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Postprandial blood glucose

Euglycaemia, blood glucose concentration within the normal range e.g. fasting blood glucose 3.5 - 6.5 mmol/1 postprandial blood glucose 5-11 mmol/1. [Pg.883]

Intensive insulin therapy, the administration of insulin three or more times daily to maintain preprandial blood glucose levels between 70 and 120 g/dL and postprandial blood glucose levels less than 180 g/dL, has been shown to decrease the incidence of proteinuria and albuminuria in patients with diabetes, both with and without documented nephropathy. The development and progression of nephropathy is also delayed in patients with type 1 DM receiving intensive insulin therapy. Continued benefits of intensive insulin therapy have been demonstrated up to 8 years after the study.16... [Pg.378]

PPBG Postprandial blood glucose RRT Registered Respiratory Therapist... [Pg.1557]

Postprandial blood glucose values less than 250 mg/dL... [Pg.313]

Alpha glucosidase inhibitors mainly reduced postprandial blood glucose and has a mean lowering of initial HbAlc of 0.5-1.0%. The major side effects are abdominal discomfort. Hence it is advised to begin with a low dose (25-50 mg) at the start of meals and increase slowly up to a dose of 100 mg three times daily, as judged by the patient s response. [Pg.757]

Delays gastric emptying Generates sensation of fullness Reduces postprandial blood glucose concentration... [Pg.364]

When acarbose or placebo was given to patients with type 1 diabetes taking insulin, acarbose reduced postprandial blood glucose but there was no difference in HbAic the only adverse effects were gastrointestinal (23). [Pg.360]

In 19 patients with type 1 diabetes using regular insulin and 21 using insulin lispro, who injected pramlintide 60 micrograms or placebo before a standardized breakfast in addition to their normal insulin treatment, there was a marked reduction in the postprandial blood glucose excursion mild hypoglycemia (25%) and mild nausea (18%) were the most frequent adverse events (5). [Pg.366]

Ellison JM, Stegmann JM, Coiner SL, Michael RH, Sharma MK, Ervin KR, Horwitz DL. Rapid changes in postprandial blood glucose produce concentration differences at finger, forearm, and thigh sampling sites. Diabetes Care 2002 25(6) 961-4. [Pg.383]

Frequent addition of isophane to a regimen of insulin aspart is unnecessary, as has been shown in a multicenter, multinational, randomized, open study in 368 patients followed for 64 weeks (10). Frequent addition of isophane up to four times daily to insulin aspart did not improve HbAic or change the number of episodes of hypoglycemia compared with regular insulin combined with isophane. Only postprandial blood glucose concentrations were reduced. [Pg.422]

During Ramadan, insulin lispro reduced the number of attacks of hypoglycemia and reduced postprandial blood glucose (4). It also reduced post-snack raised blood glucose concentrations when sugar-rich snacks were used (5). [Pg.428]

In an open, randomized, crossover study, 113 patients with at least 6 months of continuous subcutaneous insulin infusion before the study were treated with regular insulin or insulin lispro (2). Postprandial blood glucose was lower and HbAlc fell more with insulin lispro. There were no differences in catheter obstruction, hypoglycemic episodes, or other adverse effects. Satisfaction with treatment was better with insulin lispro. [Pg.428]

Premeal hyperglycemia is common. The short action of insulin lispro can then be extended by the addition of protamine zinc insulin. In a 3-month study in addition to a once-daily injection of protamine zinc insulin, at each meal insulin lispro or insulin lispro + protamine zinc insulin was injected the postprandial blood glucose concentration was lower, but the post-absorptive glucose concentration was higher in the insulin lispro-only group there was no difference in HbAic. The addition of protamine zinc insulin (30% at breakfast, 40% at lunch, and 10% at dinner) improved post-absorptive glucose and HbAic (12). [Pg.428]

In eight patients with type 2 diabetes, nateglinide given 5 minutes before a meal reduced the postprandial blood glucose excursion by 64% (18). There were no attacks of hypoglycemia even with a dosage of 180 mg/day other adverse effects were not mentioned. [Pg.434]

Gagliardino JJ. Physiological endocrine control of energy homeostasis and postprandial blood glucose levels. Eur Rev Med Pharmacol Sci. 2005 9 75-92. [Pg.493]

Dietary fibre and diabetes. The addition of gel-forming (soluble) but unabsorbable fibre (guar gum, a hydrocoUoidal polysaccharide of galactose and mannose from seeds of the cluster bean ) to the diet of diabetics reduces carbohydrate absorption and flattens the postprandial blood glucose curve. Reduced need for insixlin and oral agents are reported, but adequate amoimts (taken with lots of water) are impleasant (flatulence) and patient compliance is therefore poor. [Pg.689]


See other pages where Postprandial blood glucose is mentioned: [Pg.486]    [Pg.644]    [Pg.657]    [Pg.661]    [Pg.226]    [Pg.264]    [Pg.279]    [Pg.1275]    [Pg.8]    [Pg.430]    [Pg.431]    [Pg.435]    [Pg.450]    [Pg.582]    [Pg.41]    [Pg.187]    [Pg.251]    [Pg.256]    [Pg.127]    [Pg.486]    [Pg.213]    [Pg.311]    [Pg.909]    [Pg.1784]    [Pg.2239]    [Pg.829]    [Pg.1902]    [Pg.155]   
See also in sourсe #XX -- [ Pg.328 ]




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