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Feeding artery

Fig. 6.6. Signal-time curves determined in normal tissue and in the feeding arteries (AIF). The signal drop in the arteries is much larger than in tissue... Fig. 6.6. Signal-time curves determined in normal tissue and in the feeding arteries (AIF). The signal drop in the arteries is much larger than in tissue...
Arteriovenous malformations present most commonly with signs consistent with a space-occupying lesion or seizures and consist of an abnormal fistulous connection(s) between one or more hypertrophied feeding arteries and dilated draining veins (Clatterbuck et al. 2005) (Fig. 7.5). The blood supply is derived from one cerebral artery or, more often, several, sometimes with a contribution from branches of the external carotid artery. Arteriovenous malformations vary from a few millimeters to several centimeters in diameter. Approximately 15% are associated with aneurysms on their feeding arteries. Some grow during life but a few shrink or even disappear, and some are multiple. These fistulae occur in or on the brain, or in the dura of the intracranial sinuses. [Pg.97]

Transarterial embolization (TAE) By occluding the smaller tumour-feeding arteries, it is possible to achieve a hypoxia-induced necrosis of the HCC. This is more successful if the tumour is encapsulated. Such embolization can be carried out using collagen particles, polyvinyl alcohol, gelfoam or galactose spheres. However,... [Pg.784]

Fig. 8.3 Idealized and actual perfusion experiments. In aU of the above graphs, time is depicted on the x axis and concentration of contrast material on the y-axis. In an idealized perfusion experiment, the entirety of a bolus of contrast material would be delivered instantaneously to brain tissue via a feeding artery. This is depicted in the arterial input function in the upper left hand comer. In this case, the tissue concentration-vs.-time curve (upper right) is a residue function, reflecting the amormt of contrast bolus remaining in the tissue... Fig. 8.3 Idealized and actual perfusion experiments. In aU of the above graphs, time is depicted on the x axis and concentration of contrast material on the y-axis. In an idealized perfusion experiment, the entirety of a bolus of contrast material would be delivered instantaneously to brain tissue via a feeding artery. This is depicted in the arterial input function in the upper left hand comer. In this case, the tissue concentration-vs.-time curve (upper right) is a residue function, reflecting the amormt of contrast bolus remaining in the tissue...
Segal S.S. Integration of blood flow control to skeletal muscle key role of feed arteries. Acta Physiol. Scand. 168 511 518,2000. [Pg.1016]

AVM embolization is normally accomplished through transfemoral microcatheter delivery of the material under fluoroscopic guidance. The microcatheter tip is placed where the feeding artery branches off from healthy vasculature and... [Pg.185]

The first, high radial force coils placed to form the scaffold are oversized by 2 mm, i.e. for a 10-mm feeding artery, 12 mm diameter stainless steel or Inconel coils are first placed. These first coils may be anchored as well if there is concern about fixation in the artery. Usually several small diameter high radial force coils are placed as well into the endoskeleton , followed by several softer platinum coils until cross sectional occlusion is achieved. [Pg.39]

Polyvinyl alcohol (PVA) particles have been used in a number of series [6,10,17-20). The particles need to be suspended in iodinated contrast since PVA is not intrinsically radio-opaque otherwise it is not possible to fluoroscopically monitor the embolization. Because the PVA is flow directed, delivery of this embolic agent is less precise than with coils and more subject to local hemodynamics. If the catheter is obturating the feeding artery, the particles will not flow away as readily. Also as the vessel starts to become ocduded by the PVA, the resistance to flow increases and hence the potential for reflux to nontarget segments of bowel will increase. [Pg.79]

Fig.10.5. 4a-f. A 37-year-old patient with heavy bleeding related to a 7-cm intramural fibroid. a,b Right uterine artery angiogram demonstrates spasm (arrow) due to catheterization, c Left uterine artery angiogram shows the feeding artery to the fibroid (arrow), d Embolization of the main feeding artery and patency of the myometrial arteries, e MRI obtained prior to the embolization shows a large intramural mass, f MRI obtained 10 months after the embolization shows an almost normal uterus... [Pg.182]

