Acute laryngitis

Srinivasan V, Lanham PR. Acute laryngeal obstruction -reaction to intravenous hydrocortisone Eur J Anaesthesiol 1997 14(3) 342. 

Acute laryngeal dystonia is probably under-reported but it is potentially lethal and can mimic anaphylaxis (204). It can occur within hours to days of starting therapy with neuroleptic drugs and is characterized by sudden onset of difficulty in breathing and swallowing (SEDA-22, 51). 

Koek RJ, Pi EH. Acute laryngeal dystonic reactions to neuroleptics. Psychosomatics 1989 30(4) 359-64. 

Fines RE, Brady WJ Jr, Martin ML. Acute laryngeal dystonia related to neuroleptic agents. Am J Emerg Med 1999 17(3) 319-20. 

Christodoulou C, Kalaitzi C. Antipsychotic drug-induced acute laryngeal dystonia two case reports and a mini review. J Psychopharmacol 2005 19 307-12. 

Most of us will experience a bout of acute laryngitis in our lifetimes. Whether brought on by a cold or flu or simply talking too much, the cure is the same rest the... 

Anaphylactic shock has been described after intranasal hydrocortisone acetate, intramuscular methylprednisolone (SEDA-21, 419) (251), intravenous methylprednisolone (SEDA-22, 448) (252), intramuscular dexamethasone (SEDA-22, 448) (253), and intra-articular methylprednisolone (SEDA-22, 449) (254). A life-threatening anaphylac-tic-like reaction to intravenous hydrocortisone has been described in patients with asthma (255). Acute laryngeal obstruction has been described for the first time after the intravenous administration of hydrocortisone (SEDA-22, 449) (256). There is some reason to believe that sodium succinate esters are more likely to cause hypersensitivity reactions (SEDA-17, 449), but unconjugated glucocorticoids can definitely produce allergy in some cases (SEDA-16, 452). 

Maibach H. Acute laryngeal obstruction presumed secondary to thiomersal (merthiolate) delayed hypersensitivity. Contact Dermatitis 1975 l(4) 221-2. 

Upper respiratory tract disorders are diagnosed as upper respiratory infections (URIs) and include acute rhinitis, sinusitis, acute tonsillitis, and acute laryngitis. Do not confuse acute rhinitis with allergic rhinitis. 

Stridor - A harsh, high -pitched respiratory sound such as the inspiratory sound often heard in acute laryngeal obstruction. 

Acute laryngitis is an infection that causes swelling (edema) of the vocal cords. This results in the patient having a weak or hoarse voice. Acute laryngitis can be caused by a viral infection. Other times it is caused by stress or ovemse of the vocal cords—a common occurrence for fans whose team wins the Super Bowl. 

Refraining from speaking and avoiding exposure to substances that can irritate the vocal cords, such as smoking, is the preferred treatment for acute laryngitis. 

Acute laryngitis is an infection that causes swelling (edema) of the vocal cords... 

Contact dermatitis complicated by systemic symptoms, e. g. acute laryngeal obstruction (Maibach 1975 a), gastrointestinal symptoms (Mathias et al. 1979 b), and paresthesia (Fisher 1979). 

Maibach HI (1971) Allergic sensitization potential (Draize test) in man of several preservatives. Contact Dermatitis Newslett 9 213 Maibach HI (1975a) Acute laryngeal obstruction presumed secondary to thiomersal (mer-thiolate) delayed hypersensitivity. Contact Dermatitis 1 221-222 Maibach HI (1975 b) Glutaraldehyde cross reactions to formaldehyde. Contact Dermatitis 1 326-327... 

Craig TJ, Wasserman RL, Levy RJ, Bewtra AK, Schneider L, Packer F, Yang WH, Keinecke HO, Kiessling PC. Prospective study of rapid relief provided by Cl esterase inhibitor in emergency treatment of acute laryngeal attacks in hereditary angioedema. J Clin Immunol 2010 30(6) 823-9. 

Accidental exposure to liquid fuming sulfuric acid can result in skin bums as well as pulmonary edema from inhalation. Pulmonary fibrosis, residual bronchitis, and pulmonary emphysema have also been reported. A single overexposure to sulfuric acid may lead to acute laryngeal, tracheobronchial, and pulmonary edema. Concentrations of around 5 mg/ m have been found quite objectionable, often causing coughs and respiratory dysfunctions. The data in Table 35.2 summarize human responses to various levels of concentration of sulfuric acid mist. 

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