Smoking interaction

Blanc PD, Golden JA, Gamsu G, et al. 1988. Asbestos exposure-cigarette smoking interactions among shipyard workers. JAMA 259 370-373. 

Bortolasci, C.C., Vargas, H.O., Souza-Nogueira, A. et al. Lowered plasma paraoxonase (PON)l activity is a trait marker of major depression and PONl Q192R gene poly-morphism-smoking interactions differentially predict the odds of major depression and bipolar disorder. 2014 159 23-30. 

Smokers, 61 subjects (35M) whose serum cobalamin concentrations are significantly lower than those of non-smokers. Organic nitrites, nitric oxide, cyanates and isocyanates inhaled with cigarette smoke interact with vitamin B12, neutralizing it. 

Exposure to tobacco smoke from convoitional cigarettes and from so-called harm-reduction cigarettes (including secondhand smoke) is toxic to children from the time of conception and throughout development Studies have been reported that d onstrate how cigarette smoke interacts with the reproductive organs, can lead to spontaneous... 

Airborne material affecting the quality of indoor air may be classified as gases or particulate matter. Gases which may be potential problems are radon, CO, NOj, and hydrocarbons. Particulate matter may come from tobacco smoke, mold spores, animal dander, plant spores, and others as shown in Table 23-1. Other factors interact to influence our perception of indoor air quality, including humidity, temperature, lighting, and sound level. 

Interaction with sprinklers The likely size of fire will be smaller in buildings with sprinklers and the temperature of the smoke produced will be lower since the smoke is cooled by the sprinkler flow. 

COPD is a chronic inflammatory disease that results from prolonged and repeated inhalation of particles and gases, chronic (or latent) infection or an interaction of these factors. In many cases, the inflammation persists even when the exposure (in most cases smoking) is stopped. Prominent among the infiltrating leukocytes are neutrophils, CD8+ lymphocytes (Co-receptor for the T-cell receptor. CD8+ is specific for the class IMHC protein. It is expressed on the surface of cytotoxic T-cells and natural killer cells.) and CD68+ monocytic cells (A lysosomal antigen. All cells that rich in... 

The proplnts described above are in the realm of prior art and depict those NC proplnts with low smoke potential that are used primarily as gun proplnts. Recent research and development work has been concentrated on creating both gun proplnts and rocket proplnts with reduced smoke output in order to foil countermeasures. Lavitt (Ref 76) found that the concurrent use of optimum proportions of lead stearate and sodium barbiturate in double-base proplnts resulted in a marked reduction in smoke output. This was attributed to the synergistic interaction of the two salts to produce more complete oxidation of the exhaust products. The importance of using the optimum ratio of the two catalysts is demonstrated by the higher smoke values shown in Table 4 for Propellants 105, 106 and 107, when compared to other... 

Whereas smokes are often characterized by the mode of their formation, any coned suspension of particles ranging from 0.01 to about 5.0 pm can be considered a smoke. These particles, when suspended in air, reflect, scatter and absorb radiation in a complex manner. A comprehensive model of these interactions in terms of visibility under various conditions was described recently (Ref 62)... 

Leanderson, P. and Tagesson, C. (1992). Cigarette smoke-induced DNA damage in cultured human lung cells role of hydroxyl radicals and endonuclease activation. Chem. Biol. Interact. 81, 197—208. 

Similar interactions as clozapine may need to increase the usual olanzapine dose when a patient begins to smoke, or decrease olanzapine dose if smoking is stopped or nicotine replacement is used instead of smoking. 

Hasenfratz, M., Jaquet, F., Aeschbach, D., and Battig, K., Interactions of smoking and lunch with the effects of caffeine on cardiovascular functions and information processing. Human Psychopharmacology Clinical and Experimental 6(4), 277-284, 1991. 

Schein, J. R. (1995). Cigarette smoking and clinically significant drug interactions. Ann. Phar-macother., 29(11), 1139-48. 

Zevin, S. Benowitz, N. L. (1999). Drug interactions with tobacco smoking. An update. Clin. Pharmacokinet., 36(6), 425-38. 

The interaction of carotenoids with cigarette smoke has become a subject of interest since the results of the Alpha-Tocopherol Beta-Carotene Cancer Prevention Study Group 1994 (ATBC) and CARET (Omenn et al. 1996) studies were released. P-Carotene has been hypothesized to promote lung carcinogenesis by acting as a prooxidant in the smoke-exposed lung. Thus, the autoxidation of P-carotene in the presence of cigarette smoke was studied in model systems (toluene) (Baker et al. 1999). The major product was identified as 4-nitro-P-carotene, but apocarotenals and P-carotene epoxides were also encountered. 

Ling, D., Niu, T., Feng, Y., Xing, H., and Xu, X., Association between polymorphism of the dopamine transporter gene and early smoking onset an interaction risk on nicotine dependence, J. Hum. Genet., 49, 35, 2004. 

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