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Dilation types

Any rotational viscometer, such as the Brookfield model DV, or the Anton-Paar Rheolab QC, can be used. The suggested test fluids of the pseudoplastic type can be paper pulp or orange juice concentrate, while the dilatant type can be concentrated starch or limestone suspensions. [Pg.360]

Dilatant fluids (also known as shear thickening fluids) show an increase in viscosity with an increase in shear rate. Such an increase in viscosity may, or may not, be accompanied by a measurable change in the volume of the fluid (Metzener and Whitlock, 1958). Power law-type rheologicaJ equations with n > 1 are usually used to model this type of fluids. [Pg.8]

Laser ablation systems hold considerable promise if restenosis (reblocking of the arteries) rates are reduced. The rate as of 1995 is 30%, typically within six months. Mechanical or atherectomy devices to cut, shave, or pulverize plaque have been tested extensively in coronary arteries. Some of these have also been approved for peripheral use. The future of angioplasty, beyond the tremendous success of conventional balloon catheters, depends on approaches that can reduce restenosis rates. For example, if appHcation of a dmg to the lesion site turns out to be the solution to restenosis, balloon catheters would be used for both dilating the vessel and deUvering the dmg. An understanding of what happens to the arterial walls, at the cellular level, when these walls are subjected to the various types of angioplasty may need to come first. [Pg.182]

The vascular endothelium plays an important role in regulation of vascular tone and permeability. Dilatation of arterioles to increase blood flow and constriction of endothelial cells of postcapillary venules causing exsudation of plasma constituents illustrates the complex nature of this cell type. Moreover, by expression of adhesion molecules and secretion of chemokines endothelial cells play an important role in the recruitment of leukocytes to the inflamed area. Endothelial cells express two basic types of adhesion molecules on their surface ... [Pg.627]

Histamine is a substance present in various tissues of die body, such as die heart, lungs, gastric mucosa, and skin (Pig. 36-1). The highest concentration of histamine is found in die basophil (a type of white blood cell) and mast cells diat are found near capillaries. Histamine is produced in response to injury. It acts on areas such as die vascular system and smooth muscle, producing dilatation of arterioles and an increased permeability of capillaries and venules. Dilatation of die arterioles results in localized redness. An increase in die permeability of... [Pg.325]

Another type of cardiomyopathy is termed dilated cardiomyopathy. Mutations in the genes encoding dystrophin, muscle LIM protein (so called because it was found to contain a cysteine-tich domain originally detected in three proteins Lin-II, Isl-1, and Mec-3), and the cyclic response-element binding ptotein (CREB) have been implicated in the causation of this condition. The first two proteins help organize the conttactile ap-params of cardiac muscle cells, and CREB is involved... [Pg.570]

Method 2. Place a 3 0 g. sample of the mixture of amines in a flask, add 6 g. (4-5 ml.) of benzenesulphonyl chloride (or 6 g. of p-toluenesulphonyl chloride) and 100 ml. of a 5 per cent, solution of sodium hydroxide. Stopper the flask and shake vigorously until the odour of the acid chloride has disappeared open the flask occasionally to release the pressure developed by the heat of the reaction. Allow the mixture to cool, and dissolve any insoluble material in 60-75 ml. of ether. If a solid insoluble in both the aqueous and ether layer appears at this point (it is probably the sparingly soluble salt of a primary amine, e.g., a long chain compound of the type C,H5(CHj) NH2), add 25 ml. of water and shake if it does not dissolve, filter it off. Separate the ether and aqueous layers. The ether layer will contain the unchanged tertiary amine and the sulphonamide of the secondary amine. Acidify the alkaline aqueous layer with dilate hydrochloric acid, filter off the sulphonamide of the primary amine, and recrystaUise it from dilute alcohol. Extract the ether layer with sufficient 5 per cent, hydrochloric acid to remove all the tertiary amine present. Evaporate the ether to obtain the sulphonamide of the secondary amine recrystallise it from alcohol or dilute alcohol. Finally, render the hydrochloric acid extract alkaline by the addition of dilute sodium hydroxide solution, and isolate the tertiary amine. [Pg.651]

Our goal is to develop a property-performance relationship for different types of demulsifiers. The important interfacial properties governing water-in-oil emulsion stability are shear viscosity, dynamic tension and dilational elasticity. We have studied the relative importance of these parameters in demulsification. In this paper, some of the results of our study are presented. In particular, we have found that to be effective, a demulsifier must lower the dynamic interfacial tension gradient and its ability to do so depends on the rate of unclustering of the ethylene oxide groups at the oil-water interface. [Pg.367]

Charron, P., Tesson, E., Poirier, O., et al. (1999) Identification of a genetic risk factor for idiopathic dilated cardiomyopathy. Involvement of a polymorphism in the endothehn receptor type A gene. CARDIGENE group. Eur. Heart J. 20, 1587-1591. [Pg.184]

Field measurements in sedimentary fluvial-type calcrete deposits also suggest that present-day groundwater in these areas may also display potential to both dissolve and precipitate uranium in the near surface. Chemical dilatancy and evaporation-driven diffusion that promote de-complexing, diffusion, and reprecipitation mechanisms are seen to play integral parts in the continued chemical reworking and modification of these calcrete-hosted carnotite deposits. [Pg.429]

Histamine, an important mediator (local signaling substance) and neurotransmitter, is mainly stored in tissue mast cells and basophilic granulocytes in the blood. It is involved in inflammatory and allergic reactions. Histamine liberators such as tissue hormones, type E immunoglobulins (see p. 300), and drugs can release it. Histamine acts via various types of receptor. Binding to Hi receptors promotes contraction of smooth muscle in the bronchia, and dilates the capillary vessels and increases their permeability. Via H2 receptors, histamine slows down the heart rate and promotes the formation of HCl in the gastric mucosa. In the brain, histamine acts as a neurotransmitter. [Pg.380]


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