Myeloblastic leukemia

Example on Myeloblastic Leukemia. The term myeloblastic leukemia can be easily identified in a document or record by text mining. 

Term to UltraLink myeloblastic leukemia Concept Type DISEASE Normalized term myeloid leukemia Synonyms ... 

TUMOR NECROSIS FACTOR ALPHA STIMULATES THE GROWTH OF THE CLONOGENIC CELLS OF ACUTE MYELOBLASTIC LEUKEMIA IN SYNERGY WITH GRANULOCYTE / MACROPHAGE COLONY -STIMULATING FACTOR... 

M1 Acute myeloblastic leukemia with minimal maturation 15 17 25... 

Chuang LF, Hinton DE, Cheung ATW, et al. 1991. Induction of differentiation in human myeloblastic leukemia ML-1 cells by heptachlor, a chlorinated hydrocarbon insecticide. Toxicol AppI Pharmacol 109 98-107. 

Matsuno, N., Osato, M., Yamashita, N., Yanagida, M., Nanri, T., Fukushima, T., Motoji, T., Kusumoto, S., Towatari, M., Suzuki, R., etal. (2003) Dual mutations in the AMLl and FLT3 genes are associated with leukemogenesis in acute myeloblastic leukemia of the MO subtype. Leukemia 17, 2492-2499. 

Arylamino-substituted-pyrrolo[2,3-3]pyridines, synthesized via 4-chloropyrrolo[2,3-/ ]pyridine, have been evaluated for their biological properties on human myeloblastic leukemia cells <2007BML1934>. 

Soon after the discovery of 1-ASP as an antineoplastic agent, its antitumor specificity was studied in patients with cancer. Unfortunately, only one tumor consistently responded to the drug childhood and adult ALL. Other tumors responded inconsistently, like acute myeloblastic leukemia (AML), non-Hodgkin s... 

Xu DZ, Wang L, Dai W, Lu L (1999) A requirement for K+-channel activity in growth factor-mediated extracellular signal-regulated kinase activation in human myeloblastic leukemia ML-1 cells. Blood 94 139-145... 

Acute myeloblastic leukemia occurred in a 24-year-old man who had taken massive doses of amfetamine for more than 2 years (SED-8,13). 

Hess, C.E., Joyce, R.A. Acute myeloblastic leukemia presenting as progressive hepatic failure. South Med. J. 1981 74 1028-1029... 

In an open study in eight young healthy Japanese women, the pharmacokinetics of amikacin were affected by the phase of the menstrual cycle (12). In patients with hematological malignancies, bodyweight, renal function, acute myeloblastic leukemia, and hypoalbuminemia were the most important co-variates for the interindividual variability in amikacin pharmacokinetics (13). 

In a small fraction of patients who survive the chronic type of bone marrow damage, myeloblastic leukemia develops (41,42). In most instances this complication has appeared within a few months of the diagnosis of aplasia and was considered to be a sequel of chloramphenicol treatment. Sometimes the delay was shorter. The majority were either children or adults aged 50-70 years. 

Katayama Y, Deguchi S, Shinagawa K, Teshima T, Notohara K, Taguchi K, Omoto E, Harada M. Bone marrow necrosis in a patient with acute myeloblastic leukemia during administration of G-CSF and rapid hematologic recovery after allotransplantation of peripheral blood stem cells. Am J Hematol 1998 57(3) 238 0. 

Soutar RL. Acute myeloblastic leukemia and recombinant granulocyte colony stimulating factor. BMJ 1991 ... 

A patient with acute myeloblastic leukemia, who took tioguanine for 2 months, developed severe peliosis hepatis associated with mild lesions of the centrilobular veins withdrawal was followed by progressive improvement of liver dysfunction (7). 

A syndrome similar to that of the retinoic acid syndrome occurred after 10 days of tretinoin therapy in a patient with a relapse of acute myeloblastic leukemia (31). 

A 75-year-old woman whose acute myeloblastic leukemia relapsed was treated with one dose of intravenous idarubicin (10 mg/m ), cytarabine 20 mg subcutaneously for 10 days, and oral tretinoin 45 mg/m /day. 

IL-9 can support cell proliferation in the absence of an antigen or APC, Overproduction of it may lead to T-cell tumors, particularly of Th2 lymphocytes. In certain lymphomas, such as Hodgkin s lymphoma, it may act as an autocrine signal for cellular proliferation. IL-9 may play a role in the development of acute myeloblastic leukemia by stimulating leukemic cells to enter the S-phase rather than preventing cell death. In addition, IL-9 acts synergisticaUy with stem cell factor for recruitmg quiescent leukemic cells in the cell cycle. ... 

Cheng GY, Minden MD, Toyonaga B, Mak TW, McCulloch EA. T cell receptor and immunoglobulin gene rearrangements in acute myeloblastic leukemia. 

Gallily R, Even-Chen T, Katzavian G, Lehmann D, Dagan A, Mechoulam R (2003) y-Irradiation enhances apoptosis induced by cannabidiol, a non-psychotropic cannabinoid, in cultured HL-60 myeloblastic leukemia cells. Leuk Lymphoma 44 1767-1773... 

In Ml myeloblastic leukemia cells, the addition of either LIF or IL-6 induces the immediate early response genes jun-B, c-jun, jun-D, ICAM and MyD (i (myeloid differentiation primary response). On the other hand, My788 mRNA in Ml... 

It is used in the treatment of Hodgkin s disease, monocytic a drug of third choice in the treatment of neuroblastoma, breast tumours and mycosis fimgoides. It is combined with vincristine in the treatment of lymphocytic and myeloblastic leukemia in children. 

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