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Total flow resistance

We can distinguish between two types of stresses on an interface a shear stress and a dilatational stress. In a shear stress experiment, the interfacial area is kept constant and a shear is imposed on the interface. The resistance is characterized by a shear viscosity, similar to the Newtonian viscosity of fluids. In a dilatational stress experiment, an interface is expanded (dilated) without shear. This resistance is characterized by a dilatational viscosity. In an actual dynamic situation, the total stress is a sum of these stresses, and both these viscosities represent the total flow resistance afforded by the interface to an applied stress. There are a number of instruments to study interfacial rheology and most of them are described in Ref. [1]. The most recent instrumentation is the controlled drop tensiometer. [Pg.2]

Here, Rao denotes the total flow resistance for each of the two nephrons in equilibrium. ri and ri are the normalized radii of the active parts of the afferent arterioles for nephron 1 and nephron 2, respectively, and Pg and Pg2 are the corresponding glomerular pressures. As a base value of the hemodynamic coupling parameter we shall use s = 0.2. This parameter measures the fraction of the arteriolar length that is shared between the two nephrons. [Pg.338]

Viscous (Poiseuille) flow and molecular diffusion are non-selective. Nevertheless they play an important role in the macroporous substrate(s) supporting the separation layer and can seriously affect the total flow resistance of the membrane system. Mesoporous separation layers or supports are frequently in the transient-regime between Knudsen diffusion (flow) and molecular diffusion, with large effects on the separation factor (selectivity). [Pg.334]

For each set of 100 realizations, first flow and then advective transport were solved for the boundary conditions shown in Figure 1. With the advective transport solution, the total flow resistance is computed and compared between at... [Pg.246]

Response times of 15-40 ms seem to be currently in use. Fry et al. (1957) analyzed the dynamic characteristics of three types of commercially available, differential-pressure pneumotachographs which employed concentric cylinders, screen mesh, and parallel plates for the air resistors. Using a high-quality, differential-pressure transducer with each, they measured total flow resistance ranging from 5 to 15 cm HjO. Frequency response curves taken on one model showed fairly uniform response to 40 Hz the second model showed a slight increase in response at 50 Hz, and the third exhibited a slight drop in response at this frequency. [Pg.265]

Besides the pressure drop inside the columns there can be an additional pressure loss due to the piping and the valves between the columns. In experiments, a large pressure drop was found in an SMB-SFC apparatus due to these flow resistances [55]. Taking into account the pressure drop as a function of mass flow rates, a pressure-loss coefficient f for the total flow resistance (analogous to pipeline construction) between two columns is determined by fitting to experimental pressure drops. Then the pressure drop can be calculated from ... [Pg.307]

P-Adrenoceptor Blockers. There is no satisfactory mechanism to explain the antihypertensive activity of P-adrenoceptor blockers (see Table 1) in humans particularly after chronic treatment (228,231—233). Reductions in heart rate correlate well with decreases in blood pressure and this may be an important mechanism. Other proposed mechanisms include reduction in PRA, reduction in cardiac output, and a central action. However, pindolol produces an antihypertensive effect without lowering PRA. In long-term treatment, the cardiac output is restored despite the decrease in arterial blood pressure and total peripheral resistance. Atenolol (Table 1), which does not penetrate into the brain is an efficacious antihypertensive agent. In short-term treatment, the blood flow to most organs (except the brain) is reduced and the total peripheral resistance may increase. [Pg.141]

Methyldopa. Methyldopa reduces arterial blood pressure by decreasing adrenergic outflow and decreasing total peripheral resistance and heart rate having no change in cardiac output. Blood flow to the kidneys is not changed and that to the heart is increased. It causes regression of myocardial hypertrophy. [Pg.142]

Equation 46 states that when complete pore blockage occurs, the intensity of the increase in the total resistance with increasing filtrate volume is proportional to the square of the flow resistance. [Pg.175]

From Darcy s equation we can determine a formula for the counterforce produced by the porous material to the flowing or diffusing component A, If this counterforce is found, it can be added to the diffusion resistance force caused by component B to component A hence the sum of these two forces represents the total diffusion resistance. [Pg.138]

The discharge pressure developed hy the compressor must he equal to the process gas s total system resistance, of control valves, hand valves, orifices, heat exchangers, and any other process-related devices through which the discharge gas from the compressor must flow. As this resistance changes, the gas flow through the compressor will automatically adjust itself to equal the new resistance. ... [Pg.482]

