Precordial pain

Ergotamine tartrate Nausea and vomiting occur in up to 10% of patients. Numbness and tingling of fingers and toes muscle pain in the extremities pulselessness weakness in the legs precordial pain transient tachycardia or bradycardia localized edema itching. 

Dehydroemetine inhibits protein synthesis it may cause pain at the site of injection, weakness and muscular pain, hypotension, precordial pain and cardiac dysrhythmias. 

A 53-year-old man was given diltiazem for precordial pain and about 2 hours later developed an erythematous maculopapular rash mainly on the trunk and lower limbs. Four days later he developed abdominal pain radiating to both renal angles, accompanied by dysuria and tenesmus and followed 6 days later by acute renal insufficiency associated with raised liver function test results. 

In 70 patients with psoriasis treated with an ointment containing ammoniated mercury, symptoms and signs of mercury poisoning were detected in 33 (8) albuminuria, headache, gingivitis, erythroderma, nausea, dizziness, precordial pain, contact dermatitis, conjunctivitis, epistaxis, keratitis, tremor, neuritis, hematological changes, metallic taste in mouth, and purpura. 

Figure 3.26 A 46-year-old patient with dubious precordial pain. The ECG (B) presented very discrete changes in V2-V3 leads (slightly negative U wave with somehow positive T wave). These small changes are significant when compared...

The ECG pattern of ST-segment elevation (subepicardial injury) is found in IHD, but also in other situations as well. In the second part we will comment that the presence of clinical signs of ischaemia (precordial pain, etc.) and the presence of ST-segment elevation of the characteristics explained here (typical and atypical patterns - see Table 8.1) constitute the clinical syndrome known as ACS with ST-segment elevation (STE-ACS), which has different clinical and ECG characteristics (Tables 8.1 and 8.2) than ACS without ST-segment elevation (NSTE-ACS). However, in both clinical syndromes (STE-ACS and NSTE-ACS), there are... 

Figure 4.49 (A) A 39-year-old patient with long-standing precordial pain without ischaemic characteristics. There is an ST-segment elevation in many leads and in someone with final negative T wave but without Q waves and with PR elevation in VR with depression in II. The clinical history,...

Figure 4.58 Patient with atypical precordial pain and a clearly positive exercise test (marked ST-segment depression) without pain during the test. The SPECT test...

Figure 4.64 (A) Patient with precordial pain at exercise depression with the presence of the same pain. (C) The...

Figure 8.7 A 45-year-old patient with an acute precordial pain who presents a tall T and peaked T wave that follows a rectified ST segment in right precordial leads as the only ECG sign suggestive of ischaemia (A). Some minutes later ST-segment elevation appears (B) and very soon becomes very evident, accompanied by R-wave increase and S-wave decrease (C). The ECG pattern recorded in (C) presents...

C) Some hours after PCI the patient presented precordial pain with pseudonormalisation of ECG as a sign of... 

Figure 8.25 Exercise test of a patient with doubtful precordial pain and frequent (A) ventricular and (B) supraventricular premature beats. Observe that ST-segment depression was little evident in sinus rhythm complexes, while it was very significant in premature complexes (see V3 and V4 in (B) and V5 and V6 in (A)). (C) The patient presented severe three-vessel disease.

Figure 11.3 Exercise test in a patient with precordial pain. elevation, accompanied by precordial pain (C), which was Before the exercise test (A) and during it (B), ST segment is followed by advanced AV block (D). normal. At the end, there is an important ST-segment...

Nasal irritation (respiratory exposure) Precordial pain... 

Cardiac arrhythmias precordial pain muscle weakness cellulitis at site of injection diarrhea vomiting peripheral neuropathy heart failure headache dyspnea Diethylcarbamazine Citrate USP... 

ACUTE HEALTH RISKS. irritation of skin, eyes, and respiratory system pulmonary edema tightening of chest shortness of breath precordial pain nonproductive cough nausea headache lightheadedness vertigo chills fever muscle twitching painful muscle spasms local inflammation of skin blisters redness swelling. 

Exposure Routes, Symptoms, Target Organs (see Table 5) ER Inh SY Chest tight, precordial pain, short breath, nonproductive cough, nau head, dizz, chills, fever, lass, tremor, musefase in animais iiver, kidney damage pulm edema hemorr TO Resp sys, CNS, liver, kidneys First Aid (see Table 6) Breath Resp support ... 

Chlorpropamide is used in insulin-deficient states (diabetes meUitus) and vasopressin-deficient states (diabetes insipidus) but its mode of action in these two conditions is not thought to be identical. Peak and Horni-blow (43 ) describe recurrent hyponatrae-mic phases (with levels as low as 113 mmol/ 1) whenever more than 250 mg chlorpropamide was given to a diabetic woman who refused to use insulin. Even the combination of 250 mg chlorpropamide and 50 mg phen-formin appeared to have elicited hyponatrae-mia. The patient also had precordial pain and was decompensated, a factor which may have contributed to the toxic effect of chlorpropamide. Diuretics aggravated the hypona-traemia. Treatment with glibenclamide and frusemide achieved control of the diabetes without hyponatraemic episodes (see 44 ). 

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