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Oxygen-derived free radicals

WETTASiNGHE M and SHAHiDi F (1999) Evening Primrose Meal A source ofnatural antioxidants and scavenger of hydrogen peroxide and oxygen-derived free radicals, JAgric Food Chem, 47, 1801-12. [Pg.346]

Alpha-l-antiprotease (ai-AP) limits tissue damage arising from the actions of the leucocyte protease, elastase (Carrell and Travis, 1985), and there is much evidence available for the oxidative inactivation of this protein by oxygen-derived free-radical species and hypochlorous acid/hypochlorite anion (HOCl/OCP). The mechanism of this inactivation appears to involve the oxidation of a critical methionine residue (Met-358) to its corresponding sulphoxide and methionine sulphoxide has been detected in ai-AP samples isolated from the lungs of cigarette smokers (Carp et al., 1982) and rheumatoid synovial fluids (Wong and Travis, 1980). [Pg.4]

Bernier, M., Hearse, D.J. and Manning, A.S. (1986). Reperfusion-induced arrhythmias and oxygen-derived free radicals. Studies with anti-free radical interventions and a free radical-generating system in the isolated perfused rat heart. Circ. Res. 58, 331-340. [Pg.69]

Pallandi, R.T., Perry, M.A. and Campbell, T.J. (1987). Proar-rhythmic efects of an oxygen-derived free radical generating system on action potentials recorded from guinea pig ventricular myocardium possible cause of reperfusion-induced arrhythmias. Circ. Res. 61, 50-54. [Pg.72]

Fantone, J.C. and Ward, P.A. (1982). Role of oxygen-derived free radicals and metabolites in leukocyte-dependent infiam-matory reactions. Am. J. Pathol. 107, 397-418. [Pg.81]

McCord, J.H. (1985). Oxygen-derived free radicals in post-ischaemic tissue injur). N. Engl. J. Med. 312, 159-163. [Pg.95]

Arthur, M.J.P., Bentley, I., Kowalski-Saunders, P., Tanner, A.K, MiUward-Sadler, G.H. and Wright, R. (1985). Oxygen-derived free radicals promote hepatic injury in the rat. Gastroenterology 89, 1114-1122. [Pg.161]

Atalla, S.L., Toledo-Pereyra, L.H., MacKenzie, G.H. and Cederna, J.P. (1985). Influence of oxygen derived free radical scavengers on ischaemic livers. Transplantation 40, 584-590. [Pg.161]

Basso, D., Panozzo, M.P., Fabris, C., Del Favero, G., Meggiato, T., Fogar, P., Meani, A., Faggian, D., Plebani, M., Burlina, A. and Naccarato, R. (1990). Oxygen derived free radicals in patients with chronic pancreatic and other digestive diseases. J. Clin. Pathol. 43, 403-405. [Pg.161]

Parks, D.A., Bulkley, G.B. and Granger, D.N. (1983). Role of oxygen-derived free radicals in digestive tract diseases. Surgery 94, 415-422. [Pg.169]

Salim, A.S. (1990a). Removing oxygen-derived free radicals stimulates healing of ethanol-induced erosive gastritis in the rat. Digestion 47, 24—28. [Pg.170]

Salim, A.S. (1990c). Oxygen-derived free radicals and the prevention of duodenal ulcer relapse, a new approach. Am. J. Med. Sci. 300, 1-6. [Pg.170]

Salim, A.S. (1991). Role of oxygen-derived free radical scavengers in the treatment of recurrent pain produced by chronic pancreatitis. A new approach. Arch. Surg. 126, 1109-1114. [Pg.170]

Salim, A.S. (1992b). Use of scavenging oxygen-derived free radicals to protect rat gainst aspirin- and ethanol-induced erosive gastritis. J. Pharm. Sci. 81, 943-946. [Pg.170]

Salim, A.S. (1992d). Role of oxygen-derived free radicals in mechanism of acute and chronic duodenal ulceration in the rat. D. Dis. Sci. 35, 73-79. [Pg.170]

