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Injuries overview

There are multiple mechanisms known to underlie the neuronal cell damage associated with injury or disease that at least theoretically could be targeted for pharmaceutical intervention. Currently however, there is no clinically available therapeutic agent that can reliably protect the brain from progressive neurodegenerative processes for sustained periods. Due to the extensive amount of preclinical research that has been conducted in recent years, there is a basis for optimism, however, it appears likely that some of these approaches will result in clinically effective therapeutic modalities in the near future. A short overview of some of the investigational approaches to combat neurodegeneration appears below. [Pg.826]

An overview of animal welfare in organic farming by Vonne Lund is presented elsewhere (see Chapter 8). Animal welfare is reduced in conventional intensive housing systems because of high densities and lack of stimuli like straw, and this may lead to behavioural disturbances, injuries or some diseases. Livestock housing on organic farms should allow for the behavioural requirements of different farm animal species, facilitate efficient management, and use environmentally friendly construction materials and methods. [Pg.152]

Overview. The Toxic Substances Control Act (TSCA) became effective January 1, 1977. Its major goal is to prevent unreasonable risk of injury to health or the environment from exposure to chemical substances. It has been said that TSCA is the most powerful, wide-ranging environmental law in the world. Prior to its enactment, there had been extensive public discussion about the possible harmful effects to workers and the general public from the manufacture, processing, distribution, use, and disposal of chemicals. [Pg.365]

Disasters that are due to natural causes often result in significant losses, physical destruction of dwellings, social and economic disruption, human pain and suffering, injury, and loss of life. This chapter presents the reader with a broad overview of the most frequently occurring natural/ environmental disasters, their impact on communities, and... [Pg.327]

Petras, J.M. (1994). Neurology and neuropathology of soman-induced brain injury an overview. J. Exp. Anal. Behav. 61 319-29. [Pg.491]

Overview of Oral Health Effects. Studies in humans and animals indicate that ingestion of asbestos causes little or no risk of noncarcinogenic injury. However, there is some evidence that acute oral exposure may induce precursor lesions of colon cancer, and that chronic oral exposure may lead to an increased incidence risk of gastrointestinal tumors. Studies that provide quantitative data on the effects of ingested asbestos are summarized in Table 3-3 and Figure 3-3, and the data are discussed below. [Pg.75]

Ward, P.A. (1991). Overview of the processes of cellular injury in interstitial lung disease. Chest 100, 230-232. [Pg.226]

An overview of precautions against radiation injury is also given by D.C. Creagh and S. Martinez-Carrera in the International Tables for Crystallography, vol. C, Second edition, A.J.C. Wilson and E. Prince, Eds., Kluwer Academic Publishers, Boston/Dordrecht/ London (1999) p. 949. [Pg.280]

Higuchi H, Gores GJ. Mechanisms of liver injury An overview. Curr Mol Med 2003 3 483-90. [Pg.1847]

However, it is clear that ischaemic injury will affect the activity of all types of cells within the affected region of the brain. The purpose of this chapter is to provide an overview of cellular responses by all of the cell types found within the brain. These will be treated in alphabetical order rather than to give greater emphasis to changes in one cell type. [Pg.20]

This chapter provides an overview of the pathophysiologic mechanisms of glomerular injury and the clinical presentations of glomerulonephritis. The treatment approach for the common forms of glomerulonephritis are also discussed. Although diabetes mellitus and amyloidosis are important secondary causes of glomerular diseases, the scope of this chapter is limited to the primary causes of glomerulonephritis. [Pg.891]

Immune dysregulation leading to B-cell hyperactivity and subsequent production of pathogenic autoantibodies, coupled with defective clearance of apoptotic cells, followed by immune-complex formation, complement activation, and defective clearance of immune complexes all lead to inflammatory reactions that ultimately result in tissue injury and damage. An overview of the pathogenesis of SLE is illustrated in Fig. 85-1. [Pg.1583]

Olajos EJ, Olson CT, Salem H, Singer AW, Hayes TL, Menton RG, Miller TL, Rosso T, Maclver B. Evaluation of neutralized chemical agent identification sets (CAIS) for skin injury with an overview of the vesicant potential of agent degradation products. J Appl Toxicol. 1998 Nov-Dec 18(6) 409-20)... [Pg.149]


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Injury prevention overview

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