Masugi nephritis

Shirato, I., Hosser, H., Kimura, K., Sakai, T., Tomino, Y., and Kriz, W. (1996). The development of focal segmental glomerulosclerosis in masugi nephritis is based on progressive podocyte damage. Virchows Arch. 429, 255—273. 

Various experimental procedures were described as models for glomerulonephritis in human beings. Most of them were developed in rats and rabbits. They involve the reactions of antibodies against renal components, such as Masugi nephritis (Masugi and Sato 1934), Heymann nephritis (Heymann 1959), nephrotoxic serum nephritis (Unanue and Dixon 1967), crescentic type anti-glomerular membrane nephritis (Nagoe et al. 1994, 1998), anti-Thyl nephritis (Chen etal. 1999). 

Kawasaki et al. (1995) studied the therapeutic effect of combined treatment with monoclonal antibodies against intercellular adhesion moleculel (ICAM-1) and lymphocyte-function-associated antigenl (LFA-1) in Masugi nephritis of Wistar-Kyoto rats. 

Okubo et al. (1990) studied the immunosuppressive effects of FK506 on active Heymann s nephritis and the autologous phase of Masugi nephritis. 

Okubo Y, Tsukada Y, Maezawa A et al. (1990) FK506, a novel immunosuppressive agent, induces antigen-specific im-munotolerance in active Heymann s nephritis and in the autologous phase of Masugi nephritis. Clin Exp Immunol 82 450-455... 

Fujimoto, T., Okada, M., Kondo, Y, and Tada, T. (1964) The nature of masugi nephritis histo- and immunopathological studies. Acta Pathol. Jpn. 14, 275-310. 

IiJiMA, S., Die Ohrarterien des Kaninchens bei Masugi-Nephritis und bei experi-menteller renaler Hypertonie im intravitalen Photogramm, Beitr. pathol. Anat. 119, 433 (1958). 

---