Intestinal ischaemia

Grisham, M.B., Hernandez, L.A. and Granger, D.N. (1986). Xanthine oxidase and neutrophil infiltration in intestinal ischaemia. Am. J. Physiol. 251, G567-G574. [Pg.164]

Nilsson, U.A. Lundgren, O., Haglind, E. and Bylund-Fellenius, A-C. (1989). Radical production during in vivo intestinal ischaemia and reperfusion in the cat. Am. J. Physiol. 257, G409-G414. [Pg.168]

Younes, M., Mohr, A., Schoenberg, M.H. and Schildbeig, F.W. (1987). Inhibition of lipid peroxidation by superoxide dismutase following regional intestinal ischaemia and reperfiision. Res. Exp. Med. 187, 9-17. [Pg.174]

Hyperthyroidism Congestive liver Hyperparathyroidism Leukaemia, lymphoma, multiple myeloma Drugs, chemicals Hyperphosphatasaemia Intestinal ischaemia, coeliac disease Hyperlipidaemia Acromegaly... [Pg.101]

Crohn s disease (Ulcerative) colitis Intestinal ischaemia Intestinal tuberculosis Intestinal invagination Diverticulitis Post-radiation enteritis Adenocarcinoma Gastrointestinal stromal tumour Intestinal metastases Appendicitis Intramural haematoma... [Pg.143]

Bjorck M, Troeng T, Bergqvist D (1997) Risk factors for intestinal ischaemia after aortoiliac surgery a combined cohort and case-control study of 2824 operations. Eur J Vase Endovasc Surg 13 531-539... [Pg.258]

In anaesthetised male Sprague-Dawley rats subjected to 45 min of intestinal ischaemia followed by 3 h of reperfusion B5rjesson et al. (2000) found a 76 % increase in alveolar fluid clearance compared with the control values P <0.05). The stimulated alveolar liquid clearance seen after intestinal ischaemia-reperfusion was not inhibited by propranolol, indicating stimulation through a noncatecholamine-dependent pathway. Administration of a neutralising polyclonal anti-tumour necrosis-a antibody before induction of intestinal ischaemia completely inhibited the increased alveolar liquid clearance observed after intestinal ischaemia-reperfusion. [Pg.624]

A 49-year-old woman presented with acute abdominal pain and nausea. Abdominal CT scan revealed oedematous small bowel and ascites. She was thought to have intestinal ischaemia and was tiien subjected to a laparotomy which revealed that the intestine was vital with no perforation. ACEl-induced angioedema was suspected when a history of ingestion of lisinopril, 3 days prior to presentation was obtained. Lisinopril was replaced with amlodipine and the pain disappeared after 3 days. [Pg.280]

Sommer T. and Larsen J. F, (2004) Intraperitoneal and intraluminal microdialysis in the detection of experimental regional intestinal ischaemia. Br J Surg 91 855-61... [Pg.278]

Granger, D.N., McCord, J.M., Parks, D.A. and FloUwarth, M.E. (1986). Xanthine oxidase inhibitors attenuate ischaemia-induced vascular permeability changes in the cat intestine. Gastroenterology 90, 80-84. [Pg.164]

Joubert-Smith, M., Zhang, S.J., Sibley, D.A. and Specian, R.D. (1991). Intestinal mucosal alterations during ischaemia/reperfiision. Gastroenterolc 100, A829. [Pg.165]

Kubes, P., Hunter, J. and Granger, D.N. (1992). Ischaemia/reperfiision-induced feline intestinal dysfunction importance of granulocyte recruitment. Gastroenterology 103, 807-812. [Pg.166]

Pitt, R.M., McKelvey, T.G., Saenger, J.S., Shah, A.K., Jones, H.P., Manci, E.A. and Powell, R.W. (1991). A tungsten-supplemented diet delivered by transplacental and breastfeeding routes lowers intestinal xanthine oxidase activity and affords cytoprotection in ischaemia-reperfusion injury to the small intestine. J. Paediatr. Suig. 26, 930-935. [Pg.169]

IP4 Inositol tetrakisphosphate IPF Idiopathic pulmonary fibrosis IPO Intestinal peroxidase IpOCOCq Isopropylidene OCOCq I/R Ischaemia-reperfusion IRAP IL-1 receptor antagonist protein IRF-1 Interferon regulatory factor 1 la Short-circuit current ISCOM Immune-stimulating complexes... [Pg.283]

Ikeda H, Suzuki Y, Suzuki M, Koike M, Tamura J, Tong J, Nomura M, Itoh G (1998) Apoptosis is a major mode of cell death caused by ischaemia and ischaemia/reperfusion injury to the rat intestinal epithelium. Gut 42(4) 530-537... [Pg.227]

Aj (EC 3.1.1.4) and phospholipase D (EC 3.1.4.4) seen after ischaemia/reperfusion were prevented by the simultaneous presence of a nitric oxide donor in the intestinal lumen. [Pg.624]

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