Symptom sensory

The AEGL-1 value was based on the observation that exercising healthy human subjects could tolerate exposure to concentrations of 500 or 1,000 ppm for 4 h with no adverse effects on lung function, respiratory symptoms, sensory irritation, or cardiac symptoms (Utell et al. 1997). The exercise, which tripled the subjects minute ventilation, simulates an emergency situation and accelerates pulmonary uptake. Results of the exposure of two subjects for an additional 2 h to the 500-ppm concentration and the exposure of one subject to the 1,000-ppm concentration for an additional 2 h failed to elicit any clear alterations in neurobehavioral parameters. The 4- or 6-h 1,000-ppm concentration is a NOAEL in exercising individuals, there were no indications of response differences among tested subjects, and animal studies indicate that adverse effects occur only at considerably higher concentrations, so the 1,000-ppm value was adjusted by an uncertainty factor (UF) of 1. The intraspecies UF of 1 is supported by the lack of adverse effects in patients with severe... 

Spasticity Bladder Symptoms Sensory Symptoms Fatigue... 

The signs and symptoms of botulinum toxin exposure can be divided into early and late, and are summarised in Table 4.26. All serotypes and modes of exposure would result in the same symptoms. Sensory nerve impairment is typically absent. The presence of a fever should suggest a cause other than botulism. 

Benzodiazepine withdrawal may occur when use of the antianxiety drugs is abruptly discontinued after 3 to 4 months of therapy. Occasionally, withdrawal symptoms may occur after as little as 4 to 6 weeks of therapy. Symptoms of benzodiazepine withdrawal include increased anxiety, concentration difficultiesi, tremor, and sensory disturbances, such as paresthesias photophobia, hypersomnia, and metallic taste. To help prevent withdrawal symptoms, the nurse must make sure the dosage of the benzodiazepine is gradually decreased over a period of time, usually 4 to 6 weeks... 

An abstinence syndrome after long-term, low-dose treatment has also been described (Busto et al. 1986a Covi et al. 1973 Petursson and Lader 1981b Tyrer et al. 1981). Reported symptoms include muscle twitching, abnormal perception of movement, depersonalization or derealization, anxiety, headache, insomnia, diaphoresis, difficulty concentrating, tremor, fear, fatigue, lowered threshold to perception of sensory stimuh, and dysphoria. 

The effect of LSD on perception is sometimes referred to as illusiogen-ic, because, rather than creating a perception of a nonexisting stimulus, LSD produces a distortion of sensory input from the environment (Kulberg 1986). Visual symptoms are most frequently experienced for example, vision may be blurred, and the perception of distance and depth may be changed. Objects in the surroundings may be perceived as unusually intense in color, shape, and/or size. With the eyes closed, geometric and kaleidoscopic patterns are perceived. Synesthesia, by which a sensory stimulus of one modality is transformed into a perception from another sense, is a type of a perceptual distortion typically experienced under the influence of LSD for example, smells... 

The clinical presentation of MM in HIV-infected patients is similar to that in other patients with vasculitic neuropathy (Hoke and Comblath 2004). It is characterized by symptoms and signs of sensory involvement, with numbness and tingling in the distribution of one peripheral nerve trunk. Sequential involvement of other noncontiguous peripheral or cranial nerves progresses over days to weeks. The initial multifocal and random neurologic features may evolve to symmetrical neuropathy (Ferrari et al. 2006). 

