes. Arteriosclerosis 3 599-606. 

The CML homologue JV -(carboxyethyl)lysine (CEL, 4-231) forms in analogous intracellular reactions with methylglyoxal and is an important marker for age-dependent disease, such as cardiovascular disease in diabetic patients. The reaction of 3-deoxyhexosuloses with the e-amino group of bound lysine yields pyrraline, bound 6-(2-formyl-5-hydroxymethylpyrrol-l-yl)-L-norleucine (4-232), identified in hydrolysates of milk proteins and found in higher levels in serum of patients with diabetes, arteriosclerosis and Alzheimer s disease (Figure 4.98). 

Special risk Use with caution in the presence or history of the following Hypertension diabetes hyperthyroidism heart disease cerebral arteriosclerosis bronchial asthma. 

Aside from its use as an antimalarial compound, quinine is used for the prevention and treatment of nocturnal leg muscle cramps, especially those resulting from arthritis, diabetes, thrombophlebitis, arteriosclerosis, and varicose veins. 

Most cases of secondary ED are related to arteriosclerosis. ED is also associated with hypertension, antihypertensive therapy, and diabetes meUitus, particularly in the older diabetic. Other chronic diseases, such as psychogenic disorders and Peyronie s disease, may be associated with ED. 

It is indicated in arteriosclerosis, as adjunct in treatment of hypertension, diabetes and obesity. 

N.A. Linoleic acid, linolenic acid, phenolics, flavonoids, tannins.103,118,152 Treat asthma, arteriosclerosis, multiple sclerosis, atopic eczema, schizophrenia, diabetic neuropathy, cardiovascular diseases, antitumor. 

Of 34 Japanese patients with diabetes and chronic hepatitis, 18 were given glycyrrhizin 240-525 mg for over 1 year (447). This resulted in a significant lowering of total testosterone concentrations and increased arteriosclerotic plaque formation. The authors suggested that glycyrrhizin treatment was an independent risk factor for arteriosclerosis. The testosterone lowering effect of liquorice has been confirmed in another trial (448). 

More serious conditions for which people take herbal drugs include depression, irregular heartbeat and other heart problems, arthritis, arteriosclerosis (hardening of the arteries), asthma, high blood pressure, various cancers, diabetes, epilepsy, gallstones, sexually transmitted diseases, hepatitis, jaundice, malaria, measles, obesity, tetanus, tuberculosis, and ulcers. 

Shinomiya, K., Fukunaga, M., Kiyomoto, H., Mizushige, K., Tsuji, T., Noma, T., Ohmori, K., Kohno, M., and Senda, S. 2002. A role of oxidative stress-generated eicosanoid in the progression of arteriosclerosis in type 2 diabetes mellitus model rats. Hypertens. Res. 25, 91-98. 

Use phenylephrine 10% with caution in patients with cardiac disease, idiopathic orthostatic hypotension, hypertension, aneurysms, insulin-dependent diabetes, and advanced arteriosclerosis. 

The food we eat plays a critical role in our overall health. It is believed that a substantial amount of chronic disease risk is diet-related and could be significantly reduced through improvements in dietary habits, e.g., up to 70% of aU cancers in the United States are attributable to diet (95). Indeed, a recent report by the USDA indicates that poor diet and diet-related chronic diseases contribute to five of the ten leading causes of death (heart disease, cancer, stroke, diabetes, and arteriosclerosis) costing the United States economy an estimated 250 billion annually (96). 

M. Vaughn, G. Virella and M. Lopes-Virella, Diabetes. Autoimmunity and Arteriosclerosis, Clinical Immunology Immunopathology 52 (1989) 414-420. 

The diseases induced by LPO involve arteriosclerosis, diabetes mellitus, halotane hepatotoxicity and liver desease. Some antioxidants and scavengers inhibit peroxidation induced by ROS (i.e., a-tocopherol, glutathione and carotenoids). Recently, natural oxidants have been found from many plants, such as spices, vegatables and herbs [276]. Kaempferol and quercetin as flavonols scavenge ROS and inhibit LPO. 

Cardiac dysrhythmias, cerebral arteriosclerosis, pregnancy, narrow-angle glaucoma, cardiogenic shock Use with caution in hypertension, prostatic hypertrophy, hyperthyroidism, pregnancy, diabetes mellitus... 

Although erectile dysfunction is sometimes assumed to be a symptom of the aging process in men, it is unclear if the incidence is directly related to increasing patient age. Erectile dysfunction more likely results from concurrent medical conditions of the patient (e.g., hypertension, arteriosclerosis, hyperlipidemia, diabetes melli-tus, or psychiatric disorders) or from medications that patients may be taking for these diseases. " For example, up to 50% of patients with diabetes mellitus develop erectile dysfunction, and medications such as /3-blockers are associated with a high incidence of erectile dysfunction. 

Diseases that compromise vascular flow to the corpora caver-nosum (e.g., peripheral vascular disease, arteriosclerosis, and essential hypertension) are associated with an increased incidence of erectile dysfunction. Diseases that impair nerve conduction to the brain (e.g., spinal cord injury or stroke) or conditions that impair peripheral nerve conduction to the penile vasculature (e.g., diabetes mellitus) can result in erectile dysfunction. 

Overall, the life span of a person, once diagnosed and treated as a diabetic, is one-third less than that of their normal fellows. While there are other candidates as causes of the major long-term complications in diabetics (neuropathy, nephropathy, retinopathy, arteriosclerosis), there is good evidence that sustained hyperglycemia, especially with reference to non-insulin-sen-sitive tissues (lens, red cells, etc.), is a prime factor in initiating and aggravating these disorders. 

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