Tobacco asbestos

Environmental tobacco smoke (ETS) is the diluted mixture of pollutants caused by smoking of tobacco and emitted into the indoor air by a smoker. Constituents of ETS include submicron-size particles composed of a large number of chemicals, plus a large number of gaseous pollutants. Fibers in indoor air include those of asbestos, and man-made mineral fibers such as fiberglass, and glass wool. 

Some enviromnental agents are carcinogenic tobacco, coal tar, asbestos, alcohol, some polycyclic aromatic hydrocarbons. X-rays and other sources of radiation, and many natural products. 

Asbestos exposure has affected thousands of workers and resulted in compensation claims from the companies. Of course, not smoking would result in the greatest reduction in cancers and other health-related effects of tobacco. 

Arsenic, asbestos, benzo(a)pyrene, hK(chloromethyl)ether, chromium, nickel subsulfide, zinc chromate, tobacco, mustard gas, uranium, acrylonitrile, beryllium, cadmium, l,2-dibromo-3-chloropropane, polyaromatic hydrocarbons (PAHs)... 

If you are exposed to asbestos, many factors determine whether you ll be harmed. These factors include the dose (how much), the duration (how long), the fiber type (mineral form and size distribution), and how you come in contact with it. You must also consider the other chemicals you re exposed to and your age, sex, diet, family traits, lifestyle (including whether you smoke tobacco), and state of health. 

Hnizdo E, Sluis-Cremer GK. 1988. Effect of tobacco-smoking on the presence of asbestosis at postmortem and on the reading of irregular opacities on roentgenograms in asbestos-exposed workers. Am RevRespirDis 138 1207-1212. 

Kannio A, Ridanpaa M, Koskinen H, et al. 1996. A molecular and epidemiological study on bladder cancer P53 mutations, tobacco smoking, and occupational exposure to asbestos. Cancer Epidemiol Biomarkers Prev 5 33-39. 

Muscat JE, Wynder EL. 1992. Tobacco, alcohol, asbestos, and occupational risk factors for laryngeal cancer. Cancer 69 2244-2251. 

Nelson HH, Wiencke JK, Gunn L, et al. 1998. Chromosome 3p 14 alterations in lung cancer Evidence the FHIT exon deletion is a target of tobacco carcinogens and asbestos. Cancer Res 58 1804-1807. 

Nuorva K, Makitaro R, Huhti E, et al. 1994. p53 Protein accumulation in lung carcinomas of patients exposed to asbestos and tobacco smoke. Am J Respir Crit Care Med 150 528-533. 

Prevention of exposure and cessation of any tobacco smoking activities are the most important steps that individuals can take to prevent or minimize the development of asbestos-related health problems. 

Asbestos-exposed tobacco smokers have greater than additive risks for lung cancer than do asbestos-exposed nonsmokers. 

See a/so Asbestos Combustion Toxicology Diesel Fuel Fuel Oils Pollution, Air Respiratory Tract Tobacco Smoke Volatile Organic Compounds (VOC). 

Beryllium, chromium, isocyanates, mercury, phthalic anhydride, trimellitic anhydride Asbestos, silica, metals, toluene, oxidant gases, tobacco smoke, benzene, toluene Formaldehyde, isocyanates, ethylenediamine... 

Tobacco smoke. Tobacco smoke produces particulate matter that acts as an adsorption site for toxic vapors)26 In addition to particulates, tobacco smoke produces more than 4000 individual toxic compounds, including 43 known carcinogens. 27 Many of the toxic effects of tobacco smoke that have been established empirically cannot be ascribed to individual compounds in that smoke. With more than 4000 different toxins, the number of mixtures possible is incalculable. Numerous examples of synergism between tobacco smoke and other toxicants have been identified. These include tobacco smoke and asbestos or other mineral fibers, I28 29 alcohol, I30 31 organic solvents, 32 biological... 

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