Venous malformation

Venous malformations consist of collections of venous channels and a large draining vein. Most are asymptomatic but they may present with hemorrhage into the ventricles, or seizures. On contrast CT, the draining vein may appear as a linear enhancing streak, but a flow void on MRl is more sensitive. The definitive diagnosis is made on the venous phase of a cerebral angiogram. [Pg.98]

Cardiac arrest has been reported in a child who received lauromacrogol 400 to sclerose a symptomatic peripheral venous malformation (14). [Pg.3107]

Yamaki T, Nozaki M, Sasaki K. Color duplex-guided sclerotherapy for the treatment of venous malformations. Dermatol Surg 2000 26(4) 323-8. [Pg.3108]

Chung YT, Hung DZ et al. Intracerebral hemorrhage in a young woman with arterio-venous malformation after taking diet control pills containing phenylpropanolamines case report. Chung Hua I Hsueh Tsa Chih (Taipei) 1998 61 432-5. [Pg.74]

Venous malformation CNS spinal leptomeninges Veins and gliotic or normal brain tissue no arteries... [Pg.831]

Legiehn, G. M. Heran, M. K. S. (2008) Venous malformations classification, development, diagnosis, and interventional radiologic management. Radiologic... [Pg.200]

Injection volume and concentration of sclerosant depend on the size of the lesion and the flow rate. Some authors used it as sclerosing foam hy mixing sclerosant and C02 or air [117,118] (see also Sect. 2.2.2.5.2). The German manufacture of polidocanol recommends a maximum daily dose of 2 mg/ kg [117]. Some authors described that the maximum recommended dose in the treatment of varicose veins is 6 ml of 3% polidocanol [118], This agent is associated with less severe allergic and inflammatory reactions [119]. Skin necrosis is rare. However, Cabrera reported skin necrosis in 6% of cases with venous malformations. One reversible cardiac arrest was reported [121]. [Pg.26]

Hammer FD, Boon LM, Matheurin P, Vanwijck RR (2001) Ethanol sclerotherapy of venous malformations evaluation of systemic ethanol contamination. J Vase Interv Radiol 12 595-600... [Pg.32]

Dubois J, Sebag GH, De Prost Y et al. (1991) The treatment of soft tissue venous malformation in children percutaneous sclerotherapy with Ethibloc. Radiology 180 195-198... [Pg.32]

Marrocco-Trischitta MM, Guerrini P, Aheni D, Stillo F (2002) Reversible cardiac arrest after poUdocanol sclerotherapy of peripheral venous malformation. Dermatol Surg 28 153-155... [Pg.32]

O Donovan JC, Donaldson JS, Morello FP, et al. (1997) Symptomatic hemangiomas and venous malformations in infants, children, and young adults treatment with percutaneous injection of sodium tetradecyl sulfate. AJR Am J Roentgenol 169 723-729... [Pg.32]

Choi YH, Han MH, 0-Ki K, et al. (2002) Craniofacial cavernous venous malformations percutaneous sclerotherapy with use of ethanolamine oleate. J Vase Interv Radiol 13 475-482... [Pg.34]

Konez O, Burrows PE, Mulliken JB (2002) Cervicofacial venous malformations MRI features and interventional strategies. Interv Neuroradiol 8 227-234... [Pg.34]

Observational studies Sclerotherapy with 3% sodium tetradecyl sulfate has been studied in 13 patients with venous malformations and microcystic lymphatic malformations, all low-flow malformations extending into the subcutis, confirmed... [Pg.796]

In conclusion, patients with head and neck venous malformations obviously have an increased probability of having a DVA and an increased chance of having multiple DVAs. There are no data on whether the risk of having cavernomas is also increased in this patient subgroup. [Pg.15]

Boukobza et al. (1996) found a specific pattern of DVAs in patients with extensive venous malformations of the head and neck. The draining veins were more tortuous and dilated and more often draining into the deep venous system. Additionally, the incidence of DVA seems to be increased in patients with slow-flow vascular malformations of the head... [Pg.15]

Abdulrauf SI, Kaynar MY, Awad lA (1999) A comparison of the clinical profile of cavernous malformations with and without associated venous malformations. Neurosurgery 44 41-46, discussion 46-47... [Pg.17]

Comey CH, Kondziolka D, Yonas H (1997) Regional parenchymal enhancement with mixed cavernous/venous malformations of the brain case report. J Neurosurg 86 155-158... [Pg.17]

McLaughlin MR, Kondziolka D, Flickinger JC, Lunsford S, Lunsford LD (1998) The prospective natural history of cerebral venous malformations. Neurosurgery 43 195-200, discusssion 200-201... [Pg.17]

Mullan S, Mojtahedi S, Johnson DL, Macdonald RL (1996) Cerebral venous malformation-arteriovenous malformation transition forms. J Neurosurg 85 9-13... [Pg.17]

Vascular malformations of the brain are usually divided into arteriovenous malformations, capillary telangiectasias, venous malformations, and cavernous malformations. However, for a long time, the term angiographically occult vascular malformation or cryptic (Cohen et al. 1982 Dillon 1997 Wilson 1992) has been used to describe those vascular malformations that could not be visualized angiographically, but obviously were able to cause intracerebral hemorrhage. [Pg.20]

Kim P, Castellani R, Tresser N (1996) Cerebral venous malformation complicated by spontaneous thrombosis. Childs Nerv Syst 12(3) 172-175... [Pg.48]

Korpelainen El, Karkkainen M, Gunji Y, Vikkula M, Alitalo K (1999) Endothelials receptor tyrosine kinases activate the STAT signaling pathway mutant Tie-2 causing venous malformations signals a distinct STAT activation response. Oncogene 18 1-8... [Pg.48]

Karlsson B, Lax I, Soderman M et al. (1996) Prediction of results following Gamma-Knife surgery for brain stem and other centrally located arterio-venous malformations relation to natural course. Stereotact Funct Neurosurg 66(Suppl l) 260-268... [Pg.115]

Kwon OK, Han DH, Han MH et al. (2000) Palliatively treated cerebral arterio-venous malformations follow-up results. J Clin Neurosci 7 69-72... [Pg.116]

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