Chemical trauma

The brain is the control center of the body. Although it comprises only 2% of the body mass by weight, it uses 16% of the resting bloods supply requiring ten times the blood flow that the rest of the body needs. Because of its importance to life it is protected from physical trauma by the bony structure of the skull on the outside and from chemical trauma by the highly selective blood-brain barrier from within. 

Chemical trauma is characteristic in that the chemical product is immediately active in contact with tissue, and that its action stops when its concentration drops below its limit of action, which depends on the type of chemical reaction and the energy potential of the chemical aggressor (See Chap. 3). 

The differential diagnosis of OCP varies depending on the stage of the disease. It includes conditions that produce cicatricial changes of the ocular surface, such as chemical trauma, radiation injury, and other mucous membrane disorders. Conjunctival biopsy can aid in the diagnosis. Immunofluorescence study of the tissue demonstrates deposition of immunoreactants at the epithelial basement membrane zone in OCP. 

Intensive or extensive culture of aquatic animals requires chemicals that control disease, enhance the growth of cultured species, reduce handling trauma to organisms, improve water quality, disinfect water, and control aquatic vegetation, predaceous insects, or other nuisance organisms. The aquacultural chemical need for various species have been described for rainbow trout, Oncorhjnchus mjkiss (1) Adantic and Pacific salmon, Salmo and Oncorhjnchus sp. (2) channel catfish, Ictaluruspunctatus (3) striped bass, Morone saxatilis (4) milkfish, Chanos chanos (5) moUusks (6) penaeid (Penaeus sp.) shrimp (7) and a variety of other marine species (8). 

The precise etiology of bipolar disorder is unknown. Thought to be genetically based, bipolar disorder is influenced by a variety of factors that may enhance gene expression. These include trauma, environmental factors, anatomic abnormalities, exposure to chemicals or drugs, and others.3-5 Neurochemical abnormalities in bipolar disorder may be caused by these factors, as discussed further in the pathophysiology section. 

Prior to administering antidotes or other drugs, ensure that the signs and symptoms (e.g., coma, seizures, etc.) are due to chemical exposure and not the result of head trauma or other physical injury. 

That whole period is still very fuzzy to me. It s hard to remember what happened before the explosion and what happened after. I had such pain in my head and such hopelessness, and that sinking feeling, that I didn t want to go on at one point. How much of my emotional state was related to the trauma at work and how much was part of the chemical sensitivity I don t think anybody s qualified to answer that. 

Probably the most powerful coping technique for folks with environmental illness is similar to the coping techniques for people in trauma. That is to have some control over your own emotional response. Some have a real visceral chemical reaction that affects their emotions, I understand that. But to be empowered in some way, to make decisions on your own behalf, to take some responsibility for shaping the world you live in—it works. To take charge of what you can do, even if it s a small thing to make your life just a little bit better. 

It is likely that some or all of these mechanisms are involved in chemical sensitivity, and that disease occurs when the total load of biological, chemical, physical and psychological stressors exceeds a critical threshold for any particular individual. When the body is burdened by multiple stressors, susceptibility to MCS as well as other diseases is increased. It is not uncommon for trauma to precede or exacerbate this illness. 

In summary, this form of electrolysis is effective because the charge passing through the electrode generates chemicals inside the hair follicle. The resultant trauma kills the hair root. A leg or arm treated in this way remains hairless until a new, healthy root regrows later in the previously damaged follicle. 

Low secretion of saliva and less mucin in the produced saliva are not unusual in older individuals. Saliva has a number of protective functions of which the most important is protection against irritation by both chemical and mechanical trauma to... 

In all patients and ages BPPV can also be an effect of damage to the peripheral vestibular system by a head trauma, ear infection or ototoxicity. Ototoxity can be a result of different drugs or chemicals in the surrounding environment. 

Hexanone has also been shown to potentiate the neurotoxic effects of some compounds. In hens, dermal or inhalation exposure to 2-hexanone in combination with dermal application of the pesticide O-ethyl-O-4-nitrophenyl phenylphosphonothioate (EPN) has resulted in earlier onset and far more severe clinical and histological manifestations of neurotoxic effects than with either chemical exposure alone (Abou-Donia et al. 1985a, 1985b). The authors speculated that this potentiation effect may have been due to induction of hepatic microsomal cytochrome P-450 by EPN, leading to increased metabolism of 2-hexanone to its neurotoxic metabolite, 2,5-hexanedione. An alternate explanation is that local trauma to the nervous tissue produced by 2-hexanone and EPN might increase vascular permeability and thus increase the entry of these compounds and their metabolites from circulation. 

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