Food products, inhaled

EXPOSURE ROUTES drinking water eonsumption of beverages and food products inhalation of contaminated ambient air dermal exposure to chlorinated swimming pool water... 

The dust and powder of cobalt metal, ores, and some compounds, such as cobaltous nitrate [ColNOfljT bH O], are flammable and toxic if inhaled. Cobaltous acetate [ColCjHjOjlj dHjO], which is soluble in water, is not allowed to be used in food products because of its toxicity. 

Ambient environmental levels of tin are generally quite low, except in the vicinity of pollution sources. Humans may be exposed to tin by inhalation, ingestion, or dermal absorption. However, human exposure to tin is primarily by ingestion of food, especially canned food products. 

Ethylene oxide, (CH2)20 (bp 10.7°C)—Ethylene oxide is mainly used as a mixture with carbon dioxide for vault fumigation of stored food products. Its inhalation LC50 (4 hr) in rats is 1462 ppm. 

Worldwide, sporadic cases and hmited outbreaks of botulism can occur when food and food products are prepared or preserved by improper methods that do not destroy the spores of Clostridium botulinum and permit the formation of botulinum toxin. In industrially developed countries, the case fatality rate of food-borne botulism is 5-10%. Person-to-person transmission of botulism is not known. Botulinum toxin is the most poisonous substance known and poses a major bioweapon threat. In addition to the clinical forms of natural botulism (food-borne, wound, and intestinal), there is a fourth, man-made form of inhalational botulism that results from aerosolized botulinum toxin. 

Sodium sulfite is used as an antioxidant in applications similar to those for sodium metabisulfite It is also an effective antimicrobial preservative, particularly against fungi at low pH (0.1% w/v of sodium sulfite is used). Sodium sulfite is used in cosmetics, food products, and pharmaceutical applications such as parenteral formulations, inhalations, oral formulations, and topical preparations. 

The presence of DAAB as a dye contaminant in cosmetics and food products could result in low level exposures via the oral and dermal routes. Occupational exposure may occur through inhalation and dermal contact where these chemicals are produced or used as a chemical intermediate and polymer additive. DAAB is harmful to the respiratory tract, skin, and eyes. Most exposures to the general population are typically through consumption of food and use of cosmetics containing DAAB impurities. 

Occupational exposure may occur through inhalation or dermal contact where the compound is produced, in the manufacture of flavoring materials where it is a component of that flavor, or when the final flavor is being added to a food product. Most exposure for the general population is through various food products. 

The general population is exposed to propylene glycol by oral intake, dermal contact, and inhalation. The average daily intake of propylene glycol from food products in the United States has been estimated at 2400 mg day (34 mg kg body weight (bw) per... 

Occupational exposure to vanillin may occur through inhalation and dermal contact at workplaces where vanillin is produced or used. The general population may be exposed to ethyl vanillin via dermal contact with perfumes and ingestion of food products that contain vanillin as a flavor additive. 

Hazard May react explosively with mineral acids and some organic acids. Toxic by inhalation and skin absorption. Approved for food products. TLV 10 ppm STEL 15 ppm, toxic by skin absorption. 

EXPOSURE ROUTES Inhalation (contaminated air), ingestion (fumigated food products and contaminated food), absorption occupational exposure... 

Brucellosis is a zoonotic infection of domesticated and wild animals, caused by organisms of the genus Brucella. Humans become infected by ingestion of animal food products, direct contact with infected animals, or inhalation of infectious aerosols. 

Normally, there are low levels of cyanide in the body as a result of certain food products and inhalation contamination such as smoking. Small amount of cyanide is normally eliminated using an enzyme, rhodanese found in the liver. Rhodanese catalyses the reaction of cyanide (CN) with thiosulphate to produce thiocyanate (SCN) that is excreted later in the urine. This pathway is one of the means of treating cyanide poisoning as explained in the next chapter. 

The chief source of such energy is the combustion of carbon compounds to C02. You know that man exhales more carbon dioxide than he inhales in the air he breathes. This extra carbon dioxide is one of the products of the oxidation processes by which food is oxidized and energy is liberated. 

The most important routes of exposure to endosulfan for the general population are ingestion of food and the use of tobacco products with endosulfan residues remaining after treatment. Farmers, pesticide applicators, and individuals living in the vicinity of hazardous waste disposal sites contaminated with endosulfan may receive additional exposure through dermal contact and inhalation. 

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