In order to analyze the total system resistance and its relation to fen performance. Figure 12-140 is used. Without defining what comprises the system resistance, but representing it by Curve A-A, this system is to flow 13,000 cfrn of air at 1.1 in. static pressure. A fan has been selected that operates at 600 rpm and is represented by its static pressure Curve C-C. The intersection of these two curves. Point 1, is the only point of... [Pg.563]

The effects of total flow rate of fuels (CO2/CH4 = 1) on the impedance in the internal reforming of CH4 by C02 0ver ESC (NiO-YSZ- Ce02 I YSZ I (LaSr)Mn03) of SOFC system are represoited in Figure 6. It was comsidaed fliat the total resistance was dependent on the total flow rate because the conva sions of CO2 and CH4 over ESC were affected by contact time in the internal reforming system. [Pg.620]

Of major importance is the fact that the specific character of polymer chains of a given type enters the relationship (17) only through the effective size of one of its beads as indicated by the ratio f/970. Even the effect of this factor vanishes when the total internal resistance to flow is sufficiently large. Hence, in this limit, which will include nearly all actual cases of interest (see Sec. 4), the molecular frictional coefficient should depend only on the size /s and not otherwise on the nature of the polymer. Accordingly, we choose to let... [Pg.609]

Total peripheral resistance (TPR) is the resistance to blood flow offered by all systemic vessels taken together, especially by the arterioles, which are the primary resistance vessels. Therefore, MAP is regulated by cardiac activity and vascular smooth muscle tone. Any change in CO or TPR causes a change in MAP. The major factors that affect CO, TPR, and therefore MAP, are summarized in Figure 15.3, as well as in Table 15.1. These factors may be organized into several categories and will be discussed as such ... [Pg.202]

Notes CO cardiac output VR venous return HR heart rate SV stroke volume EDV end-diastolic volume ESV end-systolic volume O blood flow AP pressure gradient R resistance r vessel radius P systolic pressure Piiastoik- diastolic pressure MAP mean arterial pressure TPR total peripheral resistance, P venous pressure Era- right atrial pressure Rv venous resistance. [Pg.204]

Loss of plasma volume leads to a decrease in MAP. Baroreceptors located in the aortic and carotid sinuses detect this fall in MAP and elicit reflex responses that include an increase in the overall activity of the sympathetic nervous system. Sympathetic stimulation of the heart and blood vessels leads to an increase in cardiac output (CO) and increased total peripheral resistance (TPR). These adjustments, which increase MAP, are responsible for the short-term regulation of blood pressure. Although increases in CO and TPR are effective in temporary maintenance of MAP and blood flow to the vital organs, these activities cannot persist indefinitely. Ultimately, plasma volume must be returned to normal (see Table 19.1). [Pg.332]

The pitot tube is a relatively complex device and requires considerable effort and time to obtain an adequate number of velocity data points and to integrate these over the cross section to determine the total flow rate. On the other hand the probe offers minimal resistance to the flow and hence is very efficient from the standpoint that it results in negligible friction loss in the conduit. It is also the only practical means for determining the flow rate in very large conduits such as smokestacks. There are standardized methods for applying this method to determine the total amount of material emitted through a stack, for example. [Pg.295]

The diameter of the copper slabs is about a centimetre smaller than the inner diameter of the stainless steel housing, leaving a channel for the helium flow. The top of housing has a conical shape, to reduce the amount of 3He necessary to have the phase separation in the right position. The total Kapitza resistance at 20 mK is about 45K/W (e.g. a heat leak of 50 xW on the cold plate of the mixing chamber would give a temperature difference of about 2 mK between the liquid and the cold plate). [Pg.356]

Generally, the absolute magnitude of Q is not as important as the ratio leak rate to the total flow rate Qieaf/QtotJ. The leakage rate given by Equation (5.4) is the volume flow rate at the temperature and pressure of the leakage flow, and must be corrected to standard conditions for comparison with reactant feed rates. The total required flow rate of fuel or air to the stack is proportional to the stack current, which increases with the electrochemically active area and is inversely proportional to the cell area specific resistance (R"). [Pg.221]

Snell, R. E., and P. C. Luchsinger. Effects of sulfur dioxide on expiratory flow rates and total respiratory resistance in normal human subjects. Arch. Environ. Health 18 693-698, 1969. [Pg.414]


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See also in sourсe #XX -- [ Pg.338 ]




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