Streptococcus salivarious

Staphylococcus aureus Staphylococcus camosus Staphylococcus epidermidis Streptococcus agalactiae Streptococcus anginosus Streptococcus avium Streptococcus cremoris Streptococcus dorans Streptococcus equisimilis Streptococcusfaecalis Streptococcus ferus Streptococcus lactis Streptococcus ferns Streptococcus mitior Streptococcus mutans Streptococcus pneumoniae Streptococcus pyogenes Streptococcus salivarious Streptococcus sanguis Streptococcus sobrinus Streptococcus thermophylus... 

MaePherson, L. M. D., and Dawes, C. (1991). Urea concentration in minor mucus gland secretions and the effect of salivary film velocity on urea metabolism by streptococcus vestibularis in an artificial plaque./Periodont. Res. 26, 395- 01. 

A freshly cleaned tooth surface quickly becomes coated with a thin pellicle of salivary proteins. This provides a surface for growth of dental plaque, which contains many bacteria and adhesive polysaccharides such as dextrans.1 The latter are generated from dietary sucrose by such bacteria as Streptococcus mutans. (Chapter 20) and others.131 Many factors affect the probability of tooth decay. 

Polyphenolic catechins and theaflavins exist as either monomers or polymers in green and black teas, respectively, and are both well known to be inhibitory and bactericidal against Streptococcus spp. They bind to surface proteins of the bacteria, and induce aggregation and inhibit the enzymatic activity of glucosyl transferase. Both teas inhibit salivary and streptococcal amylase, overall, leading to reduction in plaque. Studies in animals have shown that tea consumption reduces caries, and it has been associated with lower caries levels in humans. ... 

Alaluusa A Streptococcus mutans establishment and changes in salivary IgA in young children with reference to dental caries. Proc Finn Dent Soc 1983 79(suppl 3) 1-55. 

Challacombe SJ Serum and salivary antibodies to streptococcus mutans in relation to the development and treatment of human dental caries. Arch Oral Biol 1980 25 495-502. 

Zahradnik RT, Propas D, Moreno EC In vitro enamel demineralization by streptococcus mutans in the presence of salivary pellicles. J Dent Res 1977 56 1107-1110. 

Schilling KM, Bowen WH Glucans synthesised in situ in experimental salivary pellicle function as specific binding sites for Streptococcus mutans. Infect Immun 1992 60 284-295. 

Gibbons RJ, Hay DI Adsorbed salivary acidic proline-rich proteins contribute to the adhesion of Streptococcus mutans IBP to apatitic surfaces. J Dent Res 1989 68 1303-1307. 

Gong K, Mailloux L, I ler/berg MC Salivary film expresses a complex, macromolecular binding site for Streptococcus sanguis. J Biol Chem 2000 275 8970-8974. 

Scannapieco FA, Beigey EJ, Reddy MS, Levine MJ Characterization of salivary a-amylase binding to Streptococcus sanguis. Infect Immun 1989 57 2853-2863. 

Gibbons RJ, Hay DI, Schlesinger DH Delineation of a segment of adsorbed salivary acidic pro-line-rich proteins which promotes adhesion of Streptococcus gordonii to apatitic surfaces. Infect Immun 1991 59 2948-2954. 

This chapter describes dental caries (tooth decay) and its causes. Sucrose and other mono- and disaccharides are metabolized to acid (lactate) by bacteria that remain in stagnation areas of the teeth. Rats and hamsters fed a 50% sucrose diet developed a caries-sensitive, predominantly gram-positive microbiota that became caries resistant when the rodents were fed penicillin (Sect. 1). Further studies identified Streptococcus mutans (S. mutans) as the etiological agent. This organism synthesizes an insoluble polysaccharide capsule that is stable and retains lactate at the enamel surface (Sect. 2). The key enzyme, glucosyl transferase, is related to salivary amylase which adheres to oral bacteria and enhances bacterial acid production. The chapter concludes with a discussion of salivary and other factors responsible for the marked variation observed in individual caries experience (Sect. 3). 

KATZ, J HARMON, C.C., BUCKNER, G.P., RICHARDSON, G.J., RUSSELL, M.W. MICHALEK, S.M. (1993) Protective salivary immunoglobulin A responses against Streptococcus mutans infection after intranasal immunization with S. mutans antigen I/II coupled to the B subunit of cholera toxin. Infection and Immunity, 61, 1964—1971. 

Ligtenbeig AJ, Veerman EC, Nieuw Amerongen AV (2000) A role for Lewis a antigens on salivary agglutinin in binding to Streptococcus mutans. Antonie Van Leeuwenhoek 77 21-30... 

Murray, P. A., Levine, M. J., Tabak, L. A., and Reddy, M. S., 1982, Specificity of salivary-bacterial interactions II. Evidence for a lectin on Streptococcus sanguis with specificity for a Neu5Aca2,3Gaipi, 3GalNAc sequence, Biochem. Biophys. Res. Commun. 106 390-396. 

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