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Carotid artery occlusion

Lin W, Celik A, Derdeyn C, An H, Lee Y, Videen T, Qstergaard L, Powers WJ. Quantitative measurements of cerebral blood flow in patients with unilateral carotid artery occlusion a PET and MR study. J Magn Reson Imaging 2001 14 659-667. [Pg.33]

Internal Carotid Artery Occlusion Acute stroke due to a distal ICA T (T = terminus) occlusion carry a much worse prognosis than MCA occlusions. In a recent analysis of 24 consecutive patients (median NIHSS 19) presenting with T occlusions of the ICA who were treated by lAT using urokinase at an average of 237 minutes from symptom onset, only four patients (16.6%) had a favorable outcome at 3 months. Partial recanalization of the intracranial ICA was achieved in 15 (63%), of the MCA in 4 (17%), and of the ACA in 8 patients (33%). Complete recanalization did not occur. The presence of good leptomeningeal collaterals and age <60 years were the only predictors of a favorable clinical outcome. New treatment strategies, such as the combination of IV rt-PA and lAT, or the use of new mechanical devices may improve the outcome in these patients. [Pg.67]

Zaidat OO, Suarez Jl, Santillan C, Sunshine JL, Tarr RW, Paras VH, Selman WR, Landis DM. Response to intra-arterial and combined intravenous and intra-arterial thrombolytic therapy in patients with distal internal carotid artery occlusion. Stroke 2002 33 1821-1826. [Pg.92]

Jovin TG, Gupta R, Uchino K, Jungreis CA, Wechsler LR, Hammer MD, Tayal A, Horowitz MB. Emergent stenting of extracranial internal carotid artery occlusion in acute stroke has a high revascularization rate. Stroke 2005 36 2426-2430. [Pg.96]

KawaiN, Keep RE, Betz AL. Effects of hyperglycemia on cerebral blood flow and edema formation after carotid artery occlusion in Fischer 344 rats. Acta Neurochir Suppl 1997 70 34-36. [Pg.190]

The basic biology of chemokines and their receptors is well covered in Chapters 2 and 3 of this book, and we will focus hereafter upon the roles of individual chemokines and receptors in atherosclerosis. The largest amount of data on the roles of chemokines in cardiovascular disease (C VD) has been obtained from in vitro studies and murine models, which will be discussed in detail. In man, genetic polymorphisms in chemokine and chemokine-receptor genes have pointed to an important role for specific chemokines in various atherosclerotic diseases including coronary artery disease and carotid artery occlusive disease. For properties see Table 1. [Pg.200]

Ghilardi G, Biondi ML, Turri O, Guagnellini E, Scorza R. Internal carotid artery occlusive disease and polymorphisms of fractalkine receptor CX3CR1 a genetic risk factor. Stroke 2004 35(6) 1276-1279. [Pg.227]

Wholey MH, Jarmolowski CR, Eles G, Levy D, Buecthel J. Endovascular stents for carotid artery occlusive disease. J Endovasc Surg 1997 4 326-338. [Pg.566]

Fig. 15.1 la-d. A 62-year-old patient with symptomatic left internal carotid artery occlusion and progressive stroke. Initial DWI shows only punctuate small lesions in the deep borderzone of the left hemisphere (a), while the hypoperfused area on the time-to-peak maps affects the complete left middle cerebral artery territory and is most pronounced in the deep borderzone area (b). On day 3, the acute lesion has grown considerably larger, paralleling progression of symptoms (c). Contrast-enhanced MRA shows proximal occlusion of the left internal carotid artery (d)... [Pg.235]

Fig. 15.12. An asymptomatic 54-year-old patient suffering from left internal carotid artery occlusion (left) shows excellent collateralization via the posterior communicating artery and a sufficient filling of the ipsilateral middle cerebral artery (middle). As a consequence, perfusion MRI (time-to-peak) detects only a slight asymmetry and delay of contrast agent arrival in the parietal parts of the middle cerebral artery territory (right)... Fig. 15.12. An asymptomatic 54-year-old patient suffering from left internal carotid artery occlusion (left) shows excellent collateralization via the posterior communicating artery and a sufficient filling of the ipsilateral middle cerebral artery (middle). As a consequence, perfusion MRI (time-to-peak) detects only a slight asymmetry and delay of contrast agent arrival in the parietal parts of the middle cerebral artery territory (right)...
Bazner H, Hennerici M (2004) Georg Friedrich Handel s strokes. Cerebrovasc Dis 17 326-331 Bogousslavsky J, Regli F (1986a) Borderzone infarctions distal to internal carotid artery occlusion prognostic implications. Ann Neurol 20 346-350... [Pg.236]

