Recovery phases

FIGURE 5.24 Components of ciliary movement, (a) Power and recovery phases of ciliary movement. Arrows indicate the direction of ciliary travel, (b) Net mucociliary transport. Dotted arrows show the direction of cilia while the solid arrows show mucus transport. Note that net gel movement is forward in I and III while no gel movement occurs in II during the cilia recovery phase. Modified from Ful-ford and Blake. ... 

Ciliary beat recovery phase Interval during which cilia bends and returns... 

When levels of conjugated bilirubin remain high in plasma, a fraction can bind covalently to albumin (delta bilirubin). Because it is bound covalently to albumin, this fraction has a longer half-life in plasma than does conventional conjugated bilirubin. Thus, it remains elevated during the recovery phase of obstructive jaundice after the remainder of the conjugated bilirubin has declined to normal levels this explains why some patients continue to appear jaundiced after conjugated bilirubin levels have returned to normal. 

In subacute thyroiditis, thyroid function tests typically run a triphasic course in this self-limited disease. Initially, serum T4 levels are elevated due to release of preformed thyroid hormone from disrupted follicles. The 24-hour RAIU during this time is less than 2% because of thyroid inflammation and TSH suppression by the elevated T4 level. As the disease progresses, intrathyroidal hormone stores are depleted, and the patient may become mildly hypothyroid with an appropriately elevated TSH level. During the recovery phase, thyroid hormone stores are replenished and serum TSH elevation gradually returns to normal. 

Pyle SJ, Amamath V, Graham DG et al. 1993. Decreased levels of the high molecular weight subunit of neurofilaments and accelerated neurofilament transport during the recovery phase of 2,5-hexanedione exposure. Cell Motil Cytoskeleton 26(2) 133-143. 

Operational costs remained constant at 1500 barrels per month from initiation to completion of the recovery phase. 

Under standard conditions, this reaction would be unfavourable but physiological conditions during recovery phase after exercise are such as to allow creatine phosphate formation to occur. 

No specific antidote has been shown to be effective in treating 1,2-dibromoethane intoxication once absorption into the bloodstream has occurred (Ellenhorn and Barceloux 1988). Intravenous infusions of glucose may limit the hepatotoxicity of 1,2-dibromoethane (ERA 1989b). During the recovery phase, a diet rich in vitamin B and carbohydrates may limit liver damage (Dreisbach and Robertson 1987 Lawrence and Michaels 1984). Hemodialysis may be needed to regulate extracellular fluid and electrolyte balance and to remove metabolic waste products if renal failure occurs (ERA 1989b). 

Fig. 37. Stacked plot of spectra recorded from the lower leg of a 35-year-old male volunteer. Spectra were recorded each 15 s during exhaustive exercise inside the imager (spectra 1 to 5) and in the recovery phase after exercise (spectra 6 to 16). Four acquisitions were recorded for each FID with TR = 3 s.

Hypothyroidism As replacement or supplemental therapy in hypothyroidism of any etiology, except transient hypothyroidism during the recovery phase of subacute thyroiditis. 

Prior sensitivity to any tricyclic drug. Not recommended for use during the acute recovery phase following myocardial infarction. Concomitant use of monoamine oxidase inhibitors (MAOIs) is generally contraindicated. 

Preexisting cardiac disease Not recommended for use during the initial recovery phase of Ml. Trazodone may be arrhythmogenic in some patients. 

Hypersensitivity to cyclobenzaprine concomitant use of monoamine oxidase (MAO) inhibitors or within 14 days after their discontinuation acute recovery phase of Ml and in patients with arrhythmias, heart block, or conduction disturbances or CHF hyperthyroidism. 

HV166 Delaney B., T. Carlson, G. H. Zheng, et al. Repeated dose oral toxicological evaluation of concentrated barley beta-glucan in CD-I mice including a recovery phase. Food Chem Toxicol 2003 41(8) 1089-1102. 

Contraindications Acuf e recovery phase of MI, arrhythmias, CHF, heart block, conduction disturbances, hyperthyroidism, use within 14 days of MAOIs... 

It appears that a number of complications await the recovering bipolar patient after an episode of mania. For example, Lucas et al. ( 44) reported on a retrospective linear discriminant analysis of 100 manic episodes (1981 to 1985) during the recovery phase and found that the incidence of subsequent depression was 30% in the first month. Many episodes were transient, however, and did not necessarily require treatment. This phenomenon could be successfully predicted in 81% of cases in which there is a premorbid history of cyclothymia with either a personal or a family history of depression. The highly significant association between family history and postmanic depression again supports the hypothesis of a genetic basis for bipolar disorder. 

Diffusion hypoxia describes the rapid movement of nitrous oxide from the blood into the alveoli in the early recovery phase. It is usual to circumvent this problem by administering 100% oxygen for a few minutes at the end of anaesthesia. 

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