Evidence, destruction

That as the laws ought to be equal, so they must be good and not evidently destructive to the safety and well-being of the people. 

The test with the step loading shows that acoustic activity of the solid adhesive joints in the tested carbon plastic is quite low. The maximum on the endurance area was fixed at the predestructive moment. The last is evidence to the fact that the prevailing defect of the adhesive joints is starting its development at the loading level, which is close to the destruction point. 

Destruction of the fiber filler may occur during compression of a composite. It is evident that the destroying process is more pronounced for high-modulus, brittle fibers such as glass or carbon. For the materials containing organic fibers the destruction rate is considerably lower. 

Doping of solid reactant involves the introduction of a controlled amount of an impurity into solid solution in the host lattice. Such impurities can be selected to cause the generation or destruction of those electronic or structural defects which participate in the rate process of interest. Thus, the influence of the additive on kinetic behaviour can provide evidence concerning the mechanism of reaction [46,47]. Even if the... 

The nurse inspects the patient s mouth daily for ulceration of the mucous membranes. A metallic taste may be noted before stomatitis becomes evident. The nurse advises the patient to inform the primary health care provider or nurse if a metallic taste occurs. Good oral care is necessary. The teeth should be brushed after each meal and the mouth rinsed with plain water to remove food particles. Mouthwash may also be used, but excessive use may result in oral infections due to the destruction of the normal bacteria present in the mouth. 

At the outset of 1994 there is evidently more public (and perhaps professional) interest in the destruction of organic halogen compounds (ref. 1) than in their creation. 

Combining these arguments with the observation that extensive Auger charging evidently leads to total and usually permanent molecular destruction, it can thus be concluded that molecule survival or reformation is observed to some extent when the initial species is a single atom... 

There continnes to be public concern about the possibility of their being used in future. When Saddam Hussein was in power in Iraq, there was evidence that a chemical weapon of this type was used against Kurdish villagers. Subsequently, it was widely believed that these were among the weapons of mass destruction held by Saddam Hnssein s regime weapons that failed to materialize after the invasion of Iraq in 2003. Since these events, there has been concern that weapons of this type may be in the possession of rogne states— or individual terror groups. 

Figure 3 gives an example of a typical force profile. The force is increased continuously and reaches the point - at the end of the first part of the force profile - where the pectin preparations start to flow. The so-called yield point is reached. The further increase leads to the continuous destruction of the internal structure and the proceeding shear thinning. The applied stress in part 3 of the stress profile destroys the structure of the fruit preparations completely. Now the stress is reduced linearly, see part 4 and 5, down to zero stress. The resulting flow curves 2, 3 and 4 and the enclosed calculated area from the hysteresis loop give important evidence about the time-dependent decrease of viscosity and a relative measure of its thixotropy. 

There is quite a different picture in the a-oxygen presence (Fig 3b). Just after reactor opening a large and irreversible CH4 consumption occurs now, which evidences its chemical reaction with a-oxygen to take place at room temperature. The reaction product is strongly bound to the surface, and can not desorb into the gas phase under heating without destruction, which is accompanied by the CO evolution. 

Investigation of the neurochemical substrates for the psychostimulant effects of MDMA suggests a role for the mesolimbic dopamine system. Destruction of dopamine terminal fields in the nucleus accumbens significantly attenuated the locomotor activation produced by MDMA. A similar blockade of amphetamine-induced locomotor hyperactivity is known and was observed following amphetamine injection in these same rats. Such results support the hypothesis that at least one component of MDMA-induced hyperactivity is dopamine mediated and suggest that mesolimbic dopamine specifically is the critical substrate. In this way, MDMA resembles other classical psychostimulants like amphetamine and cocaine. Interestingly, evidence for functional cross-sensitization was suggested in the study in which an injection of amphetamine followed MDMA injection. 

Effects In Humans. Neither postmortem nor functional cerebrospinal fluid (CSF) studies in humans provide firm evidence for similar, long-term damages or alterations to monoaminergic neurons in chronic stimulant abusers. In part, the lack of demonstrable neurochemical changes may well be due to the obvious preclusion of well-controlled prospective experimentation in humans, as well as to variability in critical variables (e.g., individual sensitivity or pattern of abuse) encountered in clinical research. Possible relationship of the various complications of stimulant abuse including hyperpyrexia, seizure, anoxia, and metabolic exhaustion to neuronal chromatolysis, terminal destruction, and monoamine and enzymatic depletion have not been systematically explored in human autopsy eases. It should be also noted that, under nonperturbed conditions, overt behavioral deficits are rare in... 

One of the striking aspects of this toxicity of compounds is selective destruction terminals and the cell bodies that are left intact. Dr. De Souza has recently reported some biochemical evidence for recovery of serotonin. We have now found anatomic evidence for reinnervation of depleted areas by serotonin neurons. But it is going to be a while before we figure out whether their reinnervation is appropriate or perhaps aberrant. Do they end up with complete recovery, do they end up with a better system than they started with or one that malfunctions I think that is an important area for future study. 

Patients with recurrent attacks, evidence of tophi or joint destruction, or uric acid nephrolithiasis are candidates for maintenance therapy with allopurinol or probenecid to lower serum uric add levels. 

Acute GVHD usually is not evident until the time of engraft -ment, when donor lymphoid elements begin to proliferate. The primary targets of immune-mediated destruction of host tissue by donor lymphocytes in acute GVHD are the skin, liver, and GI tract.44 A diffuse maculopapular rash on the face, truck, face, extremities, palms, soles, and ears is the usual sign of acute GVHD of the skin. In more severe cases, it can... 

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