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Arsenic poisoning epidemiology

Landrigan, P.J., R.J.Castello, and W.T.Stringer. 1982. Occupational exposure to arsine. An epidemiologic reappraisal of current standards. Scand. J. Work Environ. Health. 8 169-177. Legge, T.M. 1916. Arsenic poisoning. In Diseases of Occupations and Vocational Hygiene, G.M.Kober and W.Hanson, eds. Philadelphia, PA Blakiston. [Pg.117]

Selby, L.A., A.A. Case, G.D. Osweiler, and H.M. Hages, Jr. 1977. Epidemiology and toxicology of arsenic poisoning in domestic animals. Environ. Health Perspec. 19 183-189. [Pg.1540]

Borgono, J.M. and Greiber, R. (1971) Epidemiological study of arsenic poisoning in the city of Antofagasta. Revista Medica Chile, 99, 702-07. [Pg.341]

On the one hand there are relatively simple procedures that allow a quick determination of the sum of inorganic arsenic, i.e. As(lll) and As(V). MMA and DMA for epidemiological studies, occupational exposure, intoxication, and poisoning. On the other hand, more complex and detailed procedures are required for scientific investigations on arsenic metabolism in mammals and man. [Pg.306]

As Table 3.1 indicates, at least seven metals have been identified as human carcinogens, primarily of the lung. This may lead to the conclusion that these metals are extremely dangerous, and that any contact may result in cancer. On the contrary, based on available epidemiological evidence, it appears that the majority of metal-associated cancers are the result of chronic overexposure over a period of years or decades. With the exception of arsenic and lead, metal-induced cancers are largely preventable through the use of proper environmental controls or respiratory protective equipment. Surprisingly, two of the seven metals listed in Table 3.1, chromium and cobalt, are required in minute quantities as essential nutrients. Three others (arsenic, cadmium, and lead) may also be essential dietary nutrients. As Paracelsus indicated, it is not the substance that makes the poison, but the dose. [Pg.37]


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See also in sourсe #XX -- [ Pg.115 ]




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