Fig. 17.2a,b. PAVM diagnosed using CT with multiplanar reconstructions. CT obtained in axial view (a) and coronal maximum intensity projection view (b) shows a single PAVM of the left lower lobe. The feeding artery, aneurysmal sac and draining vein are easily identified... [Pg.282]

Treatment of PAVMs consists of transcatheter embolization performed by interventional radiologists who are specially trained. Fibered platinum coils and in some instances balloons are placed in the feeding artery to the PAVM. [Pg.283]

Follow-up of patients with treated PAVM is critical. By 3-6 months after treatment, the PAVM should be markedly reduced in size leaving a residual scar. Spiral chest CT should be repeated every 5 years in order to identify recanalization of embolized PAVMs and assess growth of any small AVM, until the threshold size (3-mm diameter feeding artery) is reached. [Pg.283]

It is usually considered that PAVMs with feeding arteries (i.e. the artery leading to the malforma-... [Pg.284]

This technique is used for closing high-flow feeding arteries. It is also useful for routine occlusion if there is any concern about movement of the coil after deployment. The first centimeters of a long coil are anchored in a side branch immediately proximal to the site to be occluded (Fig. 17.10). The remaining coil is tightly packed into a nest and additional coils are added and packed until cross sectional occlusion of the artery is achieved (Fig. 17.11). [Pg.287]

Fig. 17.15. a CT performed 6 months after embolization of a complex PAVM of the right upper lobe shows recanalization of the feeding artery (arrow) due to insufficient packing. The draining vein is still opacified (V). b Selective injection of the feeding artery confirms recanalization (arrow). Additional embolization has been performed to obtain complete cross-sectional occlusion... [Pg.292]

PAVM with large feeding artery Anchor technique Occlusion halloon-assisted technique... [Pg.293]

Lipiodol (iodine combined with ethyl esters of fatty acids of poppyseed oil) is used in transcatheter arterial chemoembolization (TACE) in the treatment of heptocellular carcinoma. A mixture of lipiodol and a chemotherapeutic agent is injected into the tumor feeding artery. This procedure has been complicated by lipiodol brain embolization in a few cases. In a patient who developed a modest monoparesis of the right arm, with transient dysarthria and dizziness, during his fourth course of TACE, a CT scan showed multiple infratentorial and supratentorial lesions consistent with deposition of lipiodol [25An MRI scan showed multiple non-enhancing cortical and subcortical hyperintense lesions (FLAIR and DWI), which represented areas of reduced diffusion on DWI. The patient recovered completely over the next 48 hours and the MRI scan returned to almost normal after 1 month. [Pg.754]

AVMs are composed of a network of channels interposed between feeding arteries and draining veins, without any direct shunt. Two different anatomic types of nidus may be more or less differentiated compact nidus, constituting a tumor-like well-circumscribed network, and diffuse nidus, with sparse, abnormal AV channels spread within normal brain parenchyma (Chin et al. 1992). [Pg.52]

Fig. 3.1. Macroscopic view of a surgically resected brain AVM depicts enlarged feeding arteries and draining veins with arteriovenous shunts. Normal brain surrounds the AVM with partially necrosed areas. (Courtesy of Pr. Mikol, Neuropathology Department, Lariboisifere, Paris, France)... Fig. 3.1. Macroscopic view of a surgically resected brain AVM depicts enlarged feeding arteries and draining veins with arteriovenous shunts. Normal brain surrounds the AVM with partially necrosed areas. (Courtesy of Pr. Mikol, Neuropathology Department, Lariboisifere, Paris, France)...
In a relatively small series of patients, Spetzler et al. (1992) evaluated the perfusion pressure of AVM arterial feeders. The difference between mean arterial blood pressure and the feeding artery pressure was higher in ruptured than in non-ruptured AVMs. Moreover, smaller AVMs had significantly higher feeding artery pressure than larger AVMs and were associated with larger hematomas. These results were partially confirmed by Kader et al. (1994), who... [Pg.59]


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