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Gastroenteritis viral

Evans, M. R., Meldrum, R., Lane, W., Gardner, D., Ribeiro, C. D., Gallimore, C. L, and Westmoreland, D. (2002). An outbreak of viral gastroenteritis following environmental contamination at a concert hall. Epidemiol. Infect. 129, 355-360. [Pg.26]

Johansen, K., Mannerqvist, K., Allard, A., Andersson, Y., Burman, L. G., Dillner, L., Hedlund, K. O., Jonsson, K., Kumlin, U., Leitner, T., Lysen, M., Thorhagen, M., et al. (2008). Norovirus strains belonging to the GII.4 genotype dominate as a cause of nosocomial outbreaks of viral gastroenteritis in Sweden 1997-2005. Arrival of new variants is associated with large nation-wide epidemics. /. Clin. Virol. 42,129-134. [Pg.29]

Lopman, B., Vennema, H., Kohli, E., Pothier, P., Sanchez, A., Negredo, A., Buesa, J., Schreier, E., Reacher, M., Brown, D., Gray, J., Iturriza, M., et al. (2004). Increase in viral gastroenteritis outbreaks in Europe and epidemic spread of new norovirus variant. Lancet 363, 682-688. [Pg.31]

Oh, D. Y., Gaedicke, G., and Schreier, E. (2003). Viral agents of acute gastroenteritis in German children Prevalence and molecular diversity. /. Med. Virol. 71, 82-93. [Pg.34]

Vivancos, R., Sundkvist, T., Barker, D., Burton, J., and Nair, P. (2010b). Effect of exclusion policy on the control of outbreaks of suspected viral gastroenteritis Analysis of outbreak investigations in care homes. Am. ]. Infect. Control 38,139-143. [Pg.38]

Werber, D., Lausevic, D., Mugosa, B., Vratnica, Z., Ivanovic-Nikolic, L., Zizic, L., Alexandre-Bird, A., Fiore, L., Ruggeri, F. M., Di Bartolo, I., Battistone, A., Gassilloud, B., et al. (2009). Massive outbreak of viral gastroenteritis associated with consumption of municipal drinking water in a European capital dty. Epidemiol. Infect. 137,1713-1720. [Pg.40]

TABLE 73-4. Agents Responsible for Acute Viral Gastroenteritis and Diarrhea... [Pg.1125]

Clark B, McKendrick M. A review of viral gastroenteritis. Curr Opin Infect Dis 2004 17 461-469. [Pg.1127]

Miscellaneous - Giddiness, influenza (more than 3%) viral gastroenteritis, urticaria, dental pain, malaise/fatigue, rash/skin eruption, dysmenorrhea (1% to 3%). [Pg.727]

Adverse reactions occurring in at least 3% of pediatric patients include abnormal gait, aggressive reaction, anorexia, ataxia, confusion, constipation, difficulty with concentration/attention, difficulty with memory, dizziness, epistaxis, fatigue, gastroenteritis, hyperkinesia, increased saliva, injury, insomnia, nausea, nervousness, personality disorder (behavior problems), pneumonia, psychomotor slowing, purpura, skin disorder, somnolence, speech disorders/related speech problems, urinary incontinence, viral infection, weight decrease. [Pg.1270]

The spectrum of gastrointestinal tract infections (GTI) cover a wide spectrum from asymptomatic Helicobacter pylori gastritis to self-limiting viral gastroenteritis to food poisoning to bacterial enterocolitis to antibiotic-associated Clostridium difficile colitis to typhoid fever with sepsis and multi-organ failure. [Pg.526]

This chapter describes the basic characteristics of viruses and the relatively limited number of drugs that can act selectively as antiviral agents. Methods of preventing viral infections (antiviral vaccines) are also briefly discussed. Finally, the current methods of treating a specific viral-induced disease—AIDS—are presented. Rehabilitation specialists often treat patients who are in the active stages of a viral infection, as well as those suffering from the sequelae of viral disorders, such as gastroenteritis, encephalitis, and influenza. Hence, the pharmacotherapeutic treatment and prophylaxis of viral infections should concern physical therapists and occupational therapists. [Pg.523]

In adults, about 70% of those with acute HAV infection develop jaundice much more commonly than with HBV or HCV. In children, acute HAV infection typically goes unrecognized and is often considered to be a viral gastroenteritis or other viral disease, since only 10% of children become jaundiced. The disease is more prolonged and serious in individuals over age 50. The specific etiological diagnosis is made with serological tests. An IgM antibody (anti-HAV IgM) appears early in the course of illness and persists for an... [Pg.1805]

Secondary lactose intolerance may occur as a result of reduced enzyme activity following diffuse intestinal damage from infections (giardiasis, bacterial overgrowth, or viral gastroenteritis), ulcerative colitis, celiac disease, and tropical sprue. This deficiency is usually reversible following recovery from the disorder. [Pg.1863]

McDonnell, S., Kirkland, K.B., and Hlady, W.G. 1997. Failure of cooking to prevent shellfish associated viral gastroenteritis. Arch. Intern. Med. 157, 111-116. [Pg.99]

Staphylococcus food poisoning Viral gastroenteritis Yersiniosis... [Pg.442]

Staphylococcus food poisoning, 215-217 Sulfur mustard poisoning, 403-406 Tetrodotoxin poisoning, 414-417 Thallium poisoning, 418-421 Trichinellosis, 222-226 Trichothecene poisoning, 422-425 Typhoid fever, 232-236 Typhus - epidemic, 237-241 Typhus - murine, 242-245 Typhus - scrub, 246-249 Viral gastroenteritis, 254-256 West Nile fever, 261-265 Yellow fever, 270-273 Yersiniosis, 274-277... [Pg.487]


See other pages where Gastroenteritis viral is mentioned: [Pg.442]    [Pg.666]    [Pg.442]    [Pg.666]    [Pg.158]    [Pg.22]    [Pg.23]    [Pg.24]    [Pg.25]    [Pg.28]    [Pg.31]    [Pg.35]    [Pg.35]    [Pg.1125]    [Pg.143]    [Pg.447]    [Pg.1270]    [Pg.30]    [Pg.89]    [Pg.95]    [Pg.144]    [Pg.591]    [Pg.219]    [Pg.224]    [Pg.434]    [Pg.547]    [Pg.258]    [Pg.472]    [Pg.665]   
See also in sourсe #XX -- [ Pg.1125 , Pg.1125 ]




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