Ulcerative differential diagnosis

Suggested Alternatives for Differential Diagnosis Epizootic lymphangitis, ulcerative lymphangitis, strangles, dourine, melioidosis, and fungal infections. 

Suggested Alternatives for Differential Diagnosis Amebiasis, cholera, salmonellosis, schisto-mosis, yersiniosis, Clostridium difficile colitis, colon cancer, Crohn s disease, ulcerative colitis. 

Aasuri MK,Venata N, Kumar VM. Differential diagnosis of microbial keratitis and contact lens-induced peripheral ulcer. Eye Contact Lens 2003 29(1 suppl) S60-S62 discussion S83-S84, SI92-SI94. 

The causal connection between NSAIDs and large bowel inflammation needs to be confirmed by appropriate epidemiological studies. Many publications have associated NSAID and colonic inflammation (SEDA-10, 77) (SEDA-15, 95), but the differential diagnosis between colonic inflammation arising de novo and exacerbation of underlying inflammatory bowel disease can be difficult, and the role of NSAIDs in aggravating ulcerative colitis or Crohn s disease or other inflammatory bowel disease is controversial (SEDA-10, 76) (SEDA-15, 95). A case-control study showed no association between appendi-cectomy for acute appendicitis and the use of NSAIDs (SEDA-22, 111). 

Law, M., Differential diagnosis of ulcerative lesions in fish. Environ. Health Perspect. 109 (Suppl 5), 681, 2001. 

The differential diagnosis includes pylorospasm (which may cause an increased muscle thickness, but only transiently), (eosinophilic) gastroenteritis, gastric and duodenal ulcers. If the US findings are not clearly diagnostic, an upper GI examination is next. 

Differential Diagnosis Between Ulcerative Colitis, Crohrfs Disease and Other Inflammatory Diseases 79... 

Table 8.2. Main ultrasonographic features in differential diagnosis between ulcerative colitis and Crohn s disease...

Limberg B, Osswald B (1994) Diagnosis and differential diagnosis of ulcerative colitis and Crohn s disease by hydrocolonic sonography. Am J Gastroenterol 89 1051-1057... 

In all cases, histopathological examination is clearly necessary to make a definite differential diagnosis with respect to spontaneous clinical forms presenting the same appearance. In the case of ulcerative lesions, for instance, histological examination does not show any specific signs, particularly at the level of the vessels and nerves. 

The patient with acute intermittent porphyria suffers a severe acute abdominal pain not definitely localized and without rigidity or tenderness of the abdominal wall. Moderate fever and leukocytosis develop. If the physician is not aware of the porphyria, he is likely to be confused and suspect appendicitis, renal or biliary colics, pancreatitis, perforated ulcer, acute bowel obstruction or another common cause of abdominal pain. The differential diagnosis of porphyria and bowel obstruction is further complicated because the attacks of porphyria hepatica are often associated with severe constipation. Abdominal X-rays of porphyric patients show colonic distension. The pathogenesis of the abdominal symptoms is not known. They could result either from a direct effect of porphobilinogen or porphyrin on the intestinal mucosa or be the consequence of an increased excitability of the autonomic system. 

Gutman AB, Swenson PC, Parsons WB. The differential diagnosis of hyperparathyroidism. JAMA 103 87-94,1934 Rogers HM, Keating FR Jr, Morlock CG, et al. Primary hypertrophy and hyperplasia of the parathyroid glands associated with duodenal ulcer. Arch Intern Med 19 301-32X, 1947. 

The diagnosis is made by appropriate combinations of colonoscopy, biopsy and contrast radiology. Disease has to be differentiated from ulcerative colitis (see above) and from infective disease, notably tuberculosis and amoebiasis as well as diverticular disease and cancer. 

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