Mercury poisoning caused

Mercury and its compounds were used for centuries as ingredients in antibacterial drugs, skin creams, and antiseptics. Mercury compounds are quite toxic, however. In the body, mercury combines with the thiol groups of critical enzymes, inactivating them. Mercury poisoning causes brain and kidney damage, often leading to death. 

This event occurred in the early 1950s and was the result of mercury poisoning caused by effluent from a factory nearby. It was one of the world s worst environmental pollution disasters. 

Metallic mercury was also the source of two cases of mercury poisoning caused by the dermal application of an over-the-counter anti-lice product (Bourgeois et al. 1986). The more severely poisoned individual... 

Harada, M. (1982). Minamata disease organic mercury poisoning caused by ingestion of contaminated fish. In Adverse Effects of Food, Jellife, E.F. and Jellife, D.B. (eds), pp. 135— 148. Plenum Press, New York. 

The first is the story of the high school physics teacher who came down with a strange illness eventually diagnosed as mercury poisoning. The cause Over the years, minute amounts of this metal had been spilled in the poorly ventilated lecture room. The students suffered no ill effects since they spent so little time there. Fortunately, the teacher recovered, the room was completely refurbished, and strict rules were introduced concerning the handling of mercury. 

Basic toxicity has been identified from careful observation and experimentation in the workplace and in the lab. Over the years, from the time of the ancient Greeks and Romans, and probably long before that, we have learned that exposure to certain substances can cause bodily harm. Hippocrates, the founder of medicine in Ancient Greece, described the occurrence of lead poisoning among lead miners and metal workers as long ago as 400 B.C. The Roman historian, Pliny, described in his encyclopedia in the second half of the first century A.D., the dangers of mercury poison-... 

Most cases of mercury poisoning led to handicap, chronic disease, or death. The most frequent symptoms include numbness of limbs, lips and tongue, speech abnormalities, limb function disorders, visual acuity disorders, deafness, and muscular atrophy. Insomnia, hyperactivity, and coma have also been reported. Methylmercury penetrates the blood-brain barrier and causes central nervous system injuries. Mercury also has a teratogenic effect, leading to congenital abnormalities or congenital Minamata disease. 

Large increases in mercury levels in water can be caused by industrial and agricultural use and waste releases. The health risk from mercury is greater from mercury in hsh than simply from water-borne mercury. Mercury poisoning may be acute, in large doses, or chronic, from lower doses taken over an extended time period. The maximum amount of mercury allowed in drinking water by the standard is 0.002 mg/L of water. That level is 13% of the total allowable daily dietary intake of mercury. 

A strange disease was reported in the area around Minamata Bay in Japan. Forty-six people died and many more became ill. The illness was due to mercury poisoning from a plastics factory. The Chisso Corporation used mercury as a catalyst in making acetaldehyde. Dimethylmercury becomes concentrated up the food chain and the heavy reliance on food from the sea life in the bay caused the epidemic. Mercury became a source of worry in many U.S. rivers and has been monitored closely since then. Chisso was finally found guilty in 1973, and 300 people had died by 1980. 

Mercury Fulminate Poisoning has symptoms of mercury poisoning. At low levels causes dermatitis and irritation of the mucous membranes Avoid all routes of exposure. Tolerance level below 0.01 mg/m3... 

The kidney is the primary target organ for Hg2+. Chronic exposure to inorganic mercury ) compounds causes proteinuria. In cases of mercury poisoning of any type, the kidney is the organ with the highest bioaccumulation of mercury. 

A case of Minamata disease caused by the methyl mercury discharged by a chemical factory in Japan provides us with bitter lessons. Inappropriate treatments of toxic chemical substances for the duration of 1932 to 1968 resulted in creating and torturing more than 14,000 victims with a neurological syndrome by severe mercury poisoning. It has been fifty years since the disease was official discovered in 1956. The case is not yet fully solved and lawsuits and claims for compensations continue even to this day. 

Mercury (Hg) is a toxic metal—it is one of the so-called heavy metals. Hg is a neurotoxin that causes damage to the central nervous system (CNS), which consists of the brain and associated parts. Hg is active at about 50pg/100mL of blood (500 ppb). Central nervous system damage manifests itself as quarrelsome behavior, headaches, depression, and muscle tremors. The classic example of mercury poisoning is the mad hatter, caused by mercury exposure during the felt-making process. 

Poisoning by bismuth and bismuth compounds has occurred more frequently during medical therapy than by exposure at workplaces. It resembles poisoning caused by lead and mercury and their compounds. After oral administration, water-insoluble bismuth compounds, such as bismuth nitrate oxide, BiO(N03), are hardly absorbed, and acute poisoning is seldom induced. Water-soluble bismuth compounds are absorbed quickly, and acute poisoning is likely to occur. 

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