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Lung cancer distribution

Jun G et al. Ethnic differences in poly CADP-ribose) polymerase pseudogene genotype distribution association with lung cancer risk. Carcinogenesis 1999 20 1465-1469. [Pg.524]

Kunz E., Sevc J. and Placek V., 1979, Lung Cancer in Man in Relation to Different Time Distribution of Radiation Exposure, Health Phys., 36, 699-709. [Pg.88]

Hofmann, W., Dose Calculations for the Respiratory Tract from Inhaled Natural Radionuclides as a Function of Age - II. Basal Cell Dose Distributions and Associated Lung Cancer Risk, Health Phys. 43 31-44 (1982). [Pg.417]

From Assumption C and Eq. (l), the distribution of Mr is accurately known. From statistics on lung cancer mortality, the distribution of M is accurately known. Thus, the distribution of Mr can be calculated mathematically, and the problem is completely solved, allowing us to derive predictions that can be tested. In particular,... [Pg.466]

The amount of particles determine the quantity of decay products that stay in the air (equilibrium fraction, F) and the fraction of activity associated with the "unattached or ultrafine mode of the size distribution (fDot) These decay products are certainly harmful at high concentrations but we cannot yet detect the effects at normal levels because the vast majority of lung cancer death are due to smoking. Models predict that potentially 9000 lung cancer deaths per year in the United States are due to indoor radon. Methods are currently available and new methods are being developed and tested for lowering the levels of radon in indoor air. [Pg.583]

Table 3. Lung and stomach cancer distribution and average content of trace elements in soils and drinking waters of various bio geochemical provinces. Table 3. Lung and stomach cancer distribution and average content of trace elements in soils and drinking waters of various bio geochemical provinces.
One hint of possible trouble to come is provided by the information we described in Chapter 4, related to airborne particulate matter (PM). The available evidence ascribes significant increases in the risks of asthma and other respiratory diseases, certain cardiovascular conditions, and lung cancer to PM exposure, particularly those that average less than 2.5 pm (2500 nm) in size. As we noted, the chemical composition of these particles varies widely, depending upon source, but may not be as important as particle size as a risk determinant. Moreover, there is some experimental evidence pointing to the so-called ultra-fines, PM with dimensions below 100 nm, as significant contributors to PM risk. In addition some experimental studies have demonstrated that ultrafines not only distribute themselves throughout the airways, but seem to be able to translocate to other parts of the body - liver, heart, perhaps the CNS. [Pg.268]

Bristol-Myers Squibb Co. manufactures and distributes paclitaxel (brand name, Taxol, and generically known as taxol), which the FDA has approved for treatment of ovarian, breast and lung cancers and AIDs-related Kaposi s sarcoma. Bristol s patent listings in the Orange Book and the related patent infringement suits demonstrate that invalid patents in combination with the lengthy pendency of a patent application can potentially generate an additional 30-month stay. [Pg.119]

Ikegami Y, Yano S, Nakao K (1996b) Effects of the new selective kinase C inhibitor 4 -N-benzoyl staurosporine on cell cyde distribution and growth inhibition in human small cell lung cancer cells. Arzndm.-Forsch 46 201-204... [Pg.75]

Radon is another example of a very curious and toxic compound that many of us regularly inhale, hopefully in small amounts. For those regularly exposed to radon, there is an increased risk for lung cancer and, for those that smoke, radon exposure results in a three-fold increase in the incidence of lung cancer. In the United States it is estimated that indoor radon exposure causes between 7000 and 30,000 lung cancer-related deaths each year, second only to tobacco smoking. Radon-222 is a colorless and odorless radioactive gas that results from the decay of radium-226, which is widely distributed in the earth s crust. Radon decays with a half-life of 3.8 days into solid particles of polonium. It is actually the breakdown of... [Pg.204]

Tsapakos MJ, Hampton TH, Wetterhahn KE. 1983b. Chromium(VI)-induced DNA lesions and chromium distribution in rat kidney, liver, and lung. Cancer Res 43 5662-5667. [Pg.467]


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See also in sourсe #XX -- [ Pg.434 ]




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