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Carbon monoxide fetal

Carmines, E.L., Rajendran, N. (2008). Evidence for carbon monoxide as the major factor contributing to lower fetal weights in rats exposed to cigarette smoke. Toxicol. Set 102 383-91. [Pg.286]

Ginsberg MD, Myers RE. Fetal brain damage following maternal carbon monoxide intoxication An experimental study. Acta Obstet Gynec Scand 1974 53 309-17. [Pg.333]

The factors are diverse, ranging from caffeine consumption during pregnancy to maternal age at conception. The environmental agents with potential adverse impacts on fetal development for which there is the strongest evidence are all culture-dependent tobacco smoke, alcohol (ethanol), cocaine, and combustion-engine carbon monoxide.4 In this book I focus only on factors for which there is evidence known to me. What the factors all have in common is that all have heavy cultural... [Pg.265]

The toxicity of ethanol is enhanced in the presence of compounds such as barbiturates, carbon monoxide, and methyl mercury. With the latter compound, ethanol enhanced the retention of mercnry in the kidney of rats and thns increased nephrotoxicity (McNeil et al. 1988). When combined with cocaine and fed to rats, increased maternal and fetal toxicity was observed (Chnrch et al. 1988). Ethanol is reported to be synergistically toxic with caffeine (Pollard 1988) and with n-bntanol and isoamyl alcohol. Prior ethanol consnmption increased the toxicity... [Pg.137]

Root, W.S., 1965. Carbon monoxide. In Fenn, W.O., Rahn, H. (Eds.), Handbook of Physiology American Physiological Society, Washington, DC, pp. 1078-1098. Rosenberg, A.A., Harris, A.R, Koehler, R.C., et al., 1986. Role of 02-hemoglobin affinity in the regulation of cerebral blood flow in fetal sheep. Am. J. Physiol. [Pg.285]


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See also in sourсe #XX -- [ Pg.277 ]




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