Depression stimulants

See Chap. 68, Substance-Related Disorders Overview and Depressants, Stimulants, and Hallucinogens, authored by Paul L. Doering and Lisa Boothby, and Chap. 69, Substance-Related Disorders Alcohol, Nicotine, and Caffeine, authored by Paul L. Doering, W. Klugh Kennedy, and Lisa A. Boothby, for a more detailed discussion of the topic. 

Ginseng interacts with phenelzine, a drug used to treat depression, stimulating the central nervous system. 

Tetrahydro-(3-carboline Metabolite from Tryptamine anti malarial, antifibrillatory, bitter taste, cardiac depressant, stimulant] MAO-A (5), MAO-B... 

Kefauver-Harris Drug Amendments passed to ensure drug efficacy and greater drug safety in response to the thalidomide birth defects disaster. 1965 Drug Abuse Control Amendments are enacted to deal with problems of abuse of depressants, stimulants, and hallucinogens. 

Substance-Related Disorders Overview AND Depressants, Stimulants,... 

Drug Abuse Control Amendments are enacted to deal with problems caused by abuse of depressants, stimulants, and hallucinogens. 

Rise in P cone. Supersaturation Formation of bone mineral P level restored, Ca level depressed Stimulation... 

It is recommended either that the provision of the Fedei act which exempts from the prohibition on unauthori/i possession, possession "(1) for personal use of the possesst or a member of this household, (2) for administration to animal owned by him or a member of his household and whl< puts the burden of proving that the possession was not for ar of the purposes mentioned on unauthorized possession with purpose to sell or otherwise dispose of a depressant stimulant drug, but exempting possession (1) for the personj use of a member of the possessor s household, or (2) fc administration to an animal owned by the possessor or member of his household, should be included in any Stal legislation. State law should not prohibit simple possession use. 

The Department of Health, Education, and Welfare s Bureau of Drug Abuse Control was responsible for the control of dangerous drugs, including depressants, stimulants, and hallucinogens, such as lysergic acid diethylamide (LSD). 

Analogs of A. are used to control blood pressure, counteract depression, stimulate the appetite and relieve asthma. 

H Airpclilc depressant and central nervous. system stimulant 876... 

Patients receiving monoamine oxidase inhibitors (MAOI) as antidepressant therapy have been especially subject to the hypertensive effects of vasoactive amines (52). These dietary amines have also been impHcated as causative agents ia migraine. Other aaturaHy occurring alkaloids (qv) have been recognized for centuries as possessing neurological stimulant and depressant properties. 

General types of physiological functions attributed to quaternary ammonium compounds are curare action, muscarinic—nicotinic action, and ganglia blocking action. The active substance of curare is a quaternary that can produce muscular paralysis without affecting the central nervous system or the heart. Muscarinic action is the stimulation of smooth-muscle tissue. Nicotinic action is primary transient stimulation and secondary persistent depression of sympathetic and parasympathetic ganglia. 

Normally, dietary tyramine is broken down in the gastrointestinal tract by MAO and is not absorbed. In the presence of MAOI, however, all of its potent sympathomimetic actions are seen. Other side effects of MAOI include excessive CNS stimulation, orthostatic hypotension, weight gain, and in rare cases hepatotoxicity. Because the monoamine oxidase inhibitors exhibit greater toxicity, yet no greater therapeutic response than other, newer agents, clinical use has been markedly curtailed. The primary use for MAOIs is in the treatment of atypical depressions, eg, those associated with increased appetite, phobic anxiety, hypersomnolence, and fatigues, but not melancholia (2). 

Other Drugs. Agents not considered to be CNS stimulants yet employed for the treatment of certain types of depression includes lithium carbonate for the treatment of bipolar disorder. In most patients, lithium is the sole agent used to control manic behavior and is very effective (see... 

The Class I agents have many similar side effects and toxicities. The anticholinergic side effects include dry mouth, constipation, and urinary hesitancy and retention. Common gastrointestinal (GI) side effects include nausea, vomiting, diarrhea, and anorexia. Cardiovascular adverse effects are hypotension, tachycardia, arrhythmias, and myocardial depression, especially in patients with congestive heart failure. Common central nervous system (CNS) side effects are headache, dizziness, mental confusion, hallucinations, CNS stimulation, paraesthesias, and convulsions. 

Because alcohol intoxication may be simulated by many pathologic conditions, including diabetic acidosis, the postconvulsive depression of epilepsy, uremia, head injuries, and poisonings by any other central nervous depressant and some stimulants (280), a diagnosis of acute alcoholism should not be made casually chemical testing of blood, urine, or expired air is always desirable. 

Health Hazards Information - Recommended Personal Protective Equipment Protective goggles, gloves Symptoms Following Exposure Irritation of mucous membranes and stimulation followed by depression of central nervous system. Breathing of vapor may also cause dizziness, headache, and in... 

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