Edema clinical presentation

A post-mortem examination and a rectal temperature of 41°C (106°F) were recorded. He had needle marks typical of intravenous drug abuse and pulmonary and cerebral edema. Abrasions and contusions of the ankles and wrists were evident from his struggling. Lidocaine was not administered to the victim during the resuscitative attempts. The clinical presentation of cocaine delirium... 

In a retrospective analysis of all cases of hyponatremia associated with ecstasy (SEDA-25, 37) at the London Centre of the National Poisons Information Service from December 1993 to March 1996,17 patients were identified with a serum sodium concentration under 130 (range 107-128 mmol/1) (96). In 10, ecstasy was identified analytically, and six of them had SIADH. The clinical presentation was very consistent, with initial vomiting and delirium, and 11 had seizures. There was complete recovery in 14, but two died of cerebral edema 5 hours after ingestion. 

Clinical presentation involves a normal postoperative course for several weeks to 6 months, followed by vision loss that is often the only symptom but may be accompanied by photophobia, injection, anterior chamber reaction, or Amsler grid distortion. There is some evidence of an increased prevalence of early age-related macular degeneration in eyes that have imdergone cataract surgery. Also, diabetic retinopathy and clinically significant diabetic macular edema may both be exacerbated by CE. Therefore the differential diagnosis of postoperative vision loss must include these conditions. 

On admission the first clinical presentation consists mainly of edema, which is of sudden onset and localized to the cervico-fadal region [33]. Dyspnoea, tachypnoea and asphyxia with chest pain following acute PPD poisoning have been reported in a number of studies [6] [12] [28]. PPD was proved to be the cause of asthmatic attacks in the sensitive individuals [46]. A case of Goodpasture s syndrome was reported to be induced by exposure to PPD [47]. Extrinsic allergic alveolitis also has been reported [48]. 

The clinical presentation of the patient is also important to consider when interpreting patterns of biochemical results. Patients with tumors that produce large amounts of epinephrine (and also metanephrine) may present with hyperglycemia, dyspnea, and pulmonary edema, signs and... 

The intoxication represents 30% of the intensive care admittance during the last 4 years. On admission the clinical presentation consist mainly in edema, important and/or sudden onset of the cervico-facial region. Facial dermatitis is observed in the case of application of the toxin (Table 1) [30]. 

II. Toxic dose. Inhalation or ingestion of as little as 1 mg of fluoroacetate is sufficient to cause serious toxicity. Death is likely after ingestion of mote than 5 mg/kg. Clinical presentation. After a delay of minutes to several hours (in one report coma was delayed 36 hours), manifestations of diffuse cellular poisoning become apparent nausea, vomiting, diarrhea, metabolic acidosis, renal failure, agitation, confusion, seizures, coma, respiratory arrest, pulmonary edema, and ventricular arrhythmias may occur. One case series reported a high incidence of hypocalcemia and hypokalemia. 

III. Clinical presentation. Exposure to moderate concentrations of phosgene causes mild cough and minimal mucous membrane irritation. After an asymptomatic interval of 30 minutes to 8 hours (depending on the duration and concentration of exposure), the victim develops dyspnea and hypoxemia. Pulmonary edema may be delayed up to 24 hours. Permanent pulmonary impairment may be a sequela of serious exposure. 

IV. Diagnosis is based on a history of exposure and the clinical presentation. Many other toxic gases may cause delayed-onset pulmonary edema (see p 7). 

The clinical presentation of acute rejection and acute infection alone is nonspecific. Manifestations can include low-grade fever, shortness of breath, nonproductive cough, and changes in measured pulmonary function. In both entities, the chest radiograph may demonstrate perihilar infiltrates, interstitial edema. 

A 56-year-old man presents to the emergency room with complaints of right lower leg pain and redness. Examining his leg, you notice that he has erythema and edema extending from his ankle to proximal tibia. The area feels warm. On questioning, the patient states that the redness started approximately 2 days ago. He has felt feverish over the previous 48 hours but did not check his temperature. He has had no other symptoms. He states that he bumped his shin on the bed frame last week and sustained a bruise but no apparent breaks in the skin. His vital signs at the clinic reveal a temperature of 38.3°C, pulse 102 beats per minute, blood pressure 11 0/72 mm Hg, and respiratory rate 20 breaths per minute. The physician diagnoses this patient with cellulitis. 

Julie Singer is a 55-year-old white woman who was admitted to the emergency department in acute distress. A previous physical examination showed hypertension and diabetes mellitus type 2. The patient s present medications include enalapril 40 mg, nifedipine 60 mg, and 100 U insulin. A physical examination revealed prominent ankle edema, a palpable spleen, and hepatomegaly. Chest radiography revealed diffuse cardiac enlargement and left ventricular hypertrophy. Based upon the history and clinical hndings, what is your diagnosis and what treatment do you recommend ... 

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