Scars corticosteroids

Topical corticosteroids are employed in some cases of bacterial keratitis. The suppression of inflammation may reduce corneal scarring. However, local immunosuppression, increased ocular pressure, and reappearance of the infection are disadvantages to their use. There is no conclusive evidence that they alter clinical outcomes. If the patient is already on topical corticosteroids when the keratitis occurs, discontinue use until the infection is eliminated.19... 

Non-alcohol steatohepatitis Fat build-up in the liver eventually causes scar tissue associated with diabetes, protein malnutrition, obesity and coronary artery disease. Treatment with corticosteroid medications. Biopsy is needed for full diagnosis. 

Scarring after anterior chemabrasion, resulting from an irregular abrasion and supposed poor follow-up. Treatment applied used topical and injected corticosteroids, antityrosinase and antioxidant cream, silicone sheet compression. 

Scabs should not be pulled off in the post-peel period. Aggressive use of OT can cause scarring and/or pigmentation problems. Intense application of OT can cause patches of erythema that can persist for up to 3 months. This prolonged erythema can be treated by the application of topical corticosteroids and total sunblock. 

Corticosteroids by the systemic route will only be needed very rarely to treat erythema. They can be administered in a single and preventive injection at the start of a phenol peel. Promethazine can be recommended, at a maximum dose of six times 25 mg/day, when the erythema causes pruritus and reflex scratching that could lead to unsightly scars, infections or dyschromia. 

Location the corticosteroids must be injected into the scar tissue itself and not in the tissue surrounding the scar. Not only is an injection underneath or beside the scar ineffective, it can also cause fat atrophy underneath or beside the scar that would make it more obvious. 

B. Specific drugs and antidotes. For most agents, there Is no specific antidote. (See p 221 for hydrofluoric acid bums and p 302 for phenol bums.) In the past, corticosteroids were used by many clinicians in the hope of reducing scarring, but this treatment has been proved ineffective. Moreover, steroids may be harmful in the patient with perforation because they mask early signs of inflammation and inhibit resistance to infection. 

Persistent erythema, due to angiogenic factors stimulating vasodilatation, is considered a physiological event if it occurs within 3 weeks after the peel procedure [6]. If erythema associated with pruritus persists for more than 3 weeks, treatment with potent topical corticosteroids, systemic corticosteroids, and/or intralesional corticosteroids should be administered. Silicone sheets or pulsating dye laser treatments may be also adopted, especially when evident thickening or scarring has occurred [6, 15]. 

Cladophiatophora bandana in a scar after treatment with intralesional corticosteroid injections. J Am Acad Dermatol 2009 61(3) 537-8. 

Patches of erythema persisting for more than 3 weeks is indicative of early hypertrophic scar formation and should be treated with jxjtent topical corticosteroid therapy. 

Figure 15.16 This patient, who had received radiation tor lymphoma, developed Intense erythema several weeks after a Baker s peel. Mild induration was noted at examination class I corticosteroids ettected a scar-free healing...

If I detect induration during the postoperative exam, I immediately institute treatment with class 1 topical steroids. I will often provide samples of these agents to patients to limit their use and ensure the patient will return. When hypertrophic scarring does occur, intrale-sional corticosteroids [triamcinolone acetonide 2.5-10 mg per mL) and Cordran tape used nightly are employed. It is interesting to note that dermabrasions and lasers produce scars in the same anatomical areas as deeper chemical peels (Figs 15.21 and 15.22). 

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