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Salicylic acids induced

Alteration of Membrane Permeability. The ability of allelochemicals to alter membrane permeability and thus inhibit mineral absorption has been investigated in detail with only phenolic acids. Salicylic acid induced the efflux of PO5 (28) and 1C" (42) from barley roots, but -hydroxybenzoic acid did not cause the efflux of K+... [Pg.173]

SIPK Salicylic acid-induced protein kinase... [Pg.21]

Kawano T, Sahashi N, Takahashi K, Uozumi N, Muto S. Salicylic acid induces extracellular superoxide generation followed by an increase in cytosolic calcium ion in tobacco suspension culture The earliest events in salicylic acid signal transduction. Plant Cell Physiol 1998 39 721-30. [Pg.30]

Kawano T, Muto S. Mechanism of peroxidase actions for salicylic acid-induced generation of active oxygen species and an increase in cytosolic calcium in tobacco suspension culture. J Experi Bot2000 51 685-693. [Pg.228]

SIPK = Salicylic Acid Induced Protein Kinase... [Pg.952]

Fig. 1. Salicylic acid and its synthetic functional analogs. Salicylic acid and its derivative acetyl salicylic acid (aspirin) induce expression of the PR genes and enhance resistance to pathogens. The two synthetic compounds, 2,6-dichloroisonicotinic acid (INA) and benzothiadiazole S-methyl ester (BTH) show structural similarities to salicylic acid and like salicylic acid induce expression of the PR genes and enhance resistance to pathogens. Fig. 1. Salicylic acid and its synthetic functional analogs. Salicylic acid and its derivative acetyl salicylic acid (aspirin) induce expression of the PR genes and enhance resistance to pathogens. The two synthetic compounds, 2,6-dichloroisonicotinic acid (INA) and benzothiadiazole S-methyl ester (BTH) show structural similarities to salicylic acid and like salicylic acid induce expression of the PR genes and enhance resistance to pathogens.
Senaratna, T., D. Touchell, E. Bunn, and K. Dixon. 2000. Acetyl salicylic acid (Aspirin) and salicylic acid induce multiple stress tolerance in bean and tomato plants. Plant Growth Regul. 30 157-161. [Pg.85]

Krajnc AU, Kristi J, Ivancic A (2011) Application of salicylic acid induces antioxidant defense responses in the phloem of Picea abies and inhibits colmization by Ips typographus. Forest Ecol Manag 261(3) 416 26. doi 10.1016/J.Foreco.2010.10.027... [Pg.4057]

Horvath, D M. and Chua, N.H. (1996) Identification of an immediate-early salicylic acid-inducible tobacco gene and characterization of induction by other compounds. Plant Mol Biol 31, 1061-1072... [Pg.225]

Kiddle, G.A., Kevin, J.D. and Wallsgrove, R.M. (1994) Salicylic acid-induced accumulation of glucosinolates in oilseed rape (Brassica napus) leaves. /. Exp. Bot., 45,1343-1346. [Pg.43]

Jessner s Solution has been used for over 100 years as a therapeutic agent to treat hyperkera-totic epidermal lesions [1]. This superficial peeling agent constitutes a mixture of salicylic acid, resorcinol, and lactic acid in 95% ethanol. Jessner s solution causes loss of corneocyte cohesion and induces intercellular and intracellular edema. Jessner s typically induces wounding to the level of the papillary dermis. Historically, resorcinol (a key component of Jessner s peels) was used in concentrations of 10-50% in the early twentieth century. High concentrations of resorcinol were associated with side effects such as allergic contact dermatitis, irritant contact... [Pg.23]

Salicylic acid (ortho hydroxybenzoic add) is a beta hydroxy acid agent. It is a lipophilic compound which produces desquamation of the stratum corneum via removal of intercellular lipids [3] (see salicylic acid section). Given its keratolytic effects, it has become an increasingly popular superficial peeling agent. Salicylic acid peels induce injury via thinning or removal of the stratum corneum. In addition, salicylic acid potentially enhances the penetration of TCA. [Pg.103]

Trichloroacetic Acid (TCA) causes precipitation of proteins and coagulative necrosis of epidermal cells [4]. The extent of damage is indeed concentration dependent. Concentrations range from 10 to 50%. Superficial TCA peeling is induced by concentrations of 10-30% whereas higher concentrations cause medium depth or deep peeling. The combination of salicylic acid followed by TCA 10-15% induces superficial wounding. [Pg.103]

Jessner s solution contains 14% resorcinol, 14% salicylic acid and 14% lactic acid. Jessner s solution has been used alone for superficial peeling, or in combination with TCA 35% to achieve a medium-depth peel. Increasing the number of coats applied to the treated area increases the depth and reaction induced by the Jessner s peel. These peels are well tolerated with minimal side effects in the author s practice. As with glycolic acid and salicylic acid peels, Jessner s peels are most commonly used as adjimctive therapy for moderate to severe facial dyschromias, acne, oily skin, texturally rough skin, fine wrinkles, and pseudofolliculitis barbae. [Pg.144]

Salicylic acid penetrates more deeply in the areas of inflammation and produces rapid drying of papules and pustules 1 or 2 days after the peeling. These beneficial effects are determined by the antimicrobic activity of salicylic acid, which also stimulates fibroblasts, inducing an improvement of the vascular component of rosacea. Application of salicylic acid in inflammatory rosacea is equally as safe and effective in residual or initial lesions. [Pg.193]

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