CBF, Cerebral blood flow CCAo, common carotid artery occlusion cGMP, cyclic guanine monophosphate ICAM-1, intercellular adhesion molecule-1 ICAo, internal carotid artery occlusion MCAo, middle cerebral artery occlusion NAA, A-acetyl-aspartate NOS, nitric oxide synthase SD, Sprague-Dawley SH, spontaneously hypertensive SOD, superoxide dismutase TUNEL, transferase dUTP nick-end labeling. [Pg.48]

A study by Dietrich et al. (23) found that 3 h of immediate postischemic hypothermia (30°C) reduced CA1 loss at short (i.e., 3 and 7 d) but not long survival times (2 mo) after 10 min 2-VO ischemia in rats. Because all of the aforementioned studies (e.g., refs. 10-14) used short survival times, it was argued that postischemic hypothermia was, by itself, of no long-term benefit. Other work has confirmed the ephemeral nature of CA1 protection afforded by brief hypothermia (24). Likewise, a slow maturation of CA1 neuronal loss occurs following short duration (e.g., 5 min) 4-VO (vertebral cauterization + bilateral carotid artery occlusion) ischemia (25). In the 4-VO model the a-amino-3-hydroxy-... [Pg.80]

Lieberman AN, Bloom W, Kishore PS, Lin JP. Carotid artery occlusion following ingestion of LSD. Stroke 1974 5(2) 213-5. [Pg.564]

Rutgers DR, Klijn CJ, Kappelle LJ et al. (2004). Recurrent stroke in patients with symptomatic carotid artery occlusion is associated with high-volume flow to the brain and increased collateral circulation. Stroke 35 1345-1349... [Pg.48]

Baumgartner RW (1999). Transcranial color-coded duplex sonography. Journal of Neurology 246 637-647 Baumgartner RW, Mattie HP, Aaslid RC et al (1997). Transcranial colour-coded duplex sonography in arterial cerebrovascular disease. Cerebrovascular Diseases 7 57-63 Bishop CCR, Powell S, Insall MC et al (1986). Effect of internal carotid artery occlusion on middle cerebral artery blood flow at rest and in response to hypercapnia. Lancet i 710-712... [Pg.168]

Kleiser B, Widder B (1992). Course of carotid artery occlusions with impaired cerebrovascular reactivity. Stroke 23 171-174... [Pg.170]

Levi CR, Mitchell A, Pitt GC et al. (1996). The accuracy of magnetic resonance angiography in the assessment of extracranial carotid artery occlusive disease. Cerebrovascular Diseases 6 231-236... [Pg.170]

Vernieri F, Pasqualetti P, Matteis MC et al. (2001). Effect of collateral blood flow and cerebral vasomotor reactivity on the outcome of carotid artery occlusion. [Pg.171]

Warnock NG, Gandhi MR, Bergvall UC et al (1993). Complications of intra-arterial digital subtraction angiography in patients investigated for cerebral vascular disease. British Journal of Radiology 66 855-858 Widder B, Kleiser B, Krapf H (1994). Course of cerebrovascular reactivity in patients with carotid artery occlusions. Stroke 25 1963-1967... [Pg.171]

Sood R, Yang Y, Taheri S, Candelario-Jatil E, Estrada EY, Walker EJ, Thompson J, Rosenberg GA (2009) Increased apparent diffusion coefficients on MRI linked with matrix metalloproteinases and edema in white matter after bilateral carotid artery occlusion in rats. J Cereb Blood Elow Metab 29 308-316... [Pg.166]

It is clear that the initiation of the breakdown of the BBB occurs within minutes of insult as demonstrated by the use of either [3H] sucrose,27 infusion with hyperosmolar L(+)arabinose28 or horseradish peroxidase (HRP) tracer studies.29 However, the opening of the BBB continues over several hours after an ischaemic insult and at least two additional phases of BBB opening may occur.27 There is also evidence for a differential localisation of such openings between different parts of the brain. Thus, after bilateral carotid artery occlusion for 10 to 25 min, followed by recirculation, there is acute opening of the BBB in neocortical regions, possibly due to reactive hyperaemia,30 with recovery suggested to occur by 24 h.27 In the striatum and... [Pg.22]

Derdeyn CP, Videen TO, Simmons NR, et al. Count-based PET method for predicting ischemic stroke in patients with symptomatic carotid arterial occlusion. Radiology 1999 212 499-506. [Pg.177]


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Occlusion

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