6-hydroxydopamine lesions

Deminiere, J.M. Taghzouti, K. Tassin, J.P. Le Moal, M. and Simon, H. Increased sensitivity to amphetamine and facilitation of amphetamine selfadministration after 6-hydroxydopamine lesions of the amygdala. Psychopharmacology 94 232-236, 1988. [Pg.121]

Joyce, E.M., and Koob, G.F. Amphetamine-, seopolamine-, and eaffeine-induced locomotor activity following 6-hydroxydopamine lesions of the mesolimbic dopamine system. Psychopharmacology 73 311-313, 1981. [Pg.122]

Martin-Iversen, M.T. Szostak, C. and Fibiger, H.C. 6-Hydroxydopamine lesions of the medial prefrontal eortex fail to influence intravenous selfadministration of cocaine. Psychopharmacology 88 310-314, 1986. [Pg.123]

Roberts, D.C. Koob, G.F. Klonoff, P. and Fibiger, H.C. Extinetion and recovery of cocaine self-administration following 6-hydroxydopamine lesions of the nucleus accumbens. Pharmacol Biochem Behav 12 781-787, 1980. [Pg.124]

Roberts, D.C.S., and Vickers, G. Increased motivation to self-administer apomorphine following 6-hydroxydopamine lesion of the nueleus aeeum-bens. In Kalivas, P.W., and Nemeroff, C.B., eds. The Mesocortico-limbic Dopamine System. Vol. 537. New York Annals of the New York Academy of Science, 1988. pp. 523-524. [Pg.124]

French, E.D., and Vantini, G. Phencyclidine-induced locomotor activity in the rat is blocked by 6-hydroxydopamine lesion of the nucleus accumbens Comparisons to other psycomotor stimulants. Psvchopharmacoloqy 82 83-88, 1984. [Pg.145]

Tenn, C. C. Niles, L. P. (1995). Central-type benzodiazepine receptors mediate the antidopaminergic effect of clonazepam and melatonin in 6-hydroxydopamine lesioned rats involvement of a GABAergic mechanism. J. Pharmacol. Exp. Ther. 274, 84-9. [Pg.312]

Huang R, Han L, Li J, Ren F, Ke W, Jiang C, Pei Y (2009) Neuroprotection in a 6-hydroxydopamine-lesioned Parkinson model using lactoferrin-modilied nanoparticles. J Gene Med 11 754-763... [Pg.26]

Parsons LH, Smith AD, Justice JB Jr. 1991b. The in vivo microdialysis recovery of dopamine is altered independently of basal level by 6-hydroxydopamine lesions to the nucleus accumbens. J Neurosci Methods 40(2—3) 139-147. [Pg.252]

Gilgun-Sherki, Y., E. Melamed, R. Mechoulam, and D. Offen. The CBl cannabinoid receptor agonist, HU-210, reduces levodopa-induced rotations in 6-hydroxydopamine-lesioned rats. Pharmacol Toxicol 2003 93(2) 66—70. Pryce, G., Z. Ahmed, D. L. Hankey, et al. Cannabinoids inhibit neurodegeneration in models of multiple sclerosis. Brain 2003 126(Pt. 10) 2191-2202. Mathew, R. J., W. H. Wilson, and R. Davis. Postural syncope after marijuana a transcranial Doppler study of the hemodynamics. Pharmacol Biochem Behav 2003 75(2) 309-318. [Pg.109]

Datla, K. P., R. D. Bennett, V. Zbarsky, et al. The antioxidant drink effective microorganism-X (EM-X) pre-treatment attenuates the loss of nigros-triatal dopaminergic neurons in 6-hydroxydopamine-lesion rat model of Parkinson s disease. J Pharm Pharmacol 2004 56(5) 649-654. [Pg.418]

Hydroxydopamine lesions, which destroy NE neurons in the dorsal and ventral bundles as well as in the locus ceruleus, block the enhanced locomotor responses to quipazine after repeated ECS. [Pg.115]

Mohapel, P., Frielingsdorf, H., Haggblad, J., Zachrisson, O., Brundin, P. (2005). Platelet-Derived Growth Factor (PDGF-BB) and Brain-Derived Neurotrophic Factor (BDNF) induce striatal neurogenesis in adult rats with 6-hydroxydopamine lesions. Neuroscience, 132, 767-76. [Pg.30]

Shen H, Kannari K, Yamato H, et al. Effects of benser-azide on L-DOPA-derived extracellular dopamine levels and aromatic L-amino acid decarboxylase activity in the striatum of 6-hydroxydopamine-lesioned rats. Tohoku J Exp Med. 2003 199 149-159. [Pg.133]

Cai G, Zhen X, Uryu K, Friedman E. Activation of extracellular signal regulated protein kinases is associated with a sensitized locomotor response to D(2) dopamine receptor stimulation in unilateral 6-hydroxydopamine lesioned rats. J Neurosci 2000 20 1849-1857. [Pg.147]

Pycock, C., Tarsy, D., Marsden, C. D. 1975c, Inhibition of circling behavior by neuroleptic dmgs in mice with unilateral 6-hydroxydopamine lesions of the striatum, Psychopharmacologia., vol. 45, no. 2, pp. 211-219. [Pg.258]

Von Voigtlander, P. E Moore, K. E. 1973, Turning behavior of mice with unilateral 6-hydroxydopamine lesions in the striatum effects of apomorphine, L-DOPA, amanthadine, amphetamine and other psychomotor stimulants, Neuropharmacology, vol. 12, no. 5, pp. 451-462. [Pg.269]