Association of Pain, neuropathic pain is defined as pain initiated or caused by a primary lesion, dysfunction in the nervous system". Neuropathy can be divided broadly into peripheral and central neuropathic pain, depending on whether the primary lesion or dysfunction is situated in the peripheral or central nervous system. In the periphery, neuropathic pain can result from disease or inflammatory states that affect peripheral nerves (e.g. diabetes mellitus, herpes zoster, HIV) or alternatively due to neuroma formation (amputation, nerve transection), nerve compression (e.g. tumours, entrapment) or other injuries (e.g. nerve crush, trauma). Central pain syndromes, on the other hand, result from alterations in different regions of the brain or the spinal cord. Examples include tumour or trauma affecting particular CNS structures (e.g. brainstem and thalamus) or spinal cord injury. Both the symptoms and origins of neuropathic pain are extremely diverse. Due to this variability, neuropathic pain syndromes are often difficult to treat. Some of the clinical symptoms associated with this condition include spontaneous pain, tactile allodynia (touch-evoked pain), hyperalgesia (enhanced responses to a painful stimulus) and sensory deficits. 

A description of the patient s actions before admission to the hospital often provides valuable clues to the diagnosis. Since PCP seems to diminish the user s ability to integrate sensory input into meaningful behavior (Pender 1971), symptoms and findings suggesting this problem should be sought. A demonstration of sensory deprivation was provided by a patient who was found head down in a dumpster filled with garbage. He was nude and the weather was cold, but he didn t seem to be uncomfortable. He also... 

Homonymous visual symptoms and/or unilateral sensory symptoms... 

Systemic signs (e.g., fever, weight loss, accelerated hypertension) Focal neurologic symptoms (i.e., other than typical visual or sensory aura)... 

Assess the patient complaint to yield a detailed description of headache precipitating factors presence or absence of prodromal symptoms location, intensity, and duration of pain changes in sensory acuity and neurologic alterations. 

Improved symptoms of sensory loss using physical examination every 4 hours until symptoms improve and then daily... 

One of the most sensitive systems affected by lead exposure is the nervous system. Encephalopathy is characterized by symptoms such as coma, seizures, ataxia, apathy, bizarre behavior, and incoordination (CDC 1985). Children are more sensitive to neurological changes. In children, encephalopathy has been associated with PbB levels as low as 70 pg/dL (CDC 1985). The most sensitive peripheral index of neurotoxicity of lead is reported to be slowed conduction in small motor libers of the ulnar nerve in workers with 30-40 pg/dL lead in blood (Landrigan 1989). Other potential biomarkers of lead suggested for neurotoxicity in workers are neurological and behavioral tests, as well as cognitive and visual sensory function tests (Williamson and Teo 1986). However, these tests are not specific to elevated lead exposure... 

Cannabinoid receptors are expressed throughout the cerebral cortex and the hippocampus, and a subpopulation of these cells appear to show an unusually high level of activity. It is possible that cells in these areas modulate the sensory effects of cannabis, particularly the effects on perception, task performance and memory. In addition, the anticonvulsant properties of cannabis are believed to be mediated here. Parts of the hypothalamus show high levels of receptor sites for cannabinoids this may be related to hypothermia effects. High levels in the cerebellum may be related to mediating the property of cannabinoids that produces the reduction in ataxic (muscle co-ordination) symptoms in certain disorders (Herkenham et al., 1991). 

The objective of mastitis treatments is to cure the infected udders from the infection, but cure is defined in very different ways. For example, in economic terms, the farmer needs to achieve a level of udder health that allows expected milk yields and quality parameters specified by processors/ national regulations to be achieved. On the other hand, cure with respect to antibiotic treatments, is often defined in terms of absence of bacterial pathogens in milk (bacterial cure), with the proportion of cows without detectable pathogen presence following treatment being defined as the bacterial cure rate (BCR). The main problem with using BCR as the main indicator of cure is that it was frequently shown to include a proportion of cows with drastically elevated SCC values (indicative of sub-clinical mastitis) after treatment, but without clinical (sensory) symptoms. 

Partial (focal) seizures begin in one hemisphere of the brain and, unless they become secondarily generalized, result in an asymmetric seizure. Partial seizures manifest as alterations in motor functions, sensory or somatosensory symptoms, or automatisms. If there is no loss of consciousness, the seizures are called simple partial. If there is loss of consciousness, they are termed complex partial, and the patients may have automatisms, memory loss, or aberrations of behavior. 

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