Organic mercury poisoning

Parathion poisoning in Columbia, Egypt, Iran, Malaysia, Mexico Organic mercury poisoning in seed grains (Iraq, 1972)... 

Iraq Organic mercury poisoning in seed grains ... 

Miyakawa T, Murayama E, Sumiyoshi S, et al. 1976. Late changes in human sural nerves in Minamata disease and in nerves of rats with experimental organic mercury poisoning. Acta Neuropath (Berlin) 35 131-138. 

Harada, M. (1982). Minamata disease organic mercury poisoning caused by ingestion of contaminated fish. In Adverse Effects of Food, Jellife, E.F. and Jellife, D.B. (eds), pp. 135— 148. Plenum Press, New York. 

Yoshino, Y., T. Mozai, and K. Nakao. 1966. Distribution of mercury in the brain and its subcellular units in experimental organic mercury poisoning. J. Neurochem. 13 397-406. 

Uchino, M., T. Okajima, K. Eto, T. Kumamoto, I. Mishima, and M. Ando. 1995. Neurologic features of chronic Minamata disease (organic mercury poisoning) certified at autopsy. Intern. Med. 34(8) 744-7. 

Brenner RP, Snyder RD Late EEC findings and clinical status after organic mercury poisoning. Arch Neurol 37 282-284,1980... 

Table 17.8 (which is modified from Rustam and Hamdi, 1974), indicates the relative frequency of neurological signs observed in 53 patients with confirmed organic mercury poisoning. Recovery occurred only in the mildly to moderately poisoned patients. 

Describe the major clinical features and treatment of inorganic and organic mercury poisoning. Describe the major clinical features and treatment of iron poisoning. 

Fujita, E. (1969) Experimental studies of organic mercury poisoning. The behavior of the Minamata disease causing agent in maternal bodies, and its transfer to their infants via either placenta or breast milk. J. Kumamoto Med. Soc. 43, 47. 

Matsumoto, H., Kameda, T. and Takeuchi, T. (1969) Pathological studies on organic mercury poisoning supplement to histochem-ical demonstration of mercury in the brain of Minamata disease. Adv. Neurol ScL 13, 270-278. 

Miura, K., Suzuki, K. and Imura, N. (1978) Effects of methylmercuiy on mitotic mouse glioma cells. Environ. Res. 17, 453-471. Morikawa, N. (1961) Pathological studies on organic mercury poisoning. Kumamoto Med. J. 14, 87-93. 

If a pregnant woman is affected by mercury poisoning, the consequences may affect the child. As a result, the child may suffer from profound mental deficiency, atrophy of cerebral cortex, commisure and cerebellum neuron destruction. Acrodynia is a syndrome that affects children exposed to organic and inorganic mercury compounds. Symptoms include itchy, measles-like rash followed by desquamation of palm and foot skin, essential tachycardia, generalized swellings, hypertension and salivation (Harada, 1995). 

Contraindications Acute renal impairment, organic (short-chain alkyl) mercury poisoning, G6PD deficiency (unless a life-threatening situation exists), hepatic insufficiency (unless due to arsenic poisoning), use of iron, cadmium or selenium poisoning, hypersensitivity to dimercaprol or any component of the formulations... 

The scientific community provided much of the original impetus for improved environmental protection in Sweden. Swedish scientists, for instance, focused attention on the health hazards from organic mercury compounds used in agriculture several years before the Minimata poisonings in Japan brought those problems to the attention of the rest of the world.1 The Swedish chemist Soren Jensen was the first to describe the bioaccumulation properties of PCBs, and the pioneering radiation and biochemical re-... 

The kidney is the primary target organ for Hg2+. Chronic exposure to inorganic mercury ) compounds causes proteinuria. In cases of mercury poisoning of any type, the kidney is the organ with the highest bioaccumulation of mercury. 

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