In focal segmental glomerulosclerosis

K25. Kriz, W., Hahnel, B., Rosener, S., and Eiger, M., Long-term treatment of rats with FGF-2 results in focal segmental glomerulosclerosis. Kidney Int. 48, 1435-1450 (1995). 

Shirato, I., Hosser, H., Kimura, K., Sakai, T., Tomino, Y., and Kriz, W. (1996). The development of focal segmental glomerulosclerosis in masugi nephritis is based on progressive podocyte damage. Virchows Arch. 429, 255—273. 

Nephrotic syndrome may complicate the course of many primary and secondary glomerulopathies. Diabetic nephropathy is the most common cause of nephrotic proteinuria (not always accompanied by full-blown nephrotic syndrome). Lupus nephritis and renal amyloidosis are much rarer secondary glomerulopathies resulting in nephrotic syndrome. The prevalence of primary glomerulopathies differs between Blacks and Whites (focal segmental glomerulosclerosis is more common... 

Dl. Dall Amico, R., Ghiggeri, G., Carraro, M., Artero, M., Ohio, L., Zamorani, E., Zennaro, C., Basile, G., Montini, G., Rivabella, L., Cardillo, M., Scalamogna, M., and Ginevri, F., Prediction and treatment of recurrent focal segmental glomerulosclerosis after renal transplantation in children. Am. J. Kidney Dis. 34, 1048-1055 (1999). 

Ingulli, E., and Tejani, A., Incidence, treatment and outcome of recurrent focal segmental glomerulosclerosis posttransplantation in 42 allografts in children A single-center experience. Transplantation 51, 401—405 (1991). 

L2. Lee, D. Y., Kim, W., and Kang, S. K., Angiotensin-converting enzyme gene polymorphism in patients with minimal-change nephrotic syndrome and focal segmental glomerulosclerosis. Nephron 77,471-473 (1997). 

Sharma, M., Sharma, R., McCarthy, E. T., and Savin, V. J., The FSGS factor Enrichment and in vivo effect of activity from focal segmental glomerulosclerosis plasma. J. Am. Soc. Nephrol. 10, 552-561 (1999). 

Nephrotic syndrome (proteinuria, edema, hypoalbumine-mia, hyperlipidemia) is a rare and idiosyncratic complication of lithium therapy it usually resolves on withdrawal, and can recur on rechallenge (397,398). Lithium-associated nephrotic syndrome occurred in a 59-year-old woman with lithium toxicity (serum concentration 1.9 mmol/1) whose renal biopsy showed focal segmental glomerulosclerosis. Lithium withdrawal led to resolution of edema and marked improvement in proteinuria and albuminemia (398). 

A 59-year-old woman with lithium-associated nephrotic syndrome (focal segmental glomerulosclerosis on biopsy) had resolution of edema and pleural effusions and marked improvement in albuminemia and proteinuria after withdrawal of lithium (398). 

Jadoul M. Interferon-alpha-associated focal segmental glomerulosclerosis with massive proteinuria in patients with chronic myeloid leukemia following high dose chemotherapy. Cancer 1999 85(12) 2669-70. 

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