Inclusion body myositis, sporadic muscle

Fratta P, Engel WK, McFerrin J et al. (2005) Protea-some inhibition and aggresome formation in sporadic inclusion-body myositis and in amyloid-beta precursor protein-overexpressing cultured human muscle fibers. Am J Pathol 167(2), 517-526. 

Pathogenesis of sporadic inclusion-body myositis role of aging and muscle-fiber degeneration, and accumulation of the same proteins as in Alzheimer and Parkinson brains... 

Vattemi G, Nogalska A, King Engel W et al. (2009) Amyloid-beta42 is preferentially accumulated in muscle fibers of patients with sporadic inclusion-body myositis. Acta Neuropathol 117, 569-574. 

Vattemi G, Kefi M, Engel WK et al. (2003) Nicastrin, a novel protein partidpating in amyloid-beta production, is overexpressed in sporadic inclusion-body myositis muscle. Neurology 60, A315. 

Askanas V, Engel WK, Yang CC etal. (1998) Light and electron microscopic immunolocaUzation of preseni-lin 1 in abnormal muscle fibers of patients with sporadic inclusion-body myositis and autosomal-recessive indusion-body myopathy. Am J Pathol 152, 889-895. 

Askanas V, Sarkozi E, Alvarez RB et al. (1996) Superoxide dismutase-1 gene and protein in vacuolated muscle fibers of sporadic inclusion-body myositis, hereditary inclusion-body myopathy, and cultured human muscle after beta-amyloid precursor protein gene transfer. Neurology 46, A487. 

Nogalska A, Engel W, Askanas V. (2007) Abnormalities of peroxisome proliferator-activated receptor gamma in sporadic inclusion-body myositis muscle fibers. Ann Neurol 62, S13. 

D Agostino C, Nogalska A, Engel WKetal. (2011) In sporadic inclusion-body myositis muscle fibres TDP-43-positive inclusions are less frequent and robust than p62-inclusions, and are not associated with paired helical filaments. Neuropathol Appl Neurobiol 37, 315-320. 

Guerin C, SinnreichM, Karpati G. (2008) Transcri-tional dysregulation in muscle fibers in sporadic inclusion body myositis (slBM). Neurology 7 (Suppl. 3), A304. 

Muth IE, Barthel K, Bahr M et al. (2009) Proinflam -matory cell stress in sporadic inclusion body myositis muscle overexpression of aB-crystallin is associated with amyloid precursor protein and accumulation of p-amyloid. J Neurol Neurosurg Psychiatry 80, 1344-1349. 

Wojcik S, Paciello O, Engel WK et al. (2007) In sporadic inclusion-body myositis muscle fiber cytoplasm, cytochrome c aggregates with a-synudein and amyloid-beta precursor protein, but does not activate caspase-3. Neuromuscul Disord 17, 853. 

Nogalska A, D Agostino C, Engel WK et al. (2008) Resveratrol, a polyphenol found in red wine, reduces NF-kB activation and myostatin in endoplasmic reticulum stress (ERS)-provoked cultured human muscle fibers relevance to treatment of sporadic inclusion-body myositis. Ann Neurol 64, S9. 

Koffman BM, Rugiero M, Dalakas MC. (1998) Autoimmune diseases and autoantibodies associated with sporadic inclusion body myositis. Muscle Nerve 21,115-117. 

SalajeghehM, Rakocevic G, Raju R et al. (2007) T cell receptor profiling in muscle and blood lymphocytes in sporadic inclusion body myositis. Neurology 69, 1672-1679. 

Ozden S, Cochet M, Mikol J et al. (2004) Direct evidence for a chronic CDS -I- -T-cell-mediated immune reaction to tax within the muscle of a human T-cell leukemia/lymphoma virus type 1-infected patient with sporadic inclusion body myositis. J Virol 78, 10320-10327. 

Barkhaus PE, Periquet MI, Nandedkar SD. (1999) Quantitative electrophysiologic studies in sporadic inclusion body myositis. Muscle Nerve 22(4), 480-487. 

Pathologic diagnostic criteria of sporadic inclusion-body myositis and hereditary inclusion-body myopathy muscle biopsies... 

Nogalska A, Terracciano C, D Agostino C et al. (2009) p62/SQSTMl is overexpressed and prominently accumulated in inclusions of sporadic inclusion-body myositis muscle fibers, and can help differentiating it from polymyositis and dermatomyositis. Acta Neuropathol 118, 407- 13. 

Sporadic inclusion-body myositis the main distinctive points of this condition from the GNE myopathy are the later onset and the presence of diffuse inflammatory infiltrates in the muscle sample. Also, in the sporadic condition the quadriceps is usually the first muscle to be involved. 

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