Bubser M, Koch M. 1994. Prepulse inhibition of the acoustic startle response of rats is reduced by 6-hydroxydopamine lesions of the medial prefrontal cortex. Psychopharmacology 113 487-492. [Pg.13]

Roberts AC, Desalvia MA, Wilkinson LS, Collins P, Muir JL, et al. 1994. 6-Hydroxydopamine lesions of the prefrontal cortex in monkeys enhance performance on an analog of the Wisconsin card sort test Possible interactions with subcortical dopamine. J Neurosci 14 2531-2544. [Pg.15]

De Klippel N, Sarre S, Ebinger G, Michotte Y. 1993. Effect of M and M2-muscarinic drugs on striatal dopamine release and metabolism An in vivo microdialysis study comparing normal and 6-hydroxydopamine-lesioned rats. Brain Res... [Pg.32]

Morelli M, Carboni E, Devoto S, Di Chiara G (1987) 6-Hydroxydopamine lesions reduce specific [3H]sulpiride binding in the rat substantia nigra direct evidence for the existence of nigral D-2 autoreceptors. Eur J Pharmacol 740 99-104. [Pg.101]

Cole DG, Kobierski LA, Konradi C, Hyman SE (1994) 6-Hydroxydopamine lesions of rat substantia nigra up-regulate dopamine-induced phosphorylation of the cAMP-response element-binding protein in striatal neurons. Proc Natl Acad Sci USA 97 9631-9635. [Pg.140]

Robertson GS, Herrera DG, Dragunow M, Robertson HA (1989a) L-DOPA activates c-fos in the striatum ipsilateral to a 6-hydroxydopamine lesion of the substantia nigra. Eur J Pharmacol 759 99-100. [Pg.148]

Voorn P, Roest G, Groenewegen HJ (1987) Increase of enkephalin and decrease of substance P immunoreactivity in the dorsal and ventral striatum of the rat after midbrain 6-hydroxydopamine lesions. Brain Res 472 391-396. [Pg.150]

Brecknell JE, Haque NS, Du JS, Muir EM, Fidler PS, Hlavin ML, Fawcett JW, Dunnett SB (1996) Functional and anatomical reconstruction of the 6-hydroxydopamine lesioned nigrostriatal system of the adult rat. [Pg.185]

Ingham CA, Hood SH, Arbuthnott GW (1989) Spine density on neostriatal neurones changes with 6-hydroxydopamine lesions and with age. Brain Res 505 334-338. [Pg.231]

Apicella P, Trouche E, Nieoullon A, Legallet E, Dusticier N (1990) Motor impairments and neurochemical changes after unilateral 6-hydroxydopamine lesion of the nigrostriatal dopaminergic system in monkeys. 35 655-666. [Pg.280]

Carey RJ (1986) Conditioned rotational behaviour in rats with unilateral 6-hydroxydopamine lesions of the substantia nigra. Brain Res 565 379-382. [Pg.283]

Dunnett SB, Iversen SD (1982) Sensorimotor impairments following localized kainic acid and 6-hydroxydopamine lesions of the neostriatum. Brain Res 248 121-127. [Pg.285]

Dunnett SB, Whishaw IQ, Jones GH, Isacson O (1986) Effects of dopamine-rich grafts on conditioned rotation in rats with unilateral 6-hydroxydopamine lesions. Neurosci Lett 68 127-133. [Pg.285]

Dunnett SB, Whishaw IQ, Rogers DC, Jones GH (1987) Dopamine-rich grafts ameliorate whole body motor asymmetry and sensory neglect but not independent limb use in rats with 6-hydroxydopamine lesions. Brain Res 475 63-78. [Pg.285]

Evenden JL, Robbins TW (1984) Effects of unilateral 6-hydroxydopamine lesions of the caudate-putamen on skilled forepaw use in the rat. Behav Brain Res 74 61-68. [Pg.285]

Hamilton MH, Garcia-Munoz M, Arbuthnott GW (1985) Separation of the motor consequences from other actions of unilateral 6-hydroxydopamine lesions in the nigrostriatal neurones of rat brain. Brain Res 348 220-228. [Pg.287]

Heikkila RE, Shapiro BS, Duvoisin RC (1981) The relationship between loss of dopamine nerve terminals, striatal [3H]spiroperidol binding and rotational behavior in unilaterally 6-hydroxydopamine-lesioned rats. Brain Res 211 285—292. [Pg.288]

Roeling TAP, Docter GJ, Voorn P, Melchers BPC, Wolters EC, Groenewegen HJ (1995) Effects of unilateral 6-hydroxydopamine lesions on neuropeptide immunoreactivity in the basal ganglia of the common marmoset, Callithrix jacchus, a quantitative immunohistochemical analysis. J Chem Neuroanat 9 155-164. [Pg.294]

Schwarting RKW, Huston JP (1996a) The unilateral 6-hydroxydopamine lesion model in behavioral brain research. Analysis of functional deficits, recovery and treatments. Prog Neurobiol 50 275-331. [Pg.296]

Schwarting RKW, Huston JP (1996b) Unilateral 6-hydroxydopamine lesions of meso-striatal dopamine neurons and their physiological sequelae. Prog Neurobiol 49 215-266. [Pg.296]

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