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E gene

Boylan, M., etal. (1989). Lux C, D and E genes of the Vibrio fischeri luminescence operon code for the reductase, transferase and synthetase enzymes involved in aldehyde biosynthesis. Photochem. Photobiol. 49 681-688. [Pg.383]

Cunningham, F.X., Jr. and Gantt, E., Genes and enzymes of carotenoid biosynthesis in plants, Annu. Rev. Plant Physiol. Plant Mol. Biol. 49, 557, 1998. [Pg.390]

The genetic basis for the more common late-onset AD appears more complex. Genetic susceptibility is more sporadic and it may be more dependent on environmental factors.9 The apolipoprotein E (apo E) gene on chromosome 19 has been identified as a strong risk factor for late-onset AD. There are three variants of apo E however, carriers of two or more of the apo E4 allele have an earlier onset of AD (approximately 6 years earlier) compared with non-carriers.9 Only 50% of AD patients have the apo E4 allele, thus indicating it is only a susceptibility marker. [Pg.515]

DNA polymerase III has all the enzymatic activities of DNA polymerase I. A subunit of the enzyme is the product of the dna E gene. Temperature-sensitive mutations of this gene testify to the importance... [Pg.225]

Kato, N., and Yanase, E., Gene coding for desulfurizing enzyme. Patent No. JP2000245477. 2000, Sep.12. [Pg.370]

Grundemann D, Schomig E. Gene structures of the human non-neuronal monoamine transporters EMT and OCT2. Hum Genet 2000 106(6) 627-635. [Pg.212]

Marker Known location on a chromosome (e.g., restriction enzyme cutting site, gene) whose inheritance can be monitored. Markers are located in or close to coding regions of DNA (i.e., genes) or in segments of DNA with no known coding function but whose pattern of inheritance can be determined (i.e., microsatellites). [Pg.535]

Apochromatic objectives, 16 471 Apocynaceae, alkaloids in, 2 75 Apodization process, 14 227 apo E gene, and LDL level, 5 136 Apoglucose oxidase (apo-GOD), 14 148 Apolipoprotein B deficiencies, 17 652 Apparel, nylon, 19 766 Apparent bypass... [Pg.66]

Rodriguez, J. M., Salas, M. L., and Vinuela, E. Genes homologous to ubiquitin-conjugating proteins and eukaryotic transcription factor SII in African swine fever virus. Virology 1992, 386, 40-52. [Pg.129]

Tighe H. Corr M. Roman M. Raz E Gene vaccination plasmid DNA is more than just a blueprint. Immunol Today 1998 19 89-97. [Pg.188]

Marshall E. Gene therapy death prompts review of adenovirus vector. Science 1999 286(5448) 2244 2245. [Pg.269]

D. The glucose 6-phosphata e gene is on different chromosomes in the marmoset and in the human. [Pg.114]

Salero, E., Perez-Sen, R., Aruga, J., Gimenez, C., Zafra, E. (2001) Transcription factor Zicl and Zic2 bind and transactive the apolipoprotein E gene promoter. J. Biol. Chem., 278,1881-1888. [Pg.348]

Seitz, A., Gourevitch, D., Zhang, X.M., et al. (2005) Sense and antisense transcripts of the apolipoprotein E gene in normal and APOE knockout mice, their expression after spinal cord injury and corresponding human transcripts. Hum. Mol. Genet., 14, 2661-2670. [Pg.348]

Isbir, T., Agachan, B., Yilmaz, H., et al. (2001) ApoUpoprotein-E gene polymorphism and hpid profiles in Alzheimer s disease. Am. J. Alzheimer Dis. Other Demen., 16, 77-81. [Pg.353]

BeUivier, F., Laplanche, J.L., Schurhoff, F., et al. (1997) Apohpoprotein E gene polymorphism in early and late onset bipolar patients. Neurosci. Lett., 233, 45-48. [Pg.356]

M2a. Marks, P. A., Gross, R. T., and Hurwitz, R. E., Gene action in erythrocyte deficiency of glucose-6-phosphate dehydrogenase Tissue-enzyme levels. Nature (.London) 183, 1266-1267 (1959). [Pg.39]

Kloos et al. describe the induction of cellular autolysins by the X 174E gene, which causes cell lysis, and, apparently via a cellular nuclease, the degradation of cellular nucleic acids (Kloos et al., 1994). Indeed, cellular DNA was shown to be degraded after induction of the plasmid carrying the E gene. These containment approaches are summarized in Table 11.3. [Pg.367]


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See also in sourсe #XX -- [ Pg.174 , Pg.367 , Pg.368 ]




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