Respiratory distress, severe

Acute bloodstream infection Patients with underlying illness such as HIV, renal failure, and diabetes are affected by this type of the disease, which usually results in septic shock. The symptoms of the bloodstream infection vary depending on the site of original infection, but they generally include respiratory distress, severe headache, fever, diarrhea, development of pus-filled lesions on the skin, muscle tenderness, and disorientation. This is typically an infection of short duration, and abscesses will be found throughout the body. 

Inhalation of vapors of lower chloroformates result in coughing, choking, and respiratory distress, and, with some chloroformates like methyl chloroformate, inhalation can be fatal as a result of the onset of pulmonary edema, which may not appear for several hours after exposure (55). Table 5 gives the acute toxicides of some chloroformates (55—57). 

Acute bronchospasm causes severe respiratory distress and wheezing from the forceful expiration of air and is considered a medical emergency, ft is characterized by severe respiratory distress, dyspnea, forceful expiration, and wheezing. The nurse must report these symptoms to the primary health care provider immediately. 

Sf.ef-Test 4.14A A baby with a severe bronchial infection is in respiratory distress. The anesthetist administers heliox, a mixture of helium and oxygen with 92.3% by mass 02. What is the partial pressure of oxygen being administered to the baby if atmospheric pressure is 730 Torr ... 

A classification of organophosphate poisoning has been proposed by Tafuri and Roberts (1987) modified from Namba et al. (1971). Clinical signs and symptoms of intoxication may occur when serum cholinesterase levels drop to below 50% of the normal value. Mild poisoning, with the patient still ambulatory, may occur when serum cholinesterase levels are 20-50% of normal moderate poisoning with inability to walk with levels 10-20% of normal and severe poisoning with respiratory distress and unconsciousness with serum cholinesterase levels <10% of normal. 

Phospholipids and sphingolipids are involved in several disease processes, including respiratory distress syndrome (lack of lung surfactant), multiple sclerosis... 

Depending upon the location and severity of the stroke at admission, patients may have cardiac and/or respiratory instability at the time of presentation to the emergency department (ED). They may need to be stabilized hemodynamically or intubated for airway protection or respiratory distress. Blood pressure management is often a crucial management issue, and the use of vasopressor or antihypertensive medications is common. In stroke patients at risk for malignant cerebral... 

Ognibene, F.P., Martin, S.E. and Parker, M.M. (1986). Adult respiratory distress syndrome in patients with severe neutropenia. N. Engl. J. Med. 315, 547-551. 

P. falciparum malaria is a life-threatening emergency. Complications include hypoglycemia, acute renal failure, pulmonary edema, severe anemia (high parasitism), thrombocytopenia, heart failure, cerebral congestion, seizures, coma, and adult respiratory distress syndrome. 

The plant produced pesticides. An intermediate compound in this process is methyl isocyanate (MIC). MIC is an extremely dangerous compound. It is reactive, toxic, volatile, and flammable. The maximum exposure concentration of MIC for workers over an 8-hour period is 0.02 ppm (parts per million). Individuals exposed to concentrations of MIC vapors above 21 ppm experience severe irritation of the nose and throat. Death at large concentrations of vapor is due to respiratory distress. 

Signs and Symptoms Fever, fatigue, cough, and mild chest discomfort is followed by severe respiratory distress with difficult or labored respiration, diaphoresis, a harsh vibrating sound heard during respiration in cases of obstruction of the air passages, and a blu-ish/purplish discoloration due to oxygenation of the blood. Shock and death occurs within twenty-four to thirty-six hours of severe symptoms. 

Differential Diagnosis An epidemic of inhalation anthrax in its early stage with nonspecific symptoms could be confused with a number of viral, bacteria, and fungal infections. Progression over two to three days with sudden development of severe respiratory distress followed by shock and death within twenty-four to thirty-six hours in essentially all untreated cases eliminates diagnosis other than inhalation anthrax. Other diagnosis to consider would include aerosol exposure to staphylococcal enterotoxin B (SEB), plague, or tularemia pneumonia. 

Symptoms Anthrax can fool many victims as symptoms are quite non-specific namely fever, malaise, fatigue, cough, and mild chest discomfort followed by severe respiratory distress and labored breathing, profuse perspiration, harsh vibrating sounds during respiration, and bluish discoloration from lack of oxygenation in the blood. A patient can go into shock, and death can occur within twenty-four to thirty-six hours after onset of severe symptoms. However, in some instances, there can be a short period of seeming before the victim dies. 

Symptoms Symptoms include acute onset of fever, chest tightness, cough, dyspnea, nausea, and arthralgias which occur four to eight hours after inhalational exposure. Airway necrosis and pulmonary capillary leak resulting in pulmonary edema would likely occur within eighteen to twenty-four hours, followed by severe respiratory distress and death from hypoxemia in thirty-six to seventy-two hours. 

Abrupt onset of high fever and severe respiratory distress (dyspnea, stridor, cyanosis)... 

Disulfoton exposure results in cholinergic signs such as salivation, diarrhea, pupil constriction, muscle tremors, and weight loss. Ataxia, convulsions, coma, respiratory distress, and death are common signs associated with a more severe toxicosis. Nervous tissue is evidently the most sensitive target organ. 

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