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Disease fifth

Canine parvovirus, first identified in 1978, is now endemic.455,456 Childhood fifth disease is also caused by a parvovirus.456,457 When these single-stranded DNA viruses infect cells a double-stranded replicative form of DNA arises by synthesis of the complementary negative strand alongside the original positive DNA strand. Many copies of the replicative form are then synthesized. The negative strands of the replicative forms serve as templates for synthesis of numerous new positive strands that are incorporated into the progeny viruses. The whole process may take only 20 minutes. Some parvoviruses are unable to reproduce unless the cell is also infected by a larger adenovirus. [Pg.244]

Field flow fractionation 103 Fifth disease 244 Filaggrin 370,439... [Pg.916]

Members of the Parvovirus genus cause a number of diseases in mammals, including enteritis [37, 38] and childhood fifth disease. These nonenveloped viruses have a capsid with a diameter of 255 A that encases a double-stranded DNA genome and infect only cells that are in the S phase [39]. In canine parvovirus (GPV) the T=1 capsid is composed mainly of 60 copies of viral protein 2 (VP2). There is a small amount of VPS in the capsid that is the result of proteolytic cleavage of 17 residues from the amino terminus of VP2. Up to 20% of the 60 copies of VP2 in each capsid are replaced by VPl. VPl is the product of an alternative transcriptional splicing event that places an additional 153 amino acids at the N terminus [15]. No evidence for VPl was observed in the crystal structure [40]. [Pg.420]

Academy rash Erythema infectiosum Fifth disease Slapped cheek disease Sticker s disease... [Pg.163]

Fifth disease (a childhood rash) Viral gastroenteritis in dogs... [Pg.407]

Fifth disease A normal disease in children caused by the Erythrovirus called B19, characterized by a bright red rash on the cheeks and a low-grade fever. [Pg.1135]

Diphtheria, Tetanus, and Pertussis. These vacciaes Hi combiaatioa (DTP) have beea routiaely used for active immunization of Hifants and young children sHice the 1940s. The recommended schedule calls for immunizations at 2, 4, and 6 months of age with boosters at 18 months and 4—5 years of age. SHice 1993 these vacciaes have beea available Hi combination with a vacciae that protects agaiast Haemophilus disease, thus providing protectioa agaiast four bacterial diseases Hi oae preparatioa. A booster immunization with diphtheria and tetanus only is recommended once every 10 years after the fifth dose. [Pg.357]

Inflammatory bowel disease is most common in westernized countries such as the United States. Ulcerative colitis affects up to 500,000 people and Crohn s disease affects up to 480,000 people in the United States.2-4 The age of initial presentation of IBD is bimodal, with patients typically diagnosed between the age ranges of 20 to 40 years or 60 to 80 years.5 The peak incidence of CD occurs in the second and third decades of life, with a smaller peak in the fifth decade.2,5 Peak incidence of UC occurs between the ages of 15 and 25 years.6... [Pg.282]

The first phase of DLWC has the potential to meet more than half the annual cooling demand for the whole of Toronto s downtown, and about a fifth of the peak demand. The chilled water will cost less to produce than by conventional means, but the overwhelming advantages are environmental. As noted, the downtown will be quieter and less humid in summer because there will be no need for noisy chillers or vapour-producing cooling towers, which can also be a source of bacterial contamination and cause a form of pneumonia known as Legionnaires disease. [Pg.9]

Lemmens M, Josephs R, Schuhmacher R, Grausgruber H, Buerstmayr H, Ruckenbauer P, Neuhold G, Fidesser M and Krska R (1997), Head blight (Fusarium spp.) on wheat investigations on the relationship between disease symptoms and mycotoxin content , Fifth European Fusarium Seminar, Cereal Res. Commun. 25 (3/1), 495 -65. [Pg.387]

The main aim of these products is to produce a bread that can be eaten by those suffering from an allergy to gluten. This disease is known as coeliac disease. An estimated 1% of the British population suffer from coeliac disease, with only around one fifth being diagnosed. [Pg.191]

