Arsenic, poisoning

Inhibition of sulfhydril cellular enzymes with damage to GI tract walls and peripheral blood vessels [Pg.285]

Bowel movements - diarrhea Breath, odor - garlic Eyes, vision - dim [Pg.286]

vision - light sensitivity, increased (photophobia) [Pg.286]

Bone marrow suppression Cardiac dysrhythmias Coma Dermatitis Dehydration Encephalopathy Hepatic dysfunction Intestinal perforation Intrauterine fetal death [Pg.286]

Variable with exposure intensity and time to treatment [Pg.287]

Arsenic compounds must be considered extremely poisonous. Dust or fumes irritate mucous membranes and lead to arsenical poisoning. When swallowed they irritate the stomach and affect the heart, Hver, and kidneys. Nervousness, thirst, vomiting, diarrhea, cyanosis, and coUapse are among the symptoms of arsenical poisoning (3). In spite of the toxicity of arsenic compounds, there is evidence that arsenic is an essential nutrient for several animal species (4). [Pg.332]

Arsen-saure, /, arsenic acid, -silber, n, silver arsenide, -silberblende,/, proustite, -spiegel, m. arsenic mirror, -suifid, n, arsenic sulfide, esp. the pentasulfide. -sulfur, n. arsenic trisulfide, arsenic(III) sulfide, -iir, n, (-ous) arsenide, -verbindung, /. arsenic compound, -vergiftung, /. arsenic poisoning, -wasser-stoff, m. arsenic hydride, arseniuretted hydrogen (specif, arsine, AsHa). -zink, n. zinc arsenide. [Pg.34]

The "arsenic poison" referred to in true-crime dramas is actually the oxide of arsenic, As203, rather than the element itself. Less than 0.1 g of this white, slightly soluble powder can be fatal. The classic symptoms of acute arsenic poisoning involve various unpleasant gastroin-... [Pg.573]

In the modern forensic chemistry laboratory (Figure B) arsenic is detected by analysis of hair samples, where the element tends to concentrate in chronic arsenic poisoning. A single strand of hair is sufficient to establish the presence or absence of the element. The technique most commonly used is neutron activation analysis, described in Chapter 19. If the concentration found is greater than about 0.0003%, poisoning is indicated normal arsenic levels are much lower than this. [Pg.573]

Lasch, F. 1958. Clinic and therapy in severe arsenic poisoning. Med. Klin. 53 787-789. [Pg.117]

Landrigan, P.J., R.J.Castello, and W.T.Stringer. 1982. Occupational exposure to arsine. An epidemiologic reappraisal of current standards. Scand. J. Work Environ. Health. 8 169-177. Legge, T.M. 1916. Arsenic poisoning. In Diseases of Occupations and Vocational Hygiene, G.M.Kober and W.Hanson, eds. Philadelphia, PA Blakiston. [Pg.117]

Suggested Alternatives for Differential Diagnosis Bovine viral diarrhea, East Coast fever, foot-and-mouth disease, infectious bovine rhinotracheitis, malignant catarrhal fever, vesicular stomatitis, paratuberculosis, and arsenic poisoning. [Pg.573]

Episodes of arsenic poisoning are either acute or subacute cases of chronic arsenosis are rarely encountered, except in humans. [Pg.1505]

Research results on arsenic poisoning in mammals (Table 28.6) show general agreement on eight points ... [Pg.1522]

Accumulations of 1 mg/kg BW daily for 3 months in children, or 80 mg/kg BW daily for 3 years in adults produced symptoms of chronic arsenic poisoning... [Pg.1525]

Tissue residues Residues, in mg total As/kg FW, liver or kidney in 2-10 range are considered elevated residues >10 mg/kg are indicative of arsenic poisoning (Goede 1985)... [Pg.1530]

Chronic arsenic poisoning Lifetime cumulative absorption of 1 g As, or intake of 0.7-2.6 g/year for several years (in medications) can produce symptoms after latent period of 4-24 years (NRCC 1978)... [Pg.1532]

Choprapawon, C. and A. Rodcline. 1997. Chronic arsenic poisoning in Ronpibool Nakhon Sri Thammarat, the southern province of Thailand. Pages 69-77 in C.O. Abernathy, R. Calderon, and W.R. Chappell (eds.). Arsenic. Exposure and Health Effects. Chapman Hall, London. [Pg.1535]

Samad, M.A. and A. Chowdhury. 1984. Clinical cases of arsenic poisoning in cattle. Indian Jour. Vet. Med. 4 107-108. [Pg.1540]

Selby, L.A., A.A. Case, G.D. Osweiler, and H.M. Hages, Jr. 1977. Epidemiology and toxicology of arsenic poisoning in domestic animals. Environ. Health Perspec. 19 183-189. [Pg.1540]

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