Saluja, A., Powers, R.E., Saluja, M., Rutledge, P. and Steer, M.L. (1986). The role of oxygen derived free radicals in the pathogenesis of acute pancreatitis. Gastroenterology 90, A1613. [Pg.170]

Vaananen, P.M., Meddings, J.B. and Wallace, J.L. (1991). Role of oxygen derived free radicals in indomethacin-induced gastric injury. Am. J. Physiol. 261, G470-G475. [Pg.172]

Wisner, J., Green, D., Ferrell, L. and Renner, 1. (1988). Evidence of the role of oxygen derived free radicals in the pathogenesis of caerulein induced acute pancreatitis in rats. Gut 29, 1516-1523. [Pg.173]

PMNs also produce oxygen-derived free radicals when exposed in vitro to chrysotile or crocidolite. The highly reactive superoxides could injure other cells or attack extracellular matrix molecules. Hyaluronic acid (part of the mucopolysaccharide molecule) is a particularly vulnerable matrix species, but the specific reaction mechanisms have not been elucidated. [Pg.123]

Kinins, neuropeptides, and histamine are also released at the site of tissue injury, as are complement components, cytokines, and other products of leukocytes and platelets. Stimulation of the neutrophil membranes produces oxygen-derived free radicals. Superoxide anion is formed by the reduction of molecular oxygen, which may stimulate the production of other reactive molecules such as hydrogen peroxide and hydroxyl radicals. The interaction of these substances with arachidonic acid results in the generation of chemotactic substances, thus perpetuating the inflammatory process. [Pg.796]

Garrett, I.R., Boyce, B.F., Oreffo, R.O.C., Bonewald, L., Pser, J., and Mundy, G.R. 1990. Oxygen-derived free radicals stimulate osteoclastic bone resorption in rodent bone in vitro and in vivo. J. Clin. Invest. 85, 632—639. [Pg.154]

Key, L.L., Ries, W.L., Taylor, R.G., Hays, B.D., and Pitzer, B.L. 1990. Oxygen derived free radicals in osteoclasts The specificity and location of the nitroblue tetrazolium reaction. Bone 11, 115-119. [Pg.156]

Ascorbic Acid (AA). Experimental (SI, S7, S24) and clinical (B13, B17) studies have provided some evidence for the concept that oxidative stress is the common pathway for the initiation of AP (B14). The most abundant endogenous antioxidant in the aqueous phase is ascorbic acid (AA), a bioactive form of vitamin C, which scavenges oxygen-derived free radicals produced by activated neutrophils and the hypoxanthine-xanthine oxidase system (D12). Scott et al. [Pg.64]

Sanfey, H., Bulkley, G. B., and Cameron, J.L., The pathogenesis of acute pancreatitis. The source and role of oxygen-derived free radicals in three different experimental models. Ann. Surg. 201, 633-639 (1985). [Pg.79]

Among the oxygen-derived free radicals, the species of primary concern include superoxide anion (O -), hydrogen peroxide (H202), and hydroxyl radical (OH-). The superoxide is further converted in peroxynitrite (ONOO-) by reacting with nitric oxide. [Pg.412]

Au, A. M., Chan, P. H., and Fishman, R. A. (1985). Stimulation of phospholipase A2 activity by oxygen-derived free radicals in isolated brain capillaries. J. Cell Biochem. 27, 449-453. [Pg.419]

Saari, H., Oxygen derived free radicals and synovial fluid hyaluronate, Ann. Rheum. Disease, 50, 389, 1991... [Pg.274]

Greenwald, R.A. and Moy, W.W., Effect of oxygen-derived free radicals on hyaluronic acid, Arthritis. Rheum., 23, 455,1980. [Pg.274]


See other pages where Oxygen-derived free radicals is mentioned: [Pg.11]    [Pg.1]    [Pg.4]    [Pg.5]    [Pg.5]    [Pg.54]    [Pg.54]    [Pg.87]    [Pg.271]    [Pg.285]    [Pg.272]    [Pg.98]    [Pg.45]    [Pg.148]    [Pg.807]    [Pg.25]    [Pg.312]   
See also in sourсe #XX -- [ Pg.127 ]




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