Wound maceration

High absorbent capacity and absorption rate, which promote free flow of body fluids and prevent fluid pooling at the wound site thus reducing the risk of bacterial infection and wound maceration. 

Water vapor permeable to prevent maceration and promote wound healing... 

The resinite macerals are in some ways the most varied group. They are derived from both the wound resins (terpenes) of plants and various other plant fats and waxes making up the lipid resins. The terpene-derived resinites are the most abundant type and they are found in most North American coals as ovoid masses. However, 1n some coals, especially in the western USA the resinite occurs mainly as a secondary form showing... 

Cola cordifolia (Cav.)R.Br. Bubbarn Abscess Infected wounds (B or R), Ocytocic (B) Paste powder in local use water maceration... 

Detarium senegalense J.F.Gmel. Bubukkut Infected wounds (Fr) Diarrhoea (L or B) Chews (L) Pulp in local application water maceration... 

Apart from the selective promotion of specific cell behavior, the ideal wound dressing would also provide an environment which would be more conducive to heahng. In this respect, the healing wound performs better in a moist healing environment but not in a macerated heahng environment. As such, the dressing should have the physical ability to deal with the exudates produced by the wound but at the same time not allow the wound to dry out and the tissues to desiccate. 

The basic principles of wound management are maintaining a moist environment at the wound surface (Pivec et al., 2014) and the removal of excess fluid from wound skin to prevent maceration or erosion of the wounds. The ability of a dressing to manage exudates and prevent maceration during healing is one of the most desirable properties for the cavity wound dressings (Bulck, 2008 Uzun, Anand, Shah, 2014). 

Edema is a condition of abnormally large fluid volume in the circulatory system or in tissues between the body s cells. For a healthy human body, the venous and lymphatic systems collaborate to maintain the micro- and macro-circulatory homeostasis of the tissues. Ulceration of the skin develops when the normal functions of these two systems are disturbed either as a result of diabetic diseases or by the application of excessive pressure on the tissue. This leads to large amounts of edema and wound exudate, with peri-wound skin having dermatitis, hyperemia or maceration, dark pigmentation, atrophy blanche, and fibrosis. Figure 8.1 shows an example of a patient with venous leg ulcers. 

---