Motor neuron disease is characterized clinically by weakness, muscle atrophy and spasticity. This illness, often termed Lou Gehrig s disease in the United States, is the most common adult-onset form of MND with a prevalence of approximately 2-3 per 100,000 people [1-3,10, 25, 28]. Each year in the United States, in excess of 5,000 people are diagnosed with ALS. In parts of the United Kingdom, 1 in =500 deaths are attributed to some form of MND. The principal clinical signs of ALS include progressive limb weakness, which may be symmetrical or asymmetrical atrophy of appendicular, bulbar and respiratory muscles and spasticity [1,2,26,28]. The paralysis/muscle atrophy and spasticity are the result of degeneration of motor neurons in the spinal cord/brain stem and motor cortex respectively. The onset of this illness is typically in the fifth or sixth decade of life affected individuals usually... [Pg.732]

Second, we have to compare the rate of scientific progress in the AIDS epidemic with other epidemics that have had great impacts on society for example, the Black Death (plague) caused by Yersinia pestis. Plague caused a major epidemic in the fifth century and a second major epidemic in the fourteenth century. The infectious agent Yersina pestis was isolated in 1908. Effective therapy against the disease had to wait for the development of classical antibiotics in the 1940s. [Pg.238]

See the second and third paragraphs for reference to heart problems with anorexia, the fourth and fifth paragraphs for discussion of heart problems with bulimia, and the last paragraph, where heart disease is mentioned, as a risk in obese people who suffer from bingeeating disorder. [Pg.162]

Bladder cancer is the fifth most common malignancy in men. Approximately 70% of cases present with superficial disease and 30% have muscle-invasive tumors or metastatic disease. Radical surgery remains the standard treatment for invasive disease. Following cystectomy for muscle invasive bladder cancer, up to 50% of the patients will develop distant metas-tases (see Sternberg, 1995). [Pg.718]

The estimated event-free survival was 83%, and an estimated 93% of patients had not shown progression of disease to accelerated or blast phase. Contrary to expectations, there was a declining rate of treatment failure after the start of imatinib, being 3.3% in the first year, 7.5% in the second year, 4.8% in the third, 1.5% in the fourth, and only 0.9% in the fifth year. Patients who achieved a CCR by 12 months or a complete molecular response (CMR) by 18 months had risks of progression to accelerated or blast phase of 3% and 0%, respectively. The estimated overall survival rate at 5 years was 89%. An allogeneic HSCT was carried out in 44 patients, and if these patients were censored, the estimated overall survival rate was 92% (26). [Pg.133]

Four species of plasmodium typically cause human malaria Plasmodium falciparum, P vivax, P malariae, and P ovale. A fifth species, P knowlesi, is primarily a pathogen of monkeys, but has recently been recognized to cause illness, including severe disease, in humans in Asia. Although all of the latter species may cause significant illness, P falciparum is responsible for the majority of serious complications and deaths. Drug resistance is an important therapeutic problem, most notably with P... [Pg.1117]

Heterozygous carriers of functionally relevant mutations usually present with HDL cholesterol levels that are frequently below the fifth percentile. As would be expected, apoA-I levels are also frequently below the fifth percentile (i.e., < 1.05 g/1 and < 1.1 g/1 in Caucasian men and women, respectively). In most cases, heterozygous carriers of apoA-I variants do not present with specific clinical symptoms. An important exception are some structural apoA-I variants with amino acid substitutions in the amino terminus, which have been detected in patients with familial amyloidosis of the liver, the intestine, the kidney, the heart, peripheral nerves, and in the skin. In addition, some apoA-I variants like apoA-I L178P or L159P have been associated with increased risk of premature coronary heart disease or enhanced progression of carotid intima media thickness, whereas others did not show this association, or were even claimed to have reduced cardiovascular risk and advocated as possible agents for the treatment or prevention of atherosclerosis (notably apoA-I R173CMiiano) [22,43,53]. [Pg.529]


See other pages where Disease fifth is mentioned: [Pg.244]    [Pg.244]    [Pg.340]    [Pg.298]    [Pg.1372]    [Pg.1386]    [Pg.73]    [Pg.94]    [Pg.95]    [Pg.272]    [Pg.704]    [Pg.85]    [Pg.242]    [Pg.44]    [Pg.388]    [Pg.312]    [Pg.306]    [Pg.448]    [Pg.425]    [Pg.116]    [Pg.177]    [Pg.573]    [Pg.691]    [Pg.125]    [Pg.674]    [Pg.547]    [Pg.278]    [Pg.288]   
See also in sourсe #XX -- [ Pg.244 ]

See also in sourсe #XX -- [ Pg.244 ]

See also in sourсe #XX -- [ Pg.244 ]

See also in sourсe #XX -- [ Pg.244 ]

See also in sourсe #XX -- [ Pg.407 ]




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