Intracranial multiple aneurysms

Yasargil MG (1984) Multiple aneurysms in microneurosurgery, vol 1. Thieme, Stuttgart, pp 305-328 Yasui N, Suzuki A, Nishimura H, Suzuki K, Abe T (1997) Long-term follow-up study of unruptured intracranial aneurysms. Neurosurgery 40 1155-1159 discussion 1159-1160... 

Ruby RJ, Burton JR (1977). Acute reversible hemiparesis and hyponatremia. Lancet i 1212 Sabolek M, Bachus-Banaschak K, Bachus R et al. (2005). Multiple cerebral aneurysms as delayed complication of left cardiac myxoma a case report and review. Acta Neurology Scandinavica 111 345-350 Sacco RL, Kargman DE, Gu Q et al. (1995). Race-ethnicity and determinants of intracranial atherosclerotic cerebral infarction. The Northern Manhattan Stroke Study. Stroke 26 14-20 Salgado AV (1991). Central nervous system complications of infective endocarditis. 

Arteriovenous malformations present most commonly with signs consistent with a space-occupying lesion or seizures and consist of an abnormal fistulous connection(s) between one or more hypertrophied feeding arteries and dilated draining veins (Clatterbuck et al. 2005) (Fig. 7.5). The blood supply is derived from one cerebral artery or, more often, several, sometimes with a contribution from branches of the external carotid artery. Arteriovenous malformations vary from a few millimeters to several centimeters in diameter. Approximately 15% are associated with aneurysms on their feeding arteries. Some grow during life but a few shrink or even disappear, and some are multiple. These fistulae occur in or on the brain, or in the dura of the intracranial sinuses. 

Structural intracranial lesions tumor, chronic subdural hematoma, vascular malformation, giant aneurysm Multiple sclerosis... 

SMP foams have been proposed as a treatment for intracranial aneurysms. An SMP foam would allow for the placement of a single compacted device that would gently expand to fill the entire aneurysm (Fig. 5). This would potentially reduce the complexity of multiple coil placement, avoid coil-induced rupture, decrease surgery time, and allow for a porous matrix for cell invasion and neointima formation to seal the neck of the aneurysm. 

SAH in sickle cell anemia is characterized by multiple hemorrhages, often distally and in unusual locations. CT scan demonstrates blood in the superficial cortical sulci. Angiography reveals multiple distal branch occlusions and a collateral circulation via leptomeningeal vessels. SAH is attributed to rupture of these leptomeningeal collaterals, the outcome is usually poor (Carey et al. 1990). Approximately 30% of patients with sickle cell disease and SAH are children. Some patients with sickle cell disease harbour intracranial aneurysms which maybe another cause for SAH (Vicari et al. 2004). 

Inagawa T (1991) Surgical treatment of multiple intracranial aneurysms. Acta Neurochir 108 22-29 Inagawa T, Hirano A (1990) Autopsy study of unruptured incidental intracranial aneurysms. Surg Neurol 34 361-365... 

Lee EK, Hecht ST, Lie JT (1998) Multiple intracranial and systemic aneurysms associated with infantile- onset arterial fibromuscular dysplasia. Neurology 50 828-829 Lee KC, Joo JY, Lee KS, Shin YS (1999) Recanalization of completely thrombosed giant aneurysm case report. Surg Neurol 51 94-98... 

Nehls DG, Flom RA, Carter LP, Spetzler RF (1985) Multiple intracranial aneurysms determining the site of rupture. J Neurosurg 63 342-348... 

Rinne J, Hernesniemi J, Puranen M, Saari T (1994) Multiple intracranial aneurysms in a defined population prospective angiographic and clinical study. Neurosurgery... 

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