Cellular interface

The problem of interface morphology (planar or curved interface, cellular structure unstationary shape, chaotic, turbulent) as a function of the control parameters. 

Figure 35.6 Calcium phosphate solubility at a solid-fluid interface. Cellular metabolic activity, by producing a local change in the pH, determines the distribution of calcium and phosphate between the solid phase and tissue fluid...

Fujioka, H., Thakur, R., Wang, G. J., Mizuno, K., Balian, G., and Hurwitz, S. R. 1998. Comparison of surgi-caUy attached and non-attached repair of the rat Achilles tendon-bone interface. Cellular organization and type X collagen expression. Connect. Tissue Res. 37 205-218. 

To be biocompatible is to interact with all tissues and organs of the body in a nontoxic manner, not destroying the cellular constituents of the body fluids with which the material interfaces. In some appHcations, interaction of an implant with the body is both desirable and necessary, as, for example, when a fibrous capsule forms and prevents implant movement (2). 

Figure 9.1. Decanied interface of cellularly solidified Pb-Sn alloy. Magnification xl50 (after...

Epithelium Cellular layer interfacing with external environment which con-... 

The initial development of a cellular structure from an originally flat interface has been at least partially understood [130]. Let us look only at the large-wavelength A limit (for more details see [122]). In the numerical calculations it was found [123] that for fixed cell-spacing A at increasing velocity a tail instability occurs. A side branch in the groove between two... 

N. Noel, H. Jamgotchian, B. Billia. In situ and real-time observation of the formation and dynamics of a cellular interface in a succinonitrile-0.5 wt. J Cryst Growth 181 111, 1997. 

Figure 5.2. A cellular automata model of the interface between two immiscible liquids, after the demixing process has reached an equilibrium...

Figure 5. Schematic of nonorthogonal transformations used in finite-element/Newton algorithms for calculating cellular interfaces, (a) Monge cartesian representation for almost planar interfaces, (b) Mixed cylindrical/cartesian mapping for representing deep cells.

Figure 7. Families of cellular interfaces computed for System I with k = 0.865 as a function of increasing P in a A /2 sample size. The cells are represented by the dimensionless arc length. The letters refer to sample interface shapes shown in Figure 8.

Figure 12. Families of steady and time-dependent cellular interfaces for system II as a function of increzising growth rate P, es computed in a 2Ac sample size.

FIGURE 2.1 A side view of the structure of the prototype G-protein-coupled, 7TM receptor rhodopsin. The x-ray structure of bovine rhodopsin is shown with horizontal gray lines, indicating the limits of the cellular lipid membrane. The retinal ligand is shown in a space-filling model as the cloud in the middle of the structure. The seven transmembrane (7TM) helices are shown in solid ribbon form. Note that TM-III is rather tilted (see TM-III at the extracellular and intracellular end of the helix) and that kinks are present in several of the other helices, such as TM-V (to the left), TM-VI (in front of the retinal), and TM-VII. In all of these cases, these kinks are due to the presence of a well-conserved proline residue, which creates a weak point in the helical structure. These kinks are believed to be of functional importance in the activation mechanism for 7TM receptors in general. Also note the amphipathic helix-VIII which is located parallel to the membrane at the membrane interface. 

Jarcho, M., Kay, J.L., Gumaer, R.H. and Drobeck, H.P. (1977) Tissue, cellular, and subcellular events at a bone-ceramic apatite interface. Journal of Bioengineering, 1, 79-92. 

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