Lead poisoning history

Water-pipes—Materials—History. 2. Lead—Environmental aspects— History. 3. Lead—Toxicology—History. 4. Drinking water—Lead content— History. 5. Pipe, Lead—History. 6. Lead poisoning—History. I. Title. 

As in 1925, when dozens of workers went insane from tetraethyl lead poisoning, Kehoe was the Ethyl Corporation s key man at a hearing. Patterson was a key critic, and corporate executives looked to Kehoe to give them the ten years they needed. According to the corporation s own official history, Kehoe had the fate of the company in his hands. If he wavered, the company would have been faced with disaster. ... 

Hu-Howard. Knowledge of Diagnosis and Reproductive History among Survivors of Childhood Plumbism. American Journal of Public Health. 81 (Aug. 1991) 1070-1072. Source for third-generation effects of lead poisoning. 

Warren C (2000) Brush with Death A Social History of Lead Poisoning. Baltimore, MD Johns Hopkins University Press. 

Markowitz and Rosner, Deceit and Denial, pp. 18—19 Kovarik, Context of Technological Alternatives C. Warren, Brush with Death A Social History of Lead Poisoning (Johns Hopkins University Press, Baltimore, 2.000), pp. 120-123. 

Markowitz and Rosner, Deceit and Denial, p. 35 D. Davis, Secret History of the War on Cancer (Basic Books, New York, 2007), pp. 79, 94-95 J. O. Nriagu, Clair Patterson and Robert Kehoe s Paradigm of Show Me the Data on Environmental Lead Poisoning, Environmental Research, vol. A78, pp. 71—78 (1998) A. P. Loeb, Paradigms Lost A Case Study Analysis of Models of Corporate Responsibility for... 

Lin-fu JS Modern history of lead poisoning a century of discovery and rediscovery, in Human Lead Exposure. Edited by Needleman HL. Boca Raton, FL, CRC Press,... 

Treatment of lead-poisoned animals usually involves the removal of ingested lead objects and application of antibiotics. For example, a captive bottlenose dolphin that had 40 lead-containing air pellets in its second stomach, as determined by radiography, was treated with 250.0 mg penicillamine/kg BW given orally three times daily for 5 days after the pellets had been removed from the stomach using an endoscope. Anemia in chimpanzees is sometimes associated with lead toxicity. In one case, a 19-year-old female chimpanzee with a history of excessive menstrual bleeding had a blood serum level of 1.03 mg Pb/L. The animal was successfully treated using oral chelation therapy 2,3-dimercaptosuccinic acid at 10.0 mg/kg BW per os for 5 days, then lO.Omg/kg BW for 2 weeks. 

HISTORY OF PUBLIC POLICY ADDRESSING LEAD POISONING... 

Section based on Richard Rabin, Warnings Unheeded A History of Child Lead Poisoning, American Journal of Public Health 79 (1989) 1668-1674. Citations for laws are in Appendix H. 

Publishes "Hour of Lead A Brief History of Lead Poisoning in the United States and the Efforts of the Lead Industry to Delay Regulation. ... 

Ms. Kessel and Mr. O Connor have done an admirable job of condensing into a relatively short book a large amount of basic information about childhood lead poisoning. Interspersed with case histories, they have outlined in a straightforward language the full dimensions of the problem facing our country. 

This document outlines the medical monitoring program as defined by the occupational safety and health standard for inorganic lead. It reviews the adverse health effects of lead poisoning and describes the important elements of the history and physical examinations as they relate to these adverse effects. Finally, the appropriate laboratory testing for evaluating lead exposure and toxicity is presented. 

Multiauthor compendia discussing aspects of lead history include expert consensus treatises such as those of the U.S. EPA (1986), the U.S. ATSDR (1988), the various statements on childhood lead poisoning by the U.S. CDC (1985, 1991, 2005), the U.S. NAS/NRC (1972, 1980, 1993), and the WHO (1995). Two comparative historical and scientific analyses by Mushak have also appeared (Mushak, 1992 Mushak and Mushak, 2000), ranking or otherwise comparing lead as an economic and public health issue with environmental contaminants such as other metals, notably comparisons of lead to mercury as the highly neurotoxic methyhnercury form (Mushak and Mushak, 2000). 

Second, the requirement of severe Pb exposures in mothers as necessary for lead reproductive and developmental toxicity is not a tenable premise. As noted in later chapters, embryo- and fetotoxicity is quite a sensitive endpoint in humans and occurs at relatively low systemic lead levels owing to ready transplacental movement of lead early in pregnancy (NAS/NRC, 1993 U.S. ATSDR, 1988 U.S. CDC, 1985, 1991, 2005 U.S. EPA, 1986, 2006). The occupational history of lead in the late nineteenth and early twentieth centuries establishes various adverse reproductive outcomes in pregnant women not themselves demonstrably debilitated by manifest lead poisoning (Legge, 1901 Oliver, 1911 U.S. EPA, 1986, 2006). 

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