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Growth factor, platelet-derived

PDGF plays an important role in the wound healing process. It is released at the site of damage by activated platelets, and acts as a mitogen/chemoattractant for many of the cells responsible for initiation of tissue repair. It thus tends to act primarily in a paracrine manner. It also represents an autocrine/paracrine growth factor for a variety of malignant cells. [Pg.283]

Normal skin appears to be devoid of PDGF receptors. Animal studies illustrate that rapid expression of both a and P receptor subunits is induced upon generation of an experimental wound (e.g. a surgical incision). Receptor expression is again switched off following re-epithelialization and complete healing of the wound. [Pg.284]

III. Tyr protein kinases A. Cytosolic tyrosine kinases src, fgr, abl, etc.) B. Receptor tyrosine kinases (RTKs) Plasma membrane receptors for hormones such as epidermal growth factor (EGF) or platelet-derived growth factor (PDGE) Raf (a protein kinase)... [Pg.467]

Direct laser-assisted myocardial revascularization (DMR) is an approved technique in the US, Europe, and parts of Asia to create numerous myocardial channels. This results in the induction of a massive inflammatory reaction, which in turn induces angiogenesis. The other FDA-approved pro-angiogenic therapy is the use of recombinant human platelet-derived growth factor (Regranex) for use in the treatment of diabetic neuropathic foot ulcer s. [Pg.88]

Platelet-activating Factor Platelet Aggregation Inhibitors Platelet-derived Growth Factor Receptor Platelet Inhibitors Platelets... [Pg.1500]

Insulin-like growth factors I and II Nerve growth factor Platelet-derived growth factor Prolactin... [Pg.437]

Abundance of Na /H exchanger transcripts in vascular smooth muscle and HL60 cells is increased by serum, phorbol esters, fibroblast growth factor and platelet-derived growth factor [78,79]. The increase in NHE-1 mRNA levels in PMA-treated HL60 cells is due to increased gene transcription. [Pg.268]

PDGFR Platelet-derived growth factor receptor... [Pg.285]

Muller, C. and Rinas, U., Renaturation of heterodimeric platelet-derived growth factor from inclusion bodies of recombinant Escherichia coli using size-exclusion chromatography, /. Chromatogr. A, 855, 203, 1999. [Pg.381]

Fujio Y, Yamada F, Takahashi K, Shibata N. Responses of smooth muscle cells to platelet-derived growth factor are inhibited by herbimycin-A tyrosine kinase inhibitor. Biochem Biophys Res Commun 1993 195(1) 79—83. [Pg.288]

Soskic, V., Gorlach, M., Poznanovic, S., Boehmer, F. D., and Godovac-Zimmermann, J. (1999). Functional proteomics analysis of signal transduction pathways of the platelet-derived growth factor b receptor. Biochemistry 38, 1757-1764. [Pg.122]

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