Resistance blood

Hydralazine. Hydrala2iae causes vasodilation ia all primary vascular beds and has more pronounced effects on capacitance than on resistance blood vessels. Despite the hypotension it produces, hydrala2iae iacreases renal blood flow and cardiac output. PRA iacreases with its use. Tachycardia, headache, di22iaess, and water and sodium retention are principal side effects of hydrala2iae therapy. 

Ohm s law may be rewritten to include the three factors that affect vascular resistance blood viscosity (q), vessel length (L), and vessel radius (r). The following equation is known as Poiseuille s law ... 

Kopt Ta M, Falinski R, Nowicki MO et al. BCR/ABL kinase induces self-mutagenesis via reactive oxygen species to encode imatinib resistance. Blood 2006 108 319-327. 

Prolonged oral therapy with labetalol results in cardiovascular responses similar to those obtained following conventional (3-blocker administration, that is, decreases in peripheral vascular resistance, blood pressure, and heart rate. Generally, however, the decrease in heart rate is less pronounced than after administration of propranolol or other (3-blockers. 

Mahon FX, Deininger MW, Schultheis B et al. Selection and characterization of BCR-ABL positive cell lines with differential sensitivity to the tyrosine kinase inhibitor STI571 diverse mechanisms of resistance. Blood 2000 96 1070-1079. 

A 64-year-old man with giant cell arteritis was given prednisolone 60 mg/day. Within 5 days he developed double vision and agitation and became drowsy and confused. A cranial MRI scan showed recent cerebral lesions and a Doppler scan showed high-resistant blood flow in both vertebral arteries. He had an episode of complete loss of vision and was given dexamethasone... 

Thurston G, Suri C, Smith K, et al, Leakage-resistant blood vessels in mice transgenically overexpressing angiopoietin-1. Science 1999 286 251 1-2514. 

These mediators can produce a number of effects including bronchiolar constriction, capillary dilatation, or urticaria (i.e., hives). In severe episodes of type I reactions a life-threatening anaphylaxis can develop in humans due to extreme bronchoconstriction and precipitate hypotension. Epinephrine is the principal drug used in the acute management of these critical effects since it achieves (1) an elevated blood pressure via activation of alpha receptors in peripheral resistance blood vessels and (2) relaxation of bronchiolar smooth muscle via activation of (32 receptors in the lung. Relief from the dermatological problem (i.e., hives) is also achieved via vasoconstriction of capillaries in the skin that reduce permeability, and, hence, fluid accumulation. Penicillin is a classic example of a drug that can cause a type I reaction. 

Bhardwaj R, Moore PK Endothelium-derived relaxing factor and the effects of acetylcholine and histamine on resistance blood vessels. Br J Pharmacol 95 835-43,1988... 

Rosendaal FR, Koster T, Vandenbroucke JP, Reitsma PH. High risk of thrombosis in patients homozygous for factor V Leiden (activated protein C resistance). Blood 1995 85 1504-8. 

A Method to Count Flow-Resistant Blood Microemboli... 

This drug is particularly useful by decreasing renal vascular resistance. Blood flow to the kidney is thus not compromised, and less end-organ damage is produced. 

The same cardiovascular control system regulates blood distribution and blood pressure by affecting the small arterioles of the peripheral blood vasculature. The entrance to each of these vessels is surrounded by a sphincter muscle (a ring of involuntary muscle that surrounds the arteriolar aperture) with sympathetic, and in some cases, parasympathetic, nerve fibers. The sphincter is usually contracted. When the signal comes for the muscle to relax, the neuron produces nitric oxide at the neuromuscular junction, and this gas relaxes the sphincter. When the sphincter muscle expands, it increases the area through which blood flows and decreases its resistance. With decreased resistance, blood pressure falls. 

Poly(vinyl chloride) Transparency, good scuff resistance Blood bags, catheters, cannulae, corrugated tubing, renal care products, transfusion supplies, face masks... 

Activity V. reduces peripheral vascular resistance (blood pressure lowering activity), it promotes cranial perfusion and is purported to improve cerebral metabolism. It is thus recommended as treatment for cerebrovascular disorders. V. is also used in the treatment... 

Lakshmi S, Jayakrishnan A. Migration resistant, blood-compatible plasticized polyvinyl chloride for medical and related applications. Artificial Organs 1998 22(3) 222—9. 

Vancomycin-resistant Blood, cardiovascular, intra- Quinupristm-dalfopristm,... 

The growth of the chloroquine resistant blood parasite Plasmodium falciparum (responsible for malaria) was markedly inhibited in vitro by certain quassinoids 103). The most active compound simalikalactone D (83) gave complete inhibition at 0.002 pg/ml. Glaucarubinone (36a) and soularubinone (35) were equally effective at 0.006 pg/ml, whereas chapar-rinone (40a) and simarolide (77) had little effect even at 0.01 pg/ml. These relative activities parallel the antineoplastic activities of these compounds. Indeed, the two last mentioned quassinoids (40 a) and (77) do not possess the structural requirements for antileukemic activity. 

The literature [5] indicates that there were many adhesives based on blood albumen or combinations of blood albumen with phenol-aldehydes, casein, soybean meal and other protein-based products. However, in aircraft structures blood-based glues would have been almost exclusively used in plywood [6,7]. With the aircraft industry s reluctance to use casein adhesives, due to their poor water resistance, blood albumen glues and/or blood albumen/casein or occasionally blood albumen/soya bean extract became the primary adhesive systems for the preparation of plywood until ousted by the Tego phenol-formaldehyde (P/F) system in the early to mid-1